Maria J. Carnicer

867 citations
18 papers · 611 indexed · h-index 11

Maria J. Carnicer

18 papers receiving 603 citations

Peers

Maria J. Carnicer
Comparison fields: 5 of 61
  • Hematology 162
  • Immunology 148
  • Oncology 183
  • Genetics 62
  • Molecular Biology 333
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Cathy M. Price United Kingdom
Makiko Takizawa Japan
Jayakumar Vadakekolathu United Kingdom
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Citations per field
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Citations per year

Countries citing papers authored by Maria J. Carnicer

Since Specialization
Citations

This map shows the geographic impact of Maria J. Carnicer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria J. Carnicer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria J. Carnicer more than expected).

Fields of papers citing papers by Maria J. Carnicer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maria J. Carnicer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria J. Carnicer. The network helps show where Maria J. Carnicer may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Maria J. Carnicer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Maria J. Carnicer Line = papers co-authored together Maria J. Carnicer links everyone, so they are left out of the graph.

All Works

18 of 18 papers shown
#Work
1 2015146
2 201055
3 200820
4 20084
5 20084
6 200720
7 200619
8 200510
9 200510
10 200435
11 200314
12
Id4 is deregulated by a t(6;14)(p22;q32) chromosomal translocation in a B-cell lineage acute lymphoblastic leukemia.
200328
13
Interstitial deletions at the long arm of chromosome 13 may be as common as monosomies in multiple myeloma. A genotypic study.
20027
14 20024
15 200110
16 20011
17
Interleukin-3 receptor alpha chain (CD123) is widely expressed in hematologic malignancies.
2001222
18
Leukemic relapse as T-acute lymphoblastic leukemia in a patient with acute myeloid leukemia and a minor T-cell clone at diagnosis.
20002

About Maria J. Carnicer

Maria J. Carnicer is a scholar working on Hematology, Pathology and Forensic Medicine and Immunology, having authored 18 papers that have together received 611 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (11 papers), Retinoids in leukemia and cellular processes (4 papers), Histone Deacetylase Inhibitors Research (3 papers), Lymphoma Diagnosis and Treatment (3 papers), Immune Cell Function and Interaction (3 papers), Protein Degradation and Inhibitors (3 papers), Viral-associated cancers and disorders (2 papers) and Epigenetics and DNA Methylation (2 papers). The work is most often cited by research in Hematology (162 citations), Immunology (148 citations) and Oncology (183 citations). Maria J. Carnicer has collaborated with scholars based in Spain, Italy and United Kingdom. Frequent co-authors include Jorge Sierra, Josep Nomdedéu, Salut Brunet, Mar Bellido, Luz Muñoz, Olga López, Adriana Lasa, Anna Aventı́n, Camino Estivill and Radhika Kandaswamy. Their work appears in journals such as Nature Communications, Blood and The American Journal of Surgical Pathology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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