Mark T. Lin
- Immunology top 10%
- Infectious Diseases top 5%
- Animal Science and Zoology top 2%
- Molecular Biology
- Epidemiology
- Co-authors
- Stephen A. StohlmanDavid R. HintonCornelia C. BergmannTimothy E. AlbertsonBeatriz ParraNorman W. MartenJ. DanielJosé M. Mato
- Topics
- Animal Virus Infections Studies (6 papers)Immune Cell Function and Interaction (3 papers)Viral gastroenteritis research and epidemiology (2 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Clinical OncologyThe Journal of Immunology
- Partner nations
- United StatesSpainFrance
In The Last Decade
Mark T. Lin
17 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 87
- Immunology 398
- Infectious Diseases 254
- Animal Science and Zoology 221
- Molecular Biology 216
- Epidemiology 194
Countries citing papers authored by Mark T. Lin
This map shows the geographic impact of Mark T. Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark T. Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark T. Lin more than expected).
Fields of papers citing papers by Mark T. Lin
This network shows the impact of papers produced by Mark T. Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark T. Lin. The network helps show where Mark T. Lin may publish in the future.
Co-authorship network of co-authors of Mark T. Lin
This figure shows the co-authorship network connecting the top 25 collaborators of Mark T. Lin. A scholar is included among the top collaborators of Mark T. Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark T. Lin. Mark T. Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 13 | |
| 3 | 39 | |
| 4 | 19 | |
| 5 | 71 | |
| 6 | 98 | |
| 7 | 154 | |
| 8 | 48 | |
| 9 | 56 | |
| 10 | 183 | |
| 11 | 80 | |
| 12 | 51 | |
| 13 | 10 | |
| 14 | 5 | |
| 15 | 134 | |
| 16 | 69 | |
| 17 | Immune regulation of coronavirus-induced demyelinating encephalomyelitis. | 1 |
About Mark T. Lin
Mark T. Lin is a scholar working on Animal Science and Zoology, Immunology and Hepatology, having authored 17 papers that have together received 1.0k indexed citations. Recurring topics across this work include Animal Virus Infections Studies (6 papers), Immune Cell Function and Interaction (3 papers) and Viral gastroenteritis research and epidemiology (2 papers). The work is most often cited by research in Animal Science and Zoology (221 citations), Immunology (398 citations) and Developmental Neuroscience (67 citations). Mark T. Lin has collaborated with scholars based in United States, Spain and France. Frequent co-authors include Stephen A. Stohlman, David R. Hinton, Cornelia C. Bergmann, Timothy E. Albertson, Beatriz Parra, Norman W. Marten, J. Daniel, José M. Mato, Shelly C. Lu and Roel C. van der Veen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Oncology and The Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.