Elaine Spooncer
- Molecular Biology top 5%
- Hematology top 0.5%
- Immunology top 2%
- Oncology top 5%
- Cell Biology top 2%
- Co-authors
- T. M. DexterDale R. TaylorChristopher A. SmithGwyn T. WilliamsAnthony D. WhettonRuth RobertsFrank H. BloomfieldJ. Gallagher
- Topics
- Hematopoietic Stem Cell Transplantation (23 papers)Virus-based gene therapy research (14 papers)Acute Myeloid Leukemia Research (9 papers)
- Partner nations
- United KingdomGermanyUnited States
In The Last Decade
Elaine Spooncer
76 papers receiving 4.6k citations
Hit Papers
Peers
Comparison fields: 5 of 134
- Molecular Biology 2.4k
- Hematology 1.4k
- Immunology 1.3k
- Oncology 789
- Cell Biology 751
Countries citing papers authored by Elaine Spooncer
This map shows the geographic impact of Elaine Spooncer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elaine Spooncer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elaine Spooncer more than expected).
Fields of papers citing papers by Elaine Spooncer
This network shows the impact of papers produced by Elaine Spooncer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elaine Spooncer. The network helps show where Elaine Spooncer may publish in the future.
Co-authorship network of co-authors of Elaine Spooncer
This figure shows the co-authorship network connecting the top 25 collaborators of Elaine Spooncer. A scholar is included among the top collaborators of Elaine Spooncer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elaine Spooncer. Elaine Spooncer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 16 | |
| 2 | 15 | |
| 3 | 9 | |
| 4 | 22 | |
| 5 | 143 | |
| 6 | 1 | |
| 7 | 5 | |
| 8 | 58 | |
| 9 | 42 | |
| 10 | 15 | |
| 11 | 37 | |
| 12 | 80 | |
| 13 | 60 | |
| 14 | Haemopoietic colony stimulating factors promote cell survival by suppressing apoptosisbreakdown → | 826 |
| 15 | 1 | |
| 16 | 23 | |
| 17 | Heparan sulphate bound growth factors: a mechanism for stromal cell mediated haemopoiesisbreakdown → | 693 |
| 18 | 182 | |
| 19 | 4 | |
| 20 | 52 |
About Elaine Spooncer
Elaine Spooncer is a scholar working on Hematology, Genetics and Immunology, having authored 77 papers that have together received 4.7k indexed citations. Recurring topics across this work include Hematopoietic Stem Cell Transplantation (23 papers), Virus-based gene therapy research (14 papers) and Acute Myeloid Leukemia Research (9 papers). The work is most often cited by research in Hematology (1.4k citations), Immunology and Allergy (418 citations) and Immunology (1.3k citations). Elaine Spooncer has collaborated with scholars based in United Kingdom, Germany and United States. Frequent co-authors include T. M. Dexter, Dale R. Taylor, Christopher A. Smith, Gwyn T. Williams, Anthony D. Whetton, Ruth Roberts, Frank H. Bloomfield, J. Gallagher, T. M. Dexter and Andrew Pierce. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.