Martin Lackinger
Impact in
- Behavioral Neuroscience top 10%
- Stress Responses and Cortisol
Papers in
-
- Congenital heart defects research 2
- RNA modifications and cancer 2
- RNA Research and Splicing 2
- RNA regulation and disease 1
-
- MicroRNA in disease regulation 2
- Co-authors
- Gerhard Schratt (4 shared papers)Silvia Bicker (3 shared papers)Rainer K.W. Schwarting (3 shared papers)Markus Wöhr (3 shared papers)Juan C. Brenes (1 shared paper)Günter U. Höglinger (1 shared paper)Christoph Dieterich (2 shared papers)Roberto Fiore (2 shared papers)
- Journals
- eLife (2 papers)The Journal of Comparative Neurology (1 paper)Nature Neuroscience (1 paper)EMBO Reports (1 paper)Cellular and Molecular Life Sciences (1 paper)
- Partner nations
- GermanyUnited KingdomUnited States
In The Last Decade
Martin Lackinger
5 papers receiving 319 citations
Peers
Comparison fields: 5 of 51
- Behavioral Neuroscience 39
- Developmental Neuroscience 31
- Cancer Research 103
- Biological Psychiatry 8
- Pharmacy 14
Countries citing papers authored by Martin Lackinger
This map shows the geographic impact of Martin Lackinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin Lackinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin Lackinger more than expected).
Fields of papers citing papers by Martin Lackinger
This network shows the impact of papers produced by Martin Lackinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin Lackinger. The network helps show where Martin Lackinger may publish in the future.
Co-authors
The 21 scholars most cited alongside Martin Lackinger, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 120 | |
| 2 | 2015 | 84 | |
| 3 | 2014 | 81 | |
| 4 | 2018 | 34 | |
| 5 | 2024 | 2 | |
| 6 | 2025 | 0 |
About Martin Lackinger
Martin Lackinger is a scholar working on Molecular Biology, Cancer Research, Social Psychology, Behavioral Neuroscience and Genetics, having authored 6 papers that have together received 321 indexed citations. Recurring topics across this work include Congenital heart defects research (2 papers), MicroRNA in disease regulation (2 papers), RNA modifications and cancer (2 papers), RNA Research and Splicing (2 papers), Neurogenesis and neuroplasticity mechanisms (1 paper), Genetics and Neurodevelopmental Disorders (1 paper), Neuroendocrine regulation and behavior (1 paper) and RNA regulation and disease (1 paper). The work is most often cited by research in Behavioral Neuroscience (39 citations), Developmental Neuroscience (31 citations), Cancer Research (103 citations), Biological Psychiatry (8 citations) and Pharmacy (14 citations). Martin Lackinger has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Gerhard Schratt, Silvia Bicker, Rainer K.W. Schwarting, Markus Wöhr, Juan C. Brenes, Günter U. Höglinger, Christoph Dieterich, Roberto Fiore, Franziska Metge and Ayla Aksoy‐Aksel. Their work appears in journals such as eLife, The Journal of Comparative Neurology, Nature Neuroscience, EMBO Reports and Cellular and Molecular Life Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.