Mark Aspinall-O’Dea
Impact in
- Hematology top 10%
- Chronic Myeloid Leukemia Treatments
- Acute Myeloid Leukemia Research
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- Photosynthetic Processes and Mechanisms
- FOXO transcription factor regulation
Papers in
-
- Chronic Myeloid Leukemia Treatments 2
- Oncology 2
- Chemokine receptors and signaling 1
- Pancreatic and Hepatic Oncology Research 1
- Co-authors
- Eithne Costello (3 shared papers)Andrew A. Pascal (1 shared paper)Peter Horton (1 shared paper)Alexander V. Ruban (1 shared paper)Mark Wentworth (1 shared paper)Bruno Robert (1 shared paper)Anthony David Whetton (7 shared papers)Andrew Pierce (5 shared papers)
- Journals
- Stem Cells (1 paper)Blood (1 paper)Toxicologic Pathology (1 paper)Journal of Proteomics (1 paper)PLoS ONE (1 paper)
- Partner nations
- United KingdomUnited StatesSweden
In The Last Decade
Mark Aspinall-O’Dea
12 papers receiving 396 citations
Peers
Comparison fields: 5 of 35
- Hematology 59
- Molecular Biology 140
- Oncology 44
- Spectroscopy 23
- Genetics 6
Countries citing papers authored by Mark Aspinall-O’Dea
This map shows the geographic impact of Mark Aspinall-O’Dea's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Aspinall-O’Dea with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Aspinall-O’Dea more than expected).
Fields of papers citing papers by Mark Aspinall-O’Dea
This network shows the impact of papers produced by Mark Aspinall-O’Dea. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Aspinall-O’Dea. The network helps show where Mark Aspinall-O’Dea may publish in the future.
Co-authors
The 25 scholars most cited alongside Mark Aspinall-O’Dea, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2002 | 113 | |
| 2 | 2014 | 81 | |
| 3 | 2009 | 44 | |
| 4 | 2018 | 37 | |
| 5 | 2009 | 29 | |
| 6 | 2014 | 20 | |
| 7 | 2016 | 17 | |
| 8 | 2013 | 16 | |
| 9 | 2007 | 15 | |
| 10 | 2012 | 15 | |
| 11 | 2014 | 12 | |
| 12 | 2025 | 2 |
About Mark Aspinall-O’Dea
Mark Aspinall-O’Dea is a scholar working on Hematology, Oncology, Spectroscopy, Molecular Biology and Nuclear Energy and Engineering, having authored 12 papers that have together received 401 indexed citations. Recurring topics across this work include Chronic Myeloid Leukemia Treatments (2 papers), Advanced Proteomics Techniques and Applications (1 paper), Photosynthetic Processes and Mechanisms (1 paper), Chemokine receptors and signaling (1 paper), FOXO transcription factor regulation (1 paper) and Pancreatic and Hepatic Oncology Research (1 paper). The work is most often cited by research in Hematology (59 citations), Molecular Biology (140 citations), Oncology (44 citations), Spectroscopy (23 citations) and Genetics (6 citations). Mark Aspinall-O’Dea has collaborated with scholars based in United Kingdom, United States and Sweden. Frequent co-authors include Eithne Costello, Andrew A. Pascal, Peter Horton, Alexander V. Ruban, Mark Wentworth, Bruno Robert, Anthony David Whetton, Andrew Pierce, Tessa L. Holyoake and Francesca Pellicano. Their work appears in journals such as Stem Cells, Blood, Toxicologic Pathology, Journal of Proteomics and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.