Masatoshi Kadoya
- Co-authors
- Masataka KohnoYutaka KawahitoTakahiro SenoAihiro YamamotoMasaki MiyamotoToshiaki ShichinoheYuma EbiharaRyo Takahashi
- Topics
- Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (6 papers)Systemic Sclerosis and Related Diseases (4 papers)Amyloidosis: Diagnosis, Treatment, Outcomes (3 papers)
- Journals
- SHILAP Revista de lepidopterologíaPLoS ONEScientific Reports
- Partner nations
- JapanUnited StatesSpain
In The Last Decade
Masatoshi Kadoya
29 papers receiving 269 citations
Peers
Comparison fields: 5 of 67
- Molecular Biology 121
- Pulmonary and Respiratory Medicine 60
- Immunology 56
- Oncology 35
- Surgery 29
Countries citing papers authored by Masatoshi Kadoya
This map shows the geographic impact of Masatoshi Kadoya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masatoshi Kadoya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masatoshi Kadoya more than expected).
Fields of papers citing papers by Masatoshi Kadoya
This network shows the impact of papers produced by Masatoshi Kadoya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masatoshi Kadoya. The network helps show where Masatoshi Kadoya may publish in the future.
Co-authorship network of co-authors of Masatoshi Kadoya
This figure shows the co-authorship network connecting the top 25 collaborators of Masatoshi Kadoya. A scholar is included among the top collaborators of Masatoshi Kadoya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masatoshi Kadoya. Masatoshi Kadoya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 1 | |
| 4 | 3 | |
| 5 | 4 | |
| 6 | Pigment epithelium-derived factor inhibits the growth of human esophageal squamous cell carcinoma by suppressing neovascularization. | 2 |
| 7 | 20 | |
| 8 | 7 | |
| 9 | 11 | |
| 10 | 8 | |
| 11 | 9 | |
| 12 | 10 | |
| 13 | 20 | |
| 14 | 2 | |
| 15 | 4 | |
| 16 | 3 | |
| 17 | 3 | |
| 18 | 54 | |
| 19 | 6 | |
| 20 | 2 |
About Masatoshi Kadoya
Masatoshi Kadoya is a scholar working on Rheumatology, Immunology and Allergy and Pulmonary and Respiratory Medicine, having authored 31 papers that have together received 277 indexed citations. Recurring topics across this work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (6 papers), Systemic Sclerosis and Related Diseases (4 papers) and Amyloidosis: Diagnosis, Treatment, Outcomes (3 papers). The work is most often cited by research in Immunology (56 citations), Pulmonary and Respiratory Medicine (60 citations) and Molecular Biology (121 citations). Masatoshi Kadoya has collaborated with scholars based in Japan, United States and Spain. Frequent co-authors include Masataka Kohno, Yutaka Kawahito, Takahiro Seno, Aihiro Yamamoto, Masaki Miyamoto, Toshiaki Shichinohe, Yuma Ebihara, Ryo Takahashi, Satoshi Kondo and Masahide Hamaguchi. Their work appears in journals such as SHILAP Revista de lepidopterología, PLoS ONE and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.