Mary Kate McBrayer

6.7k citations
5 papers · 560 indexed · h-index 5
  • Physiology top 5%
    • Calcium signaling and nucleotide metabolism 2
  • Cell Biology top 10%
    • Cellular transport and secretion 3
    • Microtubule and mitosis dynamics 1
  • Physiology top 10%
    • Calcium signaling and nucleotide metabolism 2
    • Cancer, Hypoxia, and Metabolism 1
    • Porphyrin Metabolism and Disorders 1
    • Protein Structure and Dynamics 1
    • Autophagy in Disease and Therapy 2
    • Cancer-related Molecular Pathways 1

Mary Kate McBrayer

5 papers receiving 559 citations

Peers

Mary Kate McBrayer
Comparison fields: 5 of 70
  • Physiology 101
  • Cell Biology 168
  • Physiology 198
  • Cancer Research 67
  • Molecular Biology 304
Replace Hristina Ivanova with:
Hristina Ivanova Belgium
Ulises Ahumada-Castro Chile
Galdo Bustos Chile
Maria Chiara Meschini Italy
Roberta Peruzzo Italy
Michael A. Kalwat United States
Cristina Ploia Italy
Merle Hillebrand Germany
Limor Avrahami Israel
Jae-Woong Chang United States
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Citations per field
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Citations per year

Countries citing papers authored by Mary Kate McBrayer

Since Specialization
Citations

This map shows the geographic impact of Mary Kate McBrayer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mary Kate McBrayer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mary Kate McBrayer more than expected).

Fields of papers citing papers by Mary Kate McBrayer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mary Kate McBrayer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mary Kate McBrayer. The network helps show where Mary Kate McBrayer may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mary Kate McBrayer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mary Kate McBrayer Line = papers co-authored together Mary Kate McBrayer links everyone, so they are left out of the graph.

All Works

5 of 5 papers shown

About Mary Kate McBrayer

Mary Kate McBrayer is a scholar working on Physiology, Cell Biology and Cancer Research, having authored 5 papers that have together received 560 indexed citations. Recurring topics across this work include Cellular transport and secretion (3 papers), Autophagy in Disease and Therapy (2 papers), Calcium signaling and nucleotide metabolism (2 papers), Porphyrin Metabolism and Disorders (1 paper), Protein Structure and Dynamics (1 paper), Cancer-related Molecular Pathways (1 paper), Microtubule and mitosis dynamics (1 paper) and Cancer, Hypoxia, and Metabolism (1 paper). The work is most often cited by research in Physiology (101 citations), Cell Biology (168 citations) and Physiology (198 citations). Mary Kate McBrayer has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Timothy E. McGraw, Anja Zeigerer, Ju‐Hyun Lee, Panaiyur S. Mohan, Asok Kumar, Ralph A. Nixon, Devin M. Wolfe, Claire H. Mitchell, Pearl P.Y. Lie and Erin E. Coffey. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Molecular Biology and Molecular Biology of the Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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