Matthew K. Waldor

32.1k total citations · 4 hit papers
284 papers, 20.7k citations indexed

About

Matthew K. Waldor is a scholar working on Endocrinology, Molecular Biology and Genetics. According to data from OpenAlex, Matthew K. Waldor has authored 284 papers receiving a total of 20.7k indexed citations (citations by other indexed papers that have themselves been cited), including 195 papers in Endocrinology, 72 papers in Molecular Biology and 65 papers in Genetics. Recurrent topics in Matthew K. Waldor's work include Vibrio bacteria research studies (160 papers), Escherichia coli research studies (103 papers) and Bacterial Genetics and Biotechnology (58 papers). Matthew K. Waldor is often cited by papers focused on Vibrio bacteria research studies (160 papers), Escherichia coli research studies (103 papers) and Bacterial Genetics and Biotechnology (58 papers). Matthew K. Waldor collaborates with scholars based in United States, China and Sweden. Matthew K. Waldor's co-authors include Brigid M. Davis, Bianca Hochhut, John J. Mekalanos, Rachel A. F. Wozniak, John W. Beaber, Vincent Burrus, Jennifer M. Ritchie, Patrick Wagner, Jonathan Livny and Felipe Cava and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

Matthew K. Waldor

278 papers receiving 20.3k citations

Hit Papers

SOS response promotes horizontal dissemination of antibio... 2003 2026 2010 2018 2003 2018 2010 2010 250 500 750

Peers

Matthew K. Waldor
Samuel I. Miller United States
Jörg Hacker Germany
Eduardo A. Groisman United States
Jorge E. Galán United States
Peter R. Reeves Australia
Andrew Camilli United States
Mark Achtman Germany
Samuel I. Miller United States
Matthew K. Waldor
Citations per year, relative to Matthew K. Waldor Matthew K. Waldor (= 1×) peers Samuel I. Miller

Countries citing papers authored by Matthew K. Waldor

Since Specialization
Citations

This map shows the geographic impact of Matthew K. Waldor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew K. Waldor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew K. Waldor more than expected).

Fields of papers citing papers by Matthew K. Waldor

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew K. Waldor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew K. Waldor. The network helps show where Matthew K. Waldor may publish in the future.

Co-authorship network of co-authors of Matthew K. Waldor

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew K. Waldor. A scholar is included among the top collaborators of Matthew K. Waldor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew K. Waldor. Matthew K. Waldor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Peterson, Stefan T., Karthik Hullahalli, Daniel Sorobetea, et al.. (2025). TNF signaling maintains local restriction of bacterial founder populations in intestinal and systemic sites during oral Yersinia infection. mBio. 16(10). e0177925–e0177925.
2.
Zhang, Hailong, Yūko Hasegawa, Masataka Suzuki, et al.. (2024). Mouse enteric neurons control intestinal plasmacytoid dendritic cell function via serotonin-HTR7 signaling. Nature Communications. 15(1). 9237–9237. 6 indexed citations
3.
Hullahalli, Karthik, et al.. (2023). Innate immune responses yield tissue-specific bottlenecks that scale with pathogen dose. Proceedings of the National Academy of Sciences. 120(37). e2309151120–e2309151120. 6 indexed citations
4.
Campbell, Ian W., Karthik Hullahalli, Jerrold R. Turner, & Matthew K. Waldor. (2023). Quantitative dose-response analysis untangles host bottlenecks to enteric infection. Nature Communications. 14(1). 456–456. 21 indexed citations
5.
Hullahalli, Karthik, Yuko Hasegawa, Masataka Suzuki, et al.. (2023). Genetic and immune determinants of E. coli liver abscess formation. Proceedings of the National Academy of Sciences. 120(51). e2310053120–e2310053120. 10 indexed citations
6.
García, K. Christopher, et al.. (2021). Identification of a Family of Vibrio Type III Secretion System Effectors That Contain a Conserved Serine/Threonine Kinase Domain. mSphere. 6(4). e0059921–e0059921. 8 indexed citations
7.
Álvarez, Laura, Brandon Sit, Oihane Irazoki, et al.. (2021). BipA exerts temperature-dependent translational control of biofilm-associated colony morphology in Vibrio cholerae. eLife. 10. 12 indexed citations
8.
Gensollen, Thomas, Xi Lin, Ting Zhang, et al.. (2021). Embryonic macrophages function during early life to determine invariant natural killer T cell levels at barrier surfaces. Nature Immunology. 22(6). 699–710. 21 indexed citations
10.
Kuehl, Carole J., et al.. (2020). An Oral Inoculation Infant Rabbit Model for Shigella Infection. mBio. 11(1). 11 indexed citations
11.
Lazarus, Jacob E., et al.. (2019). A New Suite of Allelic-Exchange Vectors for the Scarless Modification of Proteobacterial Genomes. Applied and Environmental Microbiology. 85(16). 34 indexed citations
12.
Fleurie, Aurore, Abdelrahim Zoued, Laura Álvarez, et al.. (2019). A Vibrio cholerae BolA-Like Protein Is Required for Proper Cell Shape and Cell Envelope Integrity. mBio. 10(4). 21 indexed citations
13.
Weaver, Anna I., Benjamin D. Umans, Jung‐Ho Shin, et al.. (2018). Genetic Determinants of Penicillin Tolerance in Vibrio cholerae. Antimicrobial Agents and Chemotherapy. 62(10). 29 indexed citations
14.
Pacheco, Alline R., Jacob E. Lazarus, Brandon Sit, et al.. (2018). CRISPR Screen Reveals that EHEC’s T3SS and Shiga Toxin Rely on Shared Host Factors for Infection. mBio. 9(3). 45 indexed citations
15.
Liu, Bing, Xiaohong Liu, Xiangshan Zhou, et al.. (2018). Critical role for a promoter discriminator in RpoS control of virulence in Edwardsiella piscicida. PLoS Pathogens. 14(8). e1007272–e1007272. 51 indexed citations
16.
Cendejas‐Bueno, Emilio, Brandon Sit, Matthew K. Waldor, & Felipe Cava. (2018). Anaerobic nitrate reduction divergently governs population expansion of the enteropathogen Vibrio cholerae. Nature Microbiology. 3(12). 1346–1353. 35 indexed citations
17.
Beaulaurier, John, Xue‐Song Zhang, Shijia Zhu, et al.. (2015). Single molecule-level detection and long read-based phasing of epigenetic variations in bacterial methylomes. Nature Communications. 6(1). 74 indexed citations
18.
Ringgaard, Simon, Martha A. Zepeda‐Rivera, Xiaoji Wu, et al.. (2013). ParP prevents dissociation of CheA from chemotactic signaling arrays and tethers them to a polar anchor. Proceedings of the National Academy of Sciences. 111(2). E255–64. 39 indexed citations
19.
Zhou, Xiaohui, Benjamin E. Gewurz, Jennifer M. Ritchie, et al.. (2013). A Vibrio parahaemolyticus T3SS Effector Mediates Pathogenesis by Independently Enabling Intestinal Colonization and Inhibiting TAK1 Activation. Cell Reports. 3(5). 1690–1702. 71 indexed citations
20.
Lam, Hubert, Dong‐Chan Oh, Felipe Cava, et al.. (2009). D-Amino Acids Govern Stationary Phase Cell Wall Remodeling in Bacteria. Science. 325(5947). 1552–1555. 493 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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