Galdo Bustos

993 total citations
10 papers, 445 citations indexed

About

Galdo Bustos is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Galdo Bustos has authored 10 papers receiving a total of 445 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 4 papers in Cell Biology and 4 papers in Cancer Research. Recurrent topics in Galdo Bustos's work include Mitochondrial Function and Pathology (7 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Galdo Bustos is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Galdo Bustos collaborates with scholars based in Chile, United States and France. Galdo Bustos's co-authors include César Cárdenas, Alenka Lovy, Jordi Molgó, Pablo Cruz, Fabián Jaña, Félix A. Urra, J. Kevin Foskett, Ulises Ahumada-Castro, Natalia Smith‐Cortinez and Marioly Müller and has published in prestigious journals such as Scientific Reports, International Journal of Molecular Sciences and Cell Reports.

In The Last Decade

Galdo Bustos

9 papers receiving 443 citations

Peers

Galdo Bustos
Galdo Bustos
Citations per year, relative to Galdo Bustos Galdo Bustos (= 1×) peers Luigi Sbano

Countries citing papers authored by Galdo Bustos

Since Specialization
Citations

This map shows the geographic impact of Galdo Bustos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Galdo Bustos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Galdo Bustos more than expected).

Fields of papers citing papers by Galdo Bustos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Galdo Bustos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Galdo Bustos. The network helps show where Galdo Bustos may publish in the future.

Co-authorship network of co-authors of Galdo Bustos

This figure shows the co-authorship network connecting the top 25 collaborators of Galdo Bustos. A scholar is included among the top collaborators of Galdo Bustos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Galdo Bustos. Galdo Bustos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Bustos, Galdo, Ulises Ahumada-Castro, Eduardo Silva-Pavez, et al.. (2024). The IP3R inhibitor desmethylxestospongin B reduces tumor cell migration, invasion and metastasis by impairing lysosome acidification and β1-integrin recycling. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1871(1). 167557–167557.
2.
Silva-Pavez, Eduardo, Ulises Ahumada-Castro, Galdo Bustos, et al.. (2024). Mitochondrial division inhibitor (mdivi-1) induces extracellular matrix (ECM)-detachment of viable breast cancer cells by a DRP1-independent mechanism. Scientific Reports. 14(1). 14178–14178. 4 indexed citations
3.
Ahumada-Castro, Ulises, et al.. (2021). In the Right Place at the Right Time: Regulation of Cell Metabolism by IP3R-Mediated Inter-Organelle Ca2+ Fluxes. Frontiers in Cell and Developmental Biology. 9. 629522–629522. 37 indexed citations
4.
Bustos, Galdo, Ulises Ahumada-Castro, Eduardo Silva-Pavez, et al.. (2021). The ER-mitochondria Ca2+ signaling in cancer progression: Fueling the monster. International review of cell and molecular biology. 363. 49–121. 16 indexed citations
5.
Cruz, Pablo, Ulises Ahumada-Castro, Galdo Bustos, et al.. (2021). Inhibition of InsP3R with Xestospongin B Reduces Mitochondrial Respiration and Induces Selective Cell Death in T Cell Acute Lymphoblastic Leukemia Cells. International Journal of Molecular Sciences. 22(2). 651–651. 14 indexed citations
6.
Cárdenas, César, Alenka Lovy, Eduardo Silva-Pavez, et al.. (2020). Cancer cells with defective oxidative phosphorylation require endoplasmic reticulum–to–mitochondria Ca 2+ transfer for survival. Science Signaling. 13(640). 51 indexed citations
7.
Lovy, Alenka, Ulises Ahumada-Castro, Galdo Bustos, et al.. (2020). Concerted Action of AMPK and Sirtuin-1 Induces Mitochondrial Fragmentation Upon Inhibition of Ca2+ Transfer to Mitochondria. Frontiers in Cell and Developmental Biology. 8. 378–378. 27 indexed citations
8.
Jaña, Fabián, Galdo Bustos, Pablo Cruz, et al.. (2019). Complex I and II are required for normal mitochondrial Ca2+ homeostasis. Mitochondrion. 49. 73–82. 23 indexed citations
9.
Bustos, Galdo, Pablo Cruz, Alenka Lovy, & César Cárdenas. (2017). Endoplasmic Reticulum–Mitochondria Calcium Communication and the Regulation of Mitochondrial Metabolism in Cancer: A Novel Potential Target. Frontiers in Oncology. 7. 199–199. 64 indexed citations
10.
Cárdenas, César, Marioly Müller, Andrew S. McNeal, et al.. (2016). Selective Vulnerability of Cancer Cells by Inhibition of Ca2+ Transfer from Endoplasmic Reticulum to Mitochondria. Cell Reports. 14(10). 2313–2324. 209 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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