Matthew J. Taylor

2.2k total citations
33 papers, 1.8k citations indexed

About

Matthew J. Taylor is a scholar working on Molecular Biology, Epidemiology and Cancer Research. According to data from OpenAlex, Matthew J. Taylor has authored 33 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 6 papers in Epidemiology and 6 papers in Cancer Research. Recurrent topics in Matthew J. Taylor's work include Monoclonal and Polyclonal Antibodies Research (4 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Click Chemistry and Applications (3 papers). Matthew J. Taylor is often cited by papers focused on Monoclonal and Polyclonal Antibodies Research (4 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Click Chemistry and Applications (3 papers). Matthew J. Taylor collaborates with scholars based in United States, United Kingdom and Saudi Arabia. Matthew J. Taylor's co-authors include Yatrik M. Shah, Frank J. Gonzalez, Aijuan Qu, Erik R. Anderson, Xiang Xue, Tsutomu Matsubara, Charles W. Beatty, Angelical Martin, Ellen M. Zimmermann and William T. Eckenhoff and has published in prestigious journals such as Science, Journal of the American Chemical Society and Gastroenterology.

In The Last Decade

Matthew J. Taylor

31 papers receiving 1.7k citations

Peers

Matthew J. Taylor

SURGE

ONCOL

HEMAT

William Westlin United States

Fernando Doñate United States

Anna Laurenzana Italy

Niamh Moran Ireland

Yoko Endo Japan

Malin Wickström Sweden

Tomasz Stokłosa Poland

Mirella Belleri Italy

Donald G. Munroe United States

John C. Voyta United States

William Westlin United States

Matthew J. Taylor

1.2k ×1.9

555 ×1.5

175 ×0.6

SURGE

545 ×2.3

ONCOL

218 ×0.9

HEMAT

Citations per year, relative to Matthew J. Taylor Matthew J. Taylor (= 1×) peers William Westlin

Countries citing papers authored by Matthew J. Taylor

Since Specialization

Citations

This map shows the geographic impact of Matthew J. Taylor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Taylor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Taylor more than expected).

Fields of papers citing papers by Matthew J. Taylor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Taylor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Taylor. The network helps show where Matthew J. Taylor may publish in the future.

Co-authorship network of co-authors of Matthew J. Taylor

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Taylor. A scholar is included among the top collaborators of Matthew J. Taylor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Taylor. Matthew J. Taylor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Taylor, Matthew J., et al.. (2025). C9orf72-related amyotrophic lateral sclerosis-frontotemporal dementia and links to the DNA damage response: a systematic review. Frontiers in Molecular Neuroscience. 18. 1671906–1671906.

Taylor, Matthew J., et al.. (2025). FUS-related amyotrophic lateral sclerosis-frontotemporal dementia and links to the DNA damage response: a systematic review. Frontiers in Molecular Neuroscience. 18. 1671910–1671910.

Taylor, Matthew J., et al.. (2022). Inhibition of Chk2 promotes neuroprotection, axon regeneration, and functional recovery after CNS injury. Science Advances. 8(37). eabq2611–eabq2611. 21 indexed citations

Clark, Andrew, et al.. (2020). Introduction to Stereochemistry. 2 indexed citations

Tuxworth, Richard I., Matthew J. Taylor, Ane Martín Anduaga, et al.. (2019). Attenuating the DNA damage response to double-strand breaks restores function in models of CNS neurodegeneration. Brain Communications. 1(1). fcz005–fcz005. 22 indexed citations

Taylor, Matthew J. & Richard I. Tuxworth. (2019). Continuous tracking of startled Drosophila as an alternative to the negative geotaxis climbing assay. Journal of Neurogenetics. 33(3). 190–198. 13 indexed citations

Wall, Geoffrey C., Matthew J. Taylor, & Hayden L. Smith. (2018). Prevalence and characteristics of hospital inpatients with reported fluoroquinolone allergy. International Journal of Clinical Pharmacy. 40(4). 890–894. 8 indexed citations

Mori, Hiroyuki, Yao Yao, Brian S. Learman, et al.. (2016). Induction of WNT11 by hypoxia and hypoxia-inducible factor-1α regulates cell proliferation, migration and invasion. Scientific Reports. 6(1). 21520–21520. 43 indexed citations

Finkbeiner, Stacy R., David R. Hill, Christopher Altheim, et al.. (2015). Transcriptome-wide Analysis Reveals Hallmarks of Human Intestine Development and Maturation In Vitro and In Vivo. Stem Cell Reports. 4(6). 1140–1155. 190 indexed citations

10.

Naugler, Willscott E., Branden Tarlow, Lev M. Fedorov, et al.. (2015). Fibroblast Growth Factor Signaling Controls Liver Size in Mice With Humanized Livers. Gastroenterology. 149(3). 728–740.e15. 80 indexed citations

11.

Xie, Liwei, Xiang Xue, Matthew J. Taylor, et al.. (2014). Hypoxia-Inducible Factor/MAZ-Dependent Induction of Caveolin-1 Regulates Colon Permeability through Suppression of Occludin, Leading to Hypoxia-Induced Inflammation. Molecular and Cellular Biology. 34(16). 3013–3023. 59 indexed citations

12.

Xue, Xiang, Sadeesh K. Ramakrishnan, Erik R. Anderson, et al.. (2013). Endothelial PAS Domain Protein 1 Activates the Inflammatory Response in the Intestinal Epithelium to Promote Colitis in Mice. Gastroenterology. 145(4). 831–841. 148 indexed citations

13.

White, Sarah J., Matthew J. Taylor, Ryan T. Hurt, Michael D. Jensen, & Gregory A. Poland. (2013). Leptin-based adjuvants: An innovative approach to improve vaccine response. Vaccine. 31(13). 1666–1672. 21 indexed citations

14.

Qu, Aijuan, Matthew J. Taylor, Xiang Xue, et al.. (2011). Hypoxia-inducible transcription factor 2α promotes steatohepatitis through augmenting lipid accumulation, inflammation, and fibrosis. Hepatology. 54(2). 472–483. 152 indexed citations

15.

Taylor, Matthew J., Aijuan Qu, Erik R. Anderson, et al.. (2011). Hypoxia-Inducible Factor-2α Mediates the Adaptive Increase of Intestinal Ferroportin During Iron Deficiency in Mice. Gastroenterology. 140(7). 2044–2055. 228 indexed citations

16.

Taylor, Matthew J., William T. Eckenhoff, & Tomislav Pintauer. (2010). Copper-catalyzed atom transfer radical addition (ATRA) and cyclization (ATRC) reactions in the presence of environmentally benign ascorbic acid as a reducing agent. Dalton Transactions. 39(47). 11475–11475. 69 indexed citations

17.

Taylor, Matthew J., et al.. (2003). Management of Cerebrospinal Fluid Leaks Involving the Temporal Bone: Report on 92 Patients. The Laryngoscope. 113(1). 50–56. 119 indexed citations

18.

Taylor, Matthew J., Jens U. Ponikau, David A. Sherris, et al.. (2002). Detection of Fungal Organisms in Eosinophilic Mucin Using a Fluorescein‐Labeled Chitin‐Specific Binding Protein. Otolaryngology. 127(5). 377–383. 83 indexed citations

19.

Jones, Sherri M., et al.. (2001). Compound gravity receptor polarization vectors evidenced by linear vestibular evoked potentials. Hearing Research. 154(1-2). 54–61. 8 indexed citations

20.

Spivak, David A., et al.. (1999). A comparison of flexible and constrained haptens in eliciting antibody catalysts for paraoxon hydrolysis. Bioorganic & Medicinal Chemistry. 7(6). 1145–1150. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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