Masato Ohtsuka
- Molecular Biology top 5%
- Genetics top 2%
- Immunology
- Cell Biology top 10%
- Oncology
- Co-authors
- Channabasavaiah B. GurumurthyMasahiro SatoHiromi MiuraRolen M. QuadrosShingo NakamuraHidetoshi InokoSatoshi WatanabeGou Takahashi
- Topics
- CRISPR and Genetic Engineering (59 papers)Animal Genetics and Reproduction (45 papers)Pluripotent Stem Cells Research (25 papers)
- Partner nations
- JapanUnited StatesAustralia
In The Last Decade
Masato Ohtsuka
105 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 105
- Molecular Biology 1.8k
- Genetics 915
- Immunology 169
- Cell Biology 129
- Oncology 120
Countries citing papers authored by Masato Ohtsuka
This map shows the geographic impact of Masato Ohtsuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masato Ohtsuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masato Ohtsuka more than expected).
Fields of papers citing papers by Masato Ohtsuka
This network shows the impact of papers produced by Masato Ohtsuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masato Ohtsuka. The network helps show where Masato Ohtsuka may publish in the future.
Co-authorship network of co-authors of Masato Ohtsuka
This figure shows the co-authorship network connecting the top 25 collaborators of Masato Ohtsuka. A scholar is included among the top collaborators of Masato Ohtsuka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masato Ohtsuka. Masato Ohtsuka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 3 | |
| 4 | 1 | |
| 5 | 13 | |
| 6 | 13 | |
| 7 | 79 | |
| 8 | 6 | |
| 9 | 92 | |
| 10 | 6 | |
| 11 | 13 | |
| 12 | 6 | |
| 13 | 6 | |
| 14 | 8 | |
| 15 | 1 | |
| 16 | 15 | |
| 17 | Cre発現ベクターによるトランスフェクションヘの赤の緑へのまたは緑の赤への蛍光への発現の変化を可能にする,CRTEILおよびCETRIZ,Cre-loxPベースシステムの発達 | 40 |
| 18 | Positive correlation between oxidized low-density lipoprotein levels and the extent of coronary artery disease in patients with stable angina pectoris | 1 |
| 19 | 27 | |
| 20 | 44 |
About Masato Ohtsuka
Masato Ohtsuka is a scholar working on Aging, Genetics and Molecular Biology, having authored 108 papers that have together received 2.3k indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (59 papers), Animal Genetics and Reproduction (45 papers) and Pluripotent Stem Cells Research (25 papers). The work is most often cited by research in Aging (100 citations), Business and International Management (68 citations) and Genetics (915 citations). Masato Ohtsuka has collaborated with scholars based in Japan, United States and Australia. Frequent co-authors include Channabasavaiah B. Gurumurthy, Masahiro Sato, Hiromi Miura, Rolen M. Quadros, Shingo Nakamura, Hidetoshi Inoko, Satoshi Watanabe, Gou Takahashi, Kenta Wada and Shuji Takabayashi. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.