Adriana Lasa
- Genetics top 10%
- BRCA gene mutations in cancer 6
- Chronic Lymphocytic Leukemia Research 4
- Hematology top 10%
- Acute Myeloid Leukemia Research 10
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- Muscle Physiology and Disorders 10
- DNA Repair Mechanisms 5
- Cell Biology top 10%
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- Genetic Neurodegenerative Diseases 4
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- Cancer Genomics and Diagnostics 6
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- Genetic factors in colorectal cancer 4
- Co-authors
- Montserrat BaigetP. GallanoJosep NomdedéuEduard GallardoIsabel IllaJorge SierraChie MatsudaJaume Palmer
- Cited by
- GeneticsHematologyMolecular Biology
- Partner nations
- SpainFranceUnited States
In The Last Decade
Adriana Lasa
44 papers receiving 853 citations
Peers
Comparison fields: 5 of 72
- Genetics 126
- Hematology 117
- Molecular Biology 661
- Cell Biology 127
- Cellular and Molecular Neuroscience 140
Countries citing papers authored by Adriana Lasa
This map shows the geographic impact of Adriana Lasa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adriana Lasa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adriana Lasa more than expected).
Fields of papers citing papers by Adriana Lasa
This network shows the impact of papers produced by Adriana Lasa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adriana Lasa. The network helps show where Adriana Lasa may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Adriana Lasa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 0 | |
| 2 | 2022 | 3 | |
| 3 | 2020 | 21 | |
| 4 | 2013 | 14 | |
| 5 | 2013 | 14 | |
| 6 | 2013 | 17 | |
| 7 | 2011 | 7 | |
| 8 | 2010 | 11 | |
| 9 | 2009 | 17 | |
| 10 | 2009 | 8 | |
| 11 | 2008 | 20 | |
| 12 | 2008 | 4 | |
| 13 | 2004 | 35 | |
| 14 | 2003 | 14 | |
| 15 | 2001 | 11 | |
| 16 | 1999 | 18 | |
| 17 | 1998 | 15 | |
| 18 | 1998 | 13 | |
| 19 | 1997 | 1 | |
| 20 | 1996 | 2 |
About Adriana Lasa
Adriana Lasa is a scholar working on Hematology, Cancer Research, Molecular Biology, Pathology and Forensic Medicine and Genetics, having authored 46 papers that have together received 866 indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (10 papers), Acute Myeloid Leukemia Research (10 papers), BRCA gene mutations in cancer (6 papers), Cancer Genomics and Diagnostics (6 papers), DNA Repair Mechanisms (5 papers), Chronic Lymphocytic Leukemia Research (4 papers), Genetic Neurodegenerative Diseases (4 papers) and Genetic factors in colorectal cancer (4 papers). The work is most often cited by research in Genetics (126 citations), Hematology (117 citations), Molecular Biology (661 citations), Cell Biology (127 citations) and Cellular and Molecular Neuroscience (140 citations). Adriana Lasa has collaborated with scholars based in Spain, France and United States. Frequent co-authors include Montserrat Baiget, P. Gallano, Josep Nomdedéu, Eduard Gallardo, Isabel Illa, Jorge Sierra, Chie Matsuda, Jaume Palmer, Salut Brunet and Carmen Serrano. Their work appears in journals such as Human Mutation, Leukemia Research, Annals of Hematology, Breast Cancer Research and Treatment and European Journal of Human Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.