G. Bernhard Landwehrmeyer

28.2k total citations · 2 hit papers
255 papers, 13.2k citations indexed

About

G. Bernhard Landwehrmeyer is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, G. Bernhard Landwehrmeyer has authored 255 papers receiving a total of 13.2k indexed citations (citations by other indexed papers that have themselves been cited), including 211 papers in Cellular and Molecular Neuroscience, 130 papers in Neurology and 129 papers in Molecular Biology. Recurrent topics in G. Bernhard Landwehrmeyer's work include Genetic Neurodegenerative Diseases (165 papers), Neurological disorders and treatments (112 papers) and Mitochondrial Function and Pathology (76 papers). G. Bernhard Landwehrmeyer is often cited by papers focused on Genetic Neurodegenerative Diseases (165 papers), Neurological disorders and treatments (112 papers) and Mitochondrial Function and Pathology (76 papers). G. Bernhard Landwehrmeyer collaborates with scholars based in Germany, United States and United Kingdom. G. Bernhard Landwehrmeyer's co-authors include David G. Standaert, Raymund A.C. Roos, Sarah J. Tabrizi, Alexandra Dürr, Ralf Reilmann, Anne B. Young, Julie C. Stout, David Craufurd, Rachael I. Scahill and John B. Penney and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Journal of Neuroscience.

In The Last Decade

G. Bernhard Landwehrmeyer

248 papers receiving 12.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Biological and clinical manifestations of Huntington's disease in the longitudinal TRACK-HD study: cross-sectional analysis of baseline data

2009 756 citations Sarah J. Tabrizi, Douglas R. Langbehn et al. profile →
Predictors of phenotypic progression and disease onset in premanifest and early-stage Huntington's disease in the TRACK-HD study: analysis of 36-month observational data

2013 622 citations Sarah J. Tabrizi, Rachael I. Scahill et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G. Bernhard Landwehrmeyer Germany	61	10.3k	6.6k	6.1k	1.4k	1.1k	255	13.2k
Raymund A.C. Roos Netherlands	61	8.7k 0.8×	5.5k 0.8×	6.9k 1.1×	1.1k 0.8×	1.4k 1.3×	294	13.1k
Carlos Cepeda United States	62	9.4k 0.9×	5.7k 0.9×	3.9k 0.6×	2.0k 1.4×	942 0.9×	200	12.3k
Michael S. Levine United States	69	11.5k 1.1×	6.7k 1.0×	4.8k 0.8×	2.7k 1.9×	1.2k 1.1×	270	15.7k
Paola Piccini United Kingdom	60	5.4k 0.5×	2.9k 0.4×	6.5k 1.1×	1.4k 1.0×	940 0.9×	195	11.4k
John B. Penney United States	63	13.4k 1.3×	6.4k 1.0×	7.7k 1.3×	2.6k 1.8×	1.1k 1.0×	139	17.1k
Steven M. Hersch United States	55	8.9k 0.9×	8.5k 1.3×	3.7k 0.6×	952 0.7×	945 0.9×	129	12.6k
Barbara Picconi Italy	55	7.0k 0.7×	2.8k 0.4×	4.7k 0.8×	1.9k 1.3×	903 0.8×	137	10.7k
Lüdger Schöls Germany	62	8.8k 0.9×	7.1k 1.1×	5.1k 0.8×	602 0.4×	1.1k 1.0×	352	13.2k
Antonio Pisani Italy	54	5.9k 0.6×	2.7k 0.4×	4.9k 0.8×	1.2k 0.8×	974 0.9×	245	9.6k
Edward D. Bird United States	56	7.3k 0.7×	5.4k 0.8×	3.4k 0.6×	1.0k 0.7×	2.0k 1.8×	117	11.8k

Countries citing papers authored by G. Bernhard Landwehrmeyer

Since Specialization

Citations

This map shows the geographic impact of G. Bernhard Landwehrmeyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Bernhard Landwehrmeyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Bernhard Landwehrmeyer more than expected).

Fields of papers citing papers by G. Bernhard Landwehrmeyer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Bernhard Landwehrmeyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Bernhard Landwehrmeyer. The network helps show where G. Bernhard Landwehrmeyer may publish in the future.

Co-authorship network of co-authors of G. Bernhard Landwehrmeyer

This figure shows the co-authorship network connecting the top 25 collaborators of G. Bernhard Landwehrmeyer. A scholar is included among the top collaborators of G. Bernhard Landwehrmeyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Bernhard Landwehrmeyer. G. Bernhard Landwehrmeyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Estevez‐Fraga, Carlos, Justine Y. Hansen, Paul Zeun, et al.. (2025). Cell-specific mechanisms drive connectivity across the time course of Huntington’s disease. Nature Communications. 16(1). 5519–5519.

Halbgebauer, Steffen, Gabriele Nagel, Angela Rosenbohm, et al.. (2025). Age-Specific Control and Alzheimer Disease Reference Curves and z -Scores for Glial Fibrillary Acidic Protein in Blood. Clinical Chemistry. 71(12). 1234–1242.

Wagner, Maximilian, Pallab Maity, Sebastian Wiese, et al.. (2024). General loss of proteostasis links Huntington disease to Cockayne syndrome. Neurobiology of Disease. 201. 106668–106668. 2 indexed citations

Wagner, Maximilian, et al.. (2024). Huntingtin HTT1a is generated in a CAG repeat-length-dependent manner in human tissues. Molecular Medicine. 30(1). 36–36. 10 indexed citations

Estevez‐Fraga, Carlos, André Altmann, Christopher S. Parker, et al.. (2023). Genetic topography and cortical cell loss in Huntington's disease link development and neurodegeneration. Brain. 146(11). 4532–4546. 18 indexed citations

Estevez‐Fraga, Carlos, Marina Papoutsi, Alexandra Dürr, et al.. (2023). Progressive alterations in white matter microstructure across the timecourse of Huntington's disease. Brain and Behavior. 13(4). e2940–e2940. 6 indexed citations

Tykalová, Tereza, et al.. (2023). Speech biomarkers in Huntington's disease: A cross‐sectional study in pre‐symptomatic, prodromal and early manifest stages. European Journal of Neurology. 30(5). 1262–1271. 13 indexed citations

Burg, Jorien M.M. van der, Patrick Weydt, G. Bernhard Landwehrmeyer, & N. Ahmad Aziz. (2021). Effect of Body Weight on Age at Onset in Huntington Disease. Neurology Genetics. 7(4). e603–e603. 10 indexed citations

Einem, Bjoern von, et al.. (2020). Ubiquitination and the proteasome rather than caspase‐3‐mediated C‐terminal cleavage are involved in the EAAT2 degradation by staurosporine‐induced cellular stress. Journal of Neurochemistry. 157(4). 1284–1299. 3 indexed citations

10.

Dogan, Imis, Olga A. Wudarczyk, Mikhail Votinov, et al.. (2018). Risk factors of suicidal ideation in Huntington’s disease: literature review and data from Enroll-HD. Journal of Neurology. 265(11). 2548–2561. 26 indexed citations

11.

Denk, Johannes, Johannes Kornhuber, Jens Wiltfang, et al.. (2018). Specific serum and CSF microRNA profiles distinguish sporadic behavioural variant of frontotemporal dementia compared with Alzheimer patients and cognitively healthy controls. PLoS ONE. 13(5). e0197329–e0197329. 69 indexed citations

12.

Gelderblom, Hans, Torsten Wüstenberg, Wilhelm Fischer, et al.. (2017). Bupropion for the treatment of apathy in Huntington’s disease: A multicenter, randomised, double-blind, placebo-controlled, prospective crossover trial. PLoS ONE. 12(3). e0173872–e0173872. 43 indexed citations

13.

Gorges, Martin, Hans‐Peter Müller, Thomas Kammer, et al.. (2017). Intact sensory-motor network structure and function in far from onset premanifest Huntington’s disease. Scientific Reports. 7(1). 43841–43841. 10 indexed citations

14.

Squitieri, Ferdinando, G. Bernhard Landwehrmeyer, Ralf Reilmann, et al.. (2013). One-year safety and tolerability profile of pridopidine in patients with Huntington disease. Neurology. 80(12). 1086–1094. 16 indexed citations

15.

Aziz, N. Ahmad, Caroline K. Jurgens, G. Bernhard Landwehrmeyer, et al.. (2009). Normal and mutant HTT interact to affect clinical severity and progression in Huntington disease. Neurology. 73(16). 1280–1285. 60 indexed citations

16.

Landwehrmeyer, G. Bernhard, et al.. (2008). Weight loss in Huntington's disease is related to the number of CAG repeats. Journal of Neurology Neurosurgery & Psychiatry. 79. 17–17. 1 indexed citations

17.

Wolf, Robert Christian, Nenad Vasić, Carlos Schönfeldt‐Lecuona, Daniel Ecker, & G. Bernhard Landwehrmeyer. (2008). Cortical dysfunction in patients with Huntington's disease during working memory performance. Human Brain Mapping. 30(1). 327–339. 65 indexed citations

18.

Landwehrmeyer, G. Bernhard, et al.. (2007). Choreatische Bewegungsstorungen. Ursachen, Diagnostik und Therapie. Der Nervenarzt. 78. 37–49.

19.

Standaert, David G., et al.. (2000). Expression of NMDA receptor subunit mRNAs in neurochemically identified projection and interneurons in the human striatum. The Journal of Comparative Neurology. 419(4). 407–421. 86 indexed citations

20.

Feuerstein, Thomas J., et al.. (1996). 5-HT1D-like receptors inhibit the release of endogenously formed [3H]GABA in human, but not in rabbit, neocortex. Neuroscience Letters. 209(3). 210–214. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact