Marta Soler
- Cancer Research top 5%
- Cancer-related molecular mechanisms research 9
- MicroRNA in disease regulation 4
- Molecular Biology top 10%
- RNA modifications and cancer 9
- RNA Research and Splicing 4
- Epigenetics and DNA Methylation 2
- Protein Degradation and Inhibitors 2
- Histone Deacetylase Inhibitors Research 2
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- Genetics and Neurodevelopmental Disorders 4
- Co-authors
- Manel EstellerSònia GuilAnna PortelaAlberto VillanuevaAntonio GómezRaquel Boqué-SastreCristina Oliveira-MateosCátia Moutinho
- Partner nations
- SpainUnited StatesNetherlands
In The Last Decade
Marta Soler
20 papers receiving 921 citations
Peers
Comparison fields: 5 of 74
- Cancer Research 476
- Molecular Biology 745
- Cell Biology 70
- Endocrinology 19
- Genetics 84
Countries citing papers authored by Marta Soler
This map shows the geographic impact of Marta Soler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marta Soler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marta Soler more than expected).
Fields of papers citing papers by Marta Soler
This network shows the impact of papers produced by Marta Soler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marta Soler. The network helps show where Marta Soler may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Marta Soler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 2 | |
| 3 | 2024 | 2 | |
| 4 | 2023 | 38 | |
| 5 | 2021 | 4 | |
| 6 | 2021 | 9 | |
| 7 | 2019 | 45 | |
| 8 | 2018 | 42 | |
| 9 | 2018 | 34 | |
| 10 | 2017 | 8 | |
| 11 | 2017 | 4 | |
| 12 | 2017 | 16 | |
| 13 | 2014 | 125 | |
| 14 | 2014 | 25 | |
| 15 | 2012 | 140 | |
| 16 | 2011 | 96 | |
| 17 | 2009 | 82 | |
| 18 | CYP86A33-targeted gene silencing in potato tuber alters suberin composition, distorts suberin lamellae, and impairs the periderm's water barrier function | 2009 | 3 |
| 19 | 2000 | 13 | |
| 20 | 1998 | 59 |
About Marta Soler
Marta Soler is a scholar working on Cancer Research, General Social Sciences and Molecular Biology, having authored 22 papers that have together received 928 indexed citations. Recurring topics across this work include RNA modifications and cancer (9 papers), Cancer-related molecular mechanisms research (9 papers), MicroRNA in disease regulation (4 papers), Genetics and Neurodevelopmental Disorders (4 papers), RNA Research and Splicing (4 papers), Epigenetics and DNA Methylation (2 papers), Protein Degradation and Inhibitors (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). The work is most often cited by research in Cancer Research (476 citations), Molecular Biology (745 citations) and Cell Biology (70 citations). Marta Soler has collaborated with scholars based in Spain, United States and Netherlands. Frequent co-authors include Manel Esteller, Sònia Guil, Anna Portela, Alberto Villanueva, Antonio Gómez, Raquel Boqué-Sastre, Cristina Oliveira-Mateos, Cátia Moutinho, Sergi Sayols and Jordi Carrère. Their work appears in journals such as Oncotarget, Molecular Oncology, Journal of Hepatology, Nature Communications and Cell Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.