Mauro Pessia

3.8k total citations
79 papers, 2.8k citations indexed

About

Mauro Pessia is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Mauro Pessia has authored 79 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Molecular Biology, 50 papers in Cellular and Molecular Neuroscience and 29 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Mauro Pessia's work include Ion channel regulation and function (57 papers), Cardiac electrophysiology and arrhythmias (27 papers) and Neuroscience and Neuropharmacology Research (21 papers). Mauro Pessia is often cited by papers focused on Ion channel regulation and function (57 papers), Cardiac electrophysiology and arrhythmias (27 papers) and Neuroscience and Neuropharmacology Research (21 papers). Mauro Pessia collaborates with scholars based in Italy, Malta and United States. Mauro Pessia's co-authors include Maria Cristina D’Adamo, Paola Imbrici, Stephen J. Tucker, John P. Adelman, Chris T. Bond, R A North, Lorena Salvatore, Luigi Catacuzzeno, Fabio Franciolini and Giuseppe Di Giovanni and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Neuron.

In The Last Decade

Mauro Pessia

77 papers receiving 2.8k citations

Peers

Mauro Pessia
Claudia A. Sailer Austria
Toni Schneider Germany
Sven Moosmang Germany
Dirk Isbrandt Germany
Björn C. Schroeder Germany
F. Lehmann‐Horn Germany
James Offord United States
Hai Yan United States
Stéphanie Schorge United Kingdom
Guoxin Kang United States
Claudia A. Sailer Austria
Mauro Pessia
Citations per year, relative to Mauro Pessia Mauro Pessia (= 1×) peers Claudia A. Sailer

Countries citing papers authored by Mauro Pessia

Since Specialization
Citations

This map shows the geographic impact of Mauro Pessia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mauro Pessia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mauro Pessia more than expected).

Fields of papers citing papers by Mauro Pessia

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mauro Pessia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mauro Pessia. The network helps show where Mauro Pessia may publish in the future.

Co-authorship network of co-authors of Mauro Pessia

This figure shows the co-authorship network connecting the top 25 collaborators of Mauro Pessia. A scholar is included among the top collaborators of Mauro Pessia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mauro Pessia. Mauro Pessia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Al‐Marzooq, Farah, et al.. (2025). Zinc ion dyshomeostasis in autism spectrum disorder. Nutrition Research Reviews. 38(2). 661–681.
2.
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Venkataraman, Balaji, Bhoomendra A. Bhongade, Rajesh B. Patil, et al.. (2025). Natural compounds targeting inflammation and oxidative stress in intestinal ischemia-reperfusion injury: a comprehensive review. Phytomedicine Plus. 5(3). 100855–100855. 1 indexed citations
4.
Venkataraman, Balaji, et al.. (2024). Phytochemical Compounds as Promising Therapeutics for Intestinal Fibrosis in Inflammatory Bowel Disease: A Critical Review. Nutrients. 16(21). 3633–3633. 2 indexed citations
5.
Ismail, Fatima, Manigandan Krishnan, Richard L. Jayaraj, et al.. (2024). Time dependent changes in protein expression induced by intermittent theta burst stimulation in a cell line. Frontiers in Neurology. 15. 1396776–1396776. 1 indexed citations
6.
Megaro, Alfredo, Lorena Coretti, Fabio Massimo Botti, et al.. (2020). Electromechanical coupling of the Kv1.1 voltage-gated K+ channel is fine-tuned by the simplest amino acid residue in the S4-S5 linker. Pflügers Archiv - European Journal of Physiology. 472(7). 899–909. 3 indexed citations
7.
Megaro, Alfredo, Alev Güven, Paola Imbrici, et al.. (2018). Identification of a New de Novo Mutation Underlying Regressive Episodic Ataxia Type I. Frontiers in Neurology. 9. 587–587. 9 indexed citations
8.
Romani, Luigina, Vasileios Oikonomou, Silvia Moretti, et al.. (2017). Thymosin α1 represents a potential potent single-molecule-based therapy for cystic fibrosis. Nature Medicine. 23(5). 590–600. 77 indexed citations
9.
Imbrici, Paola, Concetta Altamura, Mauro Pessia, et al.. (2015). ClC-1 chloride channels: state-of-the-art research and future challenges. Frontiers in Cellular Neuroscience. 9. 156–156. 50 indexed citations
10.
Ledonne, Ada, Mauro Federici, Michela Giustizieri, et al.. (2010). Trace amines depress D2‐autoreceptor‐mediated responses on midbrain dopaminergic cells. British Journal of Pharmacology. 160(6). 1509–1520. 20 indexed citations
11.
Imbrici, Paola, Alessandro Grottesi, Maria Cristina D’Adamo, Stephen J. Tucker, & Mauro Pessia. (2009). Contributions of the central hydrophobic residue in the PXP motif of Voltage-Dependent K+ Channels to S6 flexibility and Gating Properties. Biophysical Journal. 96(3). 656a–656a. 19 indexed citations
12.
Imbrici, Paola, Maria Cristina D’Adamo, Antonella Cusimano, & Mauro Pessia. (2006). Episodic ataxia type 1 mutation F184C alters Zn2+-induced modulation of the human K+ channel Kv1.4-Kv1.1/Kvβ1.1. American Journal of Physiology-Cell Physiology. 292(2). C778–C787. 26 indexed citations
13.
Cusimano, Antonella, Maria Cristina D’Adamo, & Mauro Pessia. (2004). An episodic ataxia type‐1 mutation in the S1 segment sensitises the hKv1.1 potassium channel to extracellular Zn2+. FEBS Letters. 576(1-2). 237–244. 15 indexed citations
14.
Imbrici, Paola, Antonella Cusimano, Maria Cristina D’Adamo, Amalia De Curtis, & Mauro Pessia. (2003). Functional characterization of an episodic ataxia type-1 mutation occurring in the S1 segment of hKv1.1 channels. Pflügers Archiv - European Journal of Physiology. 446(3). 373–379. 24 indexed citations
15.
Grassi, Silvarosa, et al.. (2001). Exogenous glutamate induces short and long-term potentiation in the rat medial vestibular nuclei. Neuroreport. 12(11). 2329–2334. 6 indexed citations
16.
Tucker, Stephen J., Paola Imbrici, Lorena Salvatore, Maria Cristina D’Adamo, & Mauro Pessia. (2000). pH Dependence of the Inwardly Rectifying Potassium Channel, Kir5.1, and Localization in Renal Tubular Epithelia. Journal of Biological Chemistry. 275(22). 16404–16407. 113 indexed citations
17.
Tucker, Stephen J., Mauro Pessia, Andrew J. Moorhouse, et al.. (1996). Heteromeric channel formation and Ca2+‐free media reduce the toxic effect of the weaver Kir 3.2 allele. FEBS Letters. 390(3). 253–257. 24 indexed citations
18.
Adelman, John P., et al.. (1995). Episodic ataxia results from voltage-dependent potassium channels with altered functions. Neuron. 15(6). 1449–1454. 180 indexed citations
19.
Bond, Chris T., Mauro Pessia, Xuefeng Xia, et al.. (1994). Errata: Cloning and expression of a family of inward rectifier potassium channels (Receptors and Channels 2 (183-191)). 2(4). 6 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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