Benjamin E. Gewurz

6.7k total citations · 1 hit paper
81 papers, 3.9k citations indexed

About

Benjamin E. Gewurz is a scholar working on Oncology, Immunology and Epidemiology. According to data from OpenAlex, Benjamin E. Gewurz has authored 81 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 50 papers in Oncology, 33 papers in Immunology and 27 papers in Epidemiology. Recurrent topics in Benjamin E. Gewurz's work include Viral-associated cancers and disorders (47 papers), Cytomegalovirus and herpesvirus research (25 papers) and Lymphoma Diagnosis and Treatment (24 papers). Benjamin E. Gewurz is often cited by papers focused on Viral-associated cancers and disorders (47 papers), Cytomegalovirus and herpesvirus research (25 papers) and Lymphoma Diagnosis and Treatment (24 papers). Benjamin E. Gewurz collaborates with scholars based in United States, China and United Kingdom. Benjamin E. Gewurz's co-authors include Hidde L. Ploegh, Domenico Tortorella, Danny J. Schust, Margo H. Furman, Bo Zhao, Sizun Jiang, Elliott Kieff, Rui Guo, Liang Wei Wang and Peter J. Krause and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Benjamin E. Gewurz

75 papers receiving 3.9k citations

Hit Papers

Viral Subversion of the Immune System 2000 2026 2008 2017 2000 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Benjamin E. Gewurz United States 37 1.5k 1.5k 1.2k 1.2k 623 81 3.9k
Takayuki Murata Japan 34 788 0.5× 1.7k 1.1× 1.7k 1.4× 1.5k 1.3× 466 0.7× 147 4.2k
Tatsuya Tsurumi Japan 42 1.1k 0.7× 2.5k 1.7× 2.5k 2.1× 1.5k 1.3× 530 0.9× 123 4.8k
Maaike E. Ressing Netherlands 37 2.7k 1.8× 1.7k 1.1× 2.1k 1.7× 1.0k 0.9× 358 0.6× 73 4.4k
Timothy F. Kowalik United States 42 850 0.6× 1.8k 1.2× 2.2k 1.8× 2.7k 2.3× 212 0.3× 84 5.6k
Marvin S. Reitz United States 36 1.8k 1.2× 1.5k 1.0× 1.0k 0.8× 1.3k 1.1× 305 0.5× 90 5.0k
Graham R. Leggatt Australia 32 2.4k 1.6× 940 0.6× 953 0.8× 866 0.7× 194 0.3× 97 3.7k
Armin Ensser Germany 30 679 0.5× 1.4k 1.0× 1.4k 1.2× 472 0.4× 163 0.3× 93 2.8k
Kanji Hirai Japan 35 737 0.5× 1.4k 0.9× 2.0k 1.6× 581 0.5× 603 1.0× 147 3.7k
Laurent Coscoy United States 25 1.1k 0.7× 1.0k 0.7× 1.1k 1.0× 1.2k 1.1× 117 0.2× 41 3.1k
Gislâine A. Martins United States 28 2.7k 1.8× 739 0.5× 892 0.7× 702 0.6× 111 0.2× 48 3.8k

Countries citing papers authored by Benjamin E. Gewurz

Since Specialization
Citations

This map shows the geographic impact of Benjamin E. Gewurz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin E. Gewurz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin E. Gewurz more than expected).

Fields of papers citing papers by Benjamin E. Gewurz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin E. Gewurz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin E. Gewurz. The network helps show where Benjamin E. Gewurz may publish in the future.

Co-authorship network of co-authors of Benjamin E. Gewurz

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin E. Gewurz. A scholar is included among the top collaborators of Benjamin E. Gewurz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin E. Gewurz. Benjamin E. Gewurz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Wang, Yin, Yizhe Sun, Bidisha Mitra, et al.. (2025). The CTLH ubiquitin ligase substrates ZMYND19 and MKLN1 negatively regulate mTORC1 at the lysosomal membrane. Nature Communications. 16(1). 10731–10731.
3.
Reynoso, Glennys V., Thomas J. Meyer, Monika A. Zelazowska, et al.. (2024). Multifaceted roles for STAT3 in gammaherpesvirus latency revealed through in vivo B cell knockout models. mBio. 15(2). e0299823–e0299823. 2 indexed citations
4.
Giulino‐Roth, Lisa, et al.. (2024). Germinal center cytokine driven epigenetic control of Epstein-Barr virus latency gene expression. PLoS Pathogens. 20(4). e1011939–e1011939. 6 indexed citations
5.
Mitra, Bidisha, et al.. (2023). Characterization of target gene regulation by the two Epstein-Barr virus oncogene LMP1 domains essential for B-cell transformation. mBio. 14(6). e0233823–e0233823. 9 indexed citations
6.
Shrock, Ellen, Richard T. Timms, Tomasz Kula, et al.. (2023). Germline-encoded amino acid–binding motifs drive immunodominant public antibody responses. Science. 380(6640). eadc9498–eadc9498. 45 indexed citations
7.
Burton, Eric, et al.. (2022). Epstein–Barr virus latency programs dynamically sensitize B cells to ferroptosis. Proceedings of the National Academy of Sciences. 119(11). e2118300119–e2118300119. 50 indexed citations
8.
Yiu, Stephanie Pei Tung, et al.. (2022). Epstein-Barr virus BNRF1 destabilizes SMC5/6 cohesin complexes to evade its restriction of replication compartments. Cell Reports. 38(10). 110411–110411. 45 indexed citations
9.
Liang, Jin, Chong Wang, Stephanie Pei Tung Yiu, et al.. (2021). Epstein-Barr Virus Induced Cytidine Metabolism Roles in Transformed B-Cell Growth and Survival. mBio. 12(4). e0153021–e0153021. 24 indexed citations
10.
Wang, Zhonghao, Rui Guo, Stephen J. Trudeau, et al.. (2021). CYB561A3 is the key lysosomal iron reductase required for Burkitt B-cell growth and survival. Blood. 138(22). 2216–2230. 20 indexed citations
11.
Soldan, Samantha S., Drew Frase, Yue Zhang, et al.. (2021). EBNA1 inhibitors have potent and selective antitumor activity in xenograft models of Epstein–Barr virus-associated gastric cancer. Gastric Cancer. 24(5). 1076–1088. 25 indexed citations
12.
Zhang, Yuchen, Rui Guo, Hardik Shah, et al.. (2021). SARS-CoV-2 hijacks folate and one-carbon metabolism for viral replication. Nature Communications. 12(1). 1676–1676. 103 indexed citations
13.
Gewurz, Benjamin E., et al.. (2018). Epigenetic crossroads of the Epstein-Barr virus B-cell relationship. Current Opinion in Virology. 32. 15–23. 19 indexed citations
14.
Ma, Yijie, Michael J. Walsh, Katharina Bernhardt, et al.. (2017). CRISPR/Cas9 Screens Reveal Epstein-Barr Virus-Transformed B Cell Host Dependency Factors. Cell Host & Microbe. 21(5). 580–591.e7. 104 indexed citations
15.
Blondel, Carlos J., Joseph S. Park, Troy P. Hubbard, et al.. (2016). CRISPR/Cas9 Screens Reveal Requirements for Host Cell Sulfation and Fucosylation in Bacterial Type III Secretion System-Mediated Cytotoxicity. Cell Host & Microbe. 20(2). 226–237. 53 indexed citations
16.
Hunter, Jill V., Jacqueline Butterworth, Bo Zhao, et al.. (2015). The NF-κB subunit c-Rel regulates Bach2 tumour suppressor expression in B-cell lymphoma. Oncogene. 35(26). 3476–3484. 28 indexed citations
17.
Greenfeld, Hannah, Kaoru Takasaki, Michael J. Walsh, et al.. (2015). TRAF1 Coordinates Polyubiquitin Signaling to Enhance Epstein-Barr Virus LMP1-Mediated Growth and Survival Pathway Activation. PLoS Pathogens. 11(5). e1004890–e1004890. 64 indexed citations
18.
Zhou, Xiaohui, Benjamin E. Gewurz, Jennifer M. Ritchie, et al.. (2013). A Vibrio parahaemolyticus T3SS Effector Mediates Pathogenesis by Independently Enabling Intestinal Colonization and Inhibiting TAK1 Activation. Cell Reports. 3(5). 1690–1702. 71 indexed citations
19.
Gewurz, Benjamin E., Francisco M. Marty, Lindsey R. Baden, & Joel T. Katz. (2008). Human herpesvirus 6 encephalitis. Current Infectious Disease Reports. 10(4). 292–9. 32 indexed citations
20.
McAdam, Alexander J., Benjamin E. Gewurz, Evan A. Farkash, & Arlene H. Sharpe. (2000). Either B7 Costimulation or IL-2 Can Elicit Generation of Primary Alloreactive CTL. The Journal of Immunology. 165(6). 3088–3093. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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