Masashi Ikuno

614 total citations
19 papers, 304 citations indexed

About

Masashi Ikuno is a scholar working on Neurology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Masashi Ikuno has authored 19 papers receiving a total of 304 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Neurology, 6 papers in Physiology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Masashi Ikuno's work include Parkinson's Disease Mechanisms and Treatments (13 papers), Neurological disorders and treatments (4 papers) and Botulinum Toxin and Related Neurological Disorders (3 papers). Masashi Ikuno is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (13 papers), Neurological disorders and treatments (4 papers) and Botulinum Toxin and Related Neurological Disorders (3 papers). Masashi Ikuno collaborates with scholars based in Japan and United States. Masashi Ikuno's co-authors include Ryōsuke Takahashi, Hodaka Yamakado, Tomoyuki Taguchi, Norihito Uemura, Jun Ueda, Masanori Sawamura, Ayumi Fukuoka, Maiko Uemura, Takahiro Yoneda and Laxmi Kumar Parajuli and has published in prestigious journals such as PLoS ONE, International Journal of Molecular Sciences and Human Molecular Genetics.

In The Last Decade

Masashi Ikuno

18 papers receiving 300 citations

Peers

Masashi Ikuno
Brittany Butler United States
Annika Kluge Germany
Elisa Menozzi United Kingdom
Yumei Liu China
Brittany Butler United States
Masashi Ikuno
Citations per year, relative to Masashi Ikuno Masashi Ikuno (= 1×) peers Brittany Butler

Countries citing papers authored by Masashi Ikuno

Since Specialization
Citations

This map shows the geographic impact of Masashi Ikuno's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masashi Ikuno with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masashi Ikuno more than expected).

Fields of papers citing papers by Masashi Ikuno

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masashi Ikuno. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masashi Ikuno. The network helps show where Masashi Ikuno may publish in the future.

Co-authorship network of co-authors of Masashi Ikuno

This figure shows the co-authorship network connecting the top 25 collaborators of Masashi Ikuno. A scholar is included among the top collaborators of Masashi Ikuno based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masashi Ikuno. Masashi Ikuno is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Ikuno, Masashi, et al.. (2025). Dialogue with the public: A catalyst for professional identity formation in medical students. PLoS ONE. 20(6). e0324506–e0324506.
2.
Sawamura, Masanori, Kenichi Kawano, Risa Sato, et al.. (2025). Impact of α-synuclein fibril structure on seeding activity in experimental models of Parkinson’s disease. npj Parkinson s Disease. 11(1). 224–224. 2 indexed citations
3.
Ueda, Jun, Norihito Uemura, Tomoyuki Taguchi, et al.. (2023). Ca2+–Calmodulin–Calcineurin Signaling Modulates α‐Synuclein Transmission. Movement Disorders. 38(6). 1056–1067. 9 indexed citations
4.
Okuda, Shinya, Takeo Nakayama, Norihito Uemura, et al.. (2022). Striatal-Inoculation of α-Synuclein Preformed Fibrils Aggravated the Phenotypes of REM Sleep without Atonia in A53T BAC-SNCA Transgenic Mice. International Journal of Molecular Sciences. 23(21). 13390–13390. 4 indexed citations
5.
Sawamura, Masanori, Hirotaka Onoe, Hideo Tsukada, et al.. (2022). Lewy Body Disease Primate Model with α‐Synuclein Propagation from the Olfactory Bulb. Movement Disorders. 37(10). 2033–2044. 18 indexed citations
6.
Okuda, Shinya, Norihito Uemura, Masanori Sawamura, et al.. (2021). Rapid Induction of Dopaminergic Neuron Loss Accompanied by Lewy Body-Like Inclusions in A53T BAC-SNCA Transgenic Mice. Neurotherapeutics. 19(1). 289–304. 8 indexed citations
7.
Ikuno, Masashi, Hodaka Yamakado, Yusuke Hatanaka, et al.. (2021). Mitochondrial dysfunction in a mouse model of prodromal Parkinson’s disease: A metabolomic analysis. Neuroscience Letters. 765. 136267–136267. 4 indexed citations
8.
Ueda, Jun, Norihito Uemura, Masanori Sawamura, et al.. (2021). Perampanel Inhibits α‐Synuclein Transmission in Parkinson's Disease Models. Movement Disorders. 36(7). 1554–1564. 24 indexed citations
9.
Uemura, Norihito, Jun Ueda, Toru Yoshihara, et al.. (2021). α‐Synuclein Spread from Olfactory Bulb Causes Hyposmia, Anxiety, and Memory Loss in BAC‐SNCA Mice. Movement Disorders. 36(9). 2036–2047. 43 indexed citations
10.
Parajuli, Laxmi Kumar, Soichiro Kakuta, Tomoyuki Taguchi, et al.. (2020). Developmental Changes in Dendritic Spine Morphology in the Striatum and Their Alteration in an A53T α-Synuclein Transgenic Mouse Model of Parkinson’s Disease. eNeuro. 7(4). ENEURO.0072–20.2020. 19 indexed citations
11.
Taguchi, Tomoyuki, Masashi Ikuno, Hodaka Yamakado, & Ryōsuke Takahashi. (2020). Animal Model for Prodromal Parkinson’s Disease. International Journal of Molecular Sciences. 21(6). 1961–1961. 45 indexed citations
12.
Takeuchi, Masato, Masashi Ikuno, Yohei Kawasaki, et al.. (2019). Real-world pharmacological treatment patterns of patients with young-onset Parkinson’s disease in Japan: a medical claims database analysis. Journal of Neurology. 266(8). 1944–1952. 17 indexed citations
13.
Ikuno, Masashi, Hodaka Yamakado, Hisako Akiyama, et al.. (2019). GBA haploinsufficiency accelerates alpha-synuclein pathology with altered lipid metabolism in a prodromal model of Parkinson’s disease. Human Molecular Genetics. 28(11). 1894–1904. 44 indexed citations
14.
Fujita, Kyota, Hidenori Homma, Kanoh Kondo, et al.. (2018). Ser46-Phosphorylated MARCKS Is a Marker of Neurite Degeneration at the Pre-aggregation Stage in PD/DLB Pathology. eNeuro. 5(4). ENEURO.0217–18.2018. 4 indexed citations
15.
Kobayashi, Atsushi, Takahiro Yoneda, Masanori Yoshikawa, et al.. (2000). The Relation of Fat-free Mass to Maximum Exercise Performance in Patients with Chronic Obstructive Pulmonary Disease. Lung. 178(2). 119–127. 32 indexed citations
16.
Okamura, Hideo, K Tsukaguchi, Masashi Ikuno, et al.. (1999). [A study of factors relating to aggravation of patients with pulmonary Mycobacterium avium complex disease--with special reference to malnutrition].. PubMed. 74(4). 341–5. 1 indexed citations
17.
Takenaka, Hideaki, T Yoneda, Atsushi Kobayashi, et al.. (1998). [Bioelectrical impedance analysis of body composition in patients with pulmonary emphysema].. PubMed. 36(8). 653–8. 4 indexed citations
18.
Yoshikawa, Masanori, Atsushi Kobayashi, Akihiro Fu, et al.. (1997). [Exercise performance and body composition in patients with chronic obstructive pulmonary disease].. PubMed. 35(5). 518–23. 2 indexed citations
19.
Tsukaguchi, K, Hideo Okamura, Masashi Ikuno, et al.. (1997). [The relation between diabetes mellitus and IFN-gamma, IL-12 and IL-10 productions by CD4+ alpha beta T cells and monocytes in patients with pulmonary tuberculosis].. PubMed. 72(11). 617–22. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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