Máikel L. Colli

4.3k total citations · 1 hit paper
39 papers, 2.8k citations indexed

About

Máikel L. Colli is a scholar working on Surgery, Genetics and Molecular Biology. According to data from OpenAlex, Máikel L. Colli has authored 39 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Surgery, 24 papers in Genetics and 12 papers in Molecular Biology. Recurrent topics in Máikel L. Colli's work include Pancreatic function and diabetes (26 papers), Diabetes and associated disorders (23 papers) and Immune Cell Function and Interaction (7 papers). Máikel L. Colli is often cited by papers focused on Pancreatic function and diabetes (26 papers), Diabetes and associated disorders (23 papers) and Immune Cell Function and Interaction (7 papers). Máikel L. Colli collaborates with scholars based in Belgium, Italy and United States. Máikel L. Colli's co-authors include Décio L. Eizirik, Fernanda Ortis, Piero Marchetti, Lorella Marselli, Fabrice Moore, Miriam Cnop, Esteban N. Gurzov, Daniel A. Cunha, Mariana Igoillo‐Esteve and Izortze Santín and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Máikel L. Colli

37 papers receiving 2.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The role of inflammation in insulitis and β-cell loss in type 1 diabetes

2009 799 citations Décio L. Eizirik, Máikel L. Colli et al. Nature Reviews Endocrinology profile →

Peers

Countries citing papers authored by Máikel L. Colli

Since Specialization

Citations

This map shows the geographic impact of Máikel L. Colli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Máikel L. Colli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Máikel L. Colli more than expected).

Fields of papers citing papers by Máikel L. Colli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Máikel L. Colli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Máikel L. Colli. The network helps show where Máikel L. Colli may publish in the future.

Co-authorship network of co-authors of Máikel L. Colli

This figure shows the co-authorship network connecting the top 25 collaborators of Máikel L. Colli. A scholar is included among the top collaborators of Máikel L. Colli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Máikel L. Colli. Máikel L. Colli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	AB0144 PHARMACOLOGICAL CHARACTERIZATION OF GLPG3667, A SELECTIVE TYK2 INHIBITOR, SUPPORTS DEVELOPMENT IN DERMATOMYOSITIS AND SYSTEMIC LUPUS ERYTHEMATOSUS 2023 ·Annals of the Rheumatic Diseases ·(unknown), Eric F. Morand, Roland Blanqué, Kenji F. Shoji, (unknown), Florence Marsais, Laetitia Furio, Máikel L. Colli, (unknown), (unknown), (unknown), (unknown), (unknown), (unknown), A. Van de Water, (unknown), Stephen E. Alves, (unknown), (unknown), René Galien	1
2	Inhibition of the type 1 diabetes candidate gene PTPN2 aggravates TNF-α-induced human beta cell dysfunction and death 2023 ·Diabetologia ·Arturo Roca-Rivada, (unknown), Máikel L. Colli, (unknown), Toshiaki Sawatani, Lorella Marselli, Miriam Cnop, Piero Marchetti, Décio L. Eizirik	21
3	Transcription and splicing regulation by NLRC5 shape the interferon response in human pancreatic β cells 2022 ·Science Advances ·(unknown), Maria Inês Alvelos, (unknown), Ângela Castela, Stéphane Demine, Máikel L. Colli, Anne Op De Beeck, Sofia Thomaidou, Lorella Marselli, Arnaud Zaldumbide, Piero Marchetti, Décio L. Eizirik	20
4	Pro-Inflammatory Cytokines Promote the Transcription of Circular RNAs in Human Pancreatic β Cells 2022 ·Non-Coding RNA ·Simranjeet Kaur, (unknown), Aashiq H. Mirza, Tina Fløyel, Reza Yarani, Máikel L. Colli, Jesper Johannesen, Joachim Størling, Décio L. Eizirik, Flemming Pociot	2
5	Gene expression signatures of target tissues in type 1 diabetes, lupus erythematosus, multiple sclerosis, and rheumatoid arthritis 2021 ·Science Advances ·(unknown), Máikel L. Colli, Mark J. Mamula, Carmella Evans‐Molina, Décio L. Eizirik	39
6	Peptides Derived From Insulin Granule Proteins Are Targeted by CD8+ T Cells Across MHC Class I Restrictions in Humans and NOD Mice 2020 ·Diabetes ·Marie Eliane Azoury, (unknown), Georgia Afonso, (unknown), Máikel L. Colli, (unknown), (unknown), (unknown), (unknown), Yann Verdier, Joëlle Vinh, Sheena Pinto, Søren Buus, Danièle Dubois‐Laforgue, Étienne Larger, Jean-Paul Beressi, Graziella Bruno, Décio L. Eizirik, Sylvaine You, Roberto Mallone	33
7	The RNA-binding profile of the splicing factor SRSF6 in immortalized human pancreatic β-cells 2020 ·Life Science Alliance ·Maria Inês Alvelos, (unknown), F.X. Reymond Sutandy, Jonàs Juan‐Mateu, Máikel L. Colli, Anke Busch, Miguel Lopes, Ângela Castela, Annemieke Aartsma‐Rus, Julian König, Kathi Zarnack, Décio L. Eizirik	14
8	Molecular Footprints of the Immune Assault on Pancreatic Beta Cells in Type 1 Diabetes 2020 ·Frontiers in Endocrinology ·Máikel L. Colli, (unknown), Décio L. Eizirik	21
9	Revisiting the role of inflammation in the loss of pancreatic β-cells in T1DM 2020 ·Nature Reviews Endocrinology ·Décio L. Eizirik, Máikel L. Colli	21
10	The impact of proinflammatory cytokines on the β-cell regulatory landscape provides insights into the genetics of type 1 diabetes 2019 ·Nature Genetics ·Mireia Ramos-Rodríguez, Helena Raurell‐Vila, Máikel L. Colli, Maria Inês Alvelos, (unknown), Jonàs Juan‐Mateu, (unknown), Jean‐Valéry Turatsinze, Ernesto Nakayasu, Bobbie‐Jo Webb‐Robertson, Jamie Inshaw, Piero Marchetti, Lorenzo Piemonti, Manel Esteller, John A. Todd, Thomas Metz, Décio L. Eizirik, Lorenzo Pasquali	95
11	Pannexin-2-deficiency sensitizes pancreatic β-cells to cytokine-induced apoptosis in vitro and impairs glucose tolerance in vivo 2017 ·Molecular and Cellular Endocrinology ·Lukas Adrian Berchtold, Michela Miani, Thi Ai Diep, Andreas Nygaard Madsen, Valentina Cigliola, Máikel L. Colli, (unknown), Flemming Pociot, Décio L. Eizirik, Paolo Meda, Birgitte Holst, Nils Billestrup, Joachim Størling	12
12	Production and characterization of a Brazilian candidate antigen for Hepatitis E Virus genotype 3 diagnosis 2016 ·FEMS Microbiology Letters ·(unknown), Michela Miani, (unknown), (unknown), Máikel L. Colli, Fernando Rosado Spilki, Noemi Rovaris Gardinali, Marcelo Alves Pinto, Luiz Carlos Kreutz, Rafael Frandoloso	7
13	Endoplasmic reticulum stress sensitizes pancreatic beta cells to interleukin-1β-induced apoptosis via Bim/A1 imbalance 2013 ·Cell Death and Disease ·Michela Miani, Jenny Barthson, Máikel L. Colli, Flora Brozzi, Miriam Cnop, Décio L. Eizirik	42
14	GLIS3, a Susceptibility Gene for Type 1 and Type 2 Diabetes, Modulates Pancreatic Beta Cell Apoptosis via Regulation of a Splice Variant of the BH3-Only Protein Bim 2013 ·PLoS Genetics ·Tatiane C. Nogueira, Flávia M. Paula, Olatz Villate, Máikel L. Colli, Rodrigo Ferreira de Moura, Daniel A. Cunha, Lorella Marselli, Piero Marchetti, Miriam Cnop, Cécile Julier, Décio L. Eizirik	136
15	The Human Pancreatic Islet Transcriptome: Expression of Candidate Genes for Type 1 Diabetes and the Impact of Pro-Inflammatory Cytokines 2012 ·PLoS Genetics ·Décio L. Eizirik, Michael Sammeth, Thomas Bouckenooghe, Guy Bottu, Giorgia Sisino, Mariana Igoillo‐Esteve, Fernanda Ortis, Izortze Santín, Máikel L. Colli, Jenny Barthson, Luc Bouwens, Linda Hughes, Lorna Gregory, Gerton Lunter, Lorella Marselli, Piero Marchetti, Mark I. McCarthy, Miriam Cnop	358
16	Differential usage of NF‐κB activating signals by IL‐1β and TNF‐α in pancreatic beta cells 2012 ·FEBS Letters ·Fernanda Ortis, Michela Miani, Máikel L. Colli, Daniel A. Cunha, Esteban N. Gurzov, Florent Allagnat, Alain Chariot, Décio L. Eizirik	53
17	Exposure to the Viral By-Product dsRNA or Coxsackievirus B5 Triggers Pancreatic Beta Cell Apoptosis via a Bim / Mcl-1 Imbalance 2011 ·PLoS Pathogens ·Máikel L. Colli, Tatiane C. Nogueira, Florent Allagnat, Daniel A. Cunha, Esteban N. Gurzov, Alessandra K. Cardozo, Merja Roivainen, Anne Op De Beeck, Décio L. Eizirik	49
18	STAT1 Is a Master Regulator of Pancreatic β-Cell Apoptosis and Islet Inflammation 2010 ·Journal of Biological Chemistry ·Fabrice Moore, Najib Naamane, Máikel L. Colli, Thomas Bouckenooghe, Fernanda Ortis, Esteban N. Gurzov, Mariana Igoillo‐Esteve, Chantal Mathieu, Gianluca Bontempi, Thomas Thykjær, Torben F. Ørntoft, Décio L. Eizirik	151
19	The role of inflammation in insulitis and β-cell loss in type 1 diabetes breakdown → 2009 ·Nature Reviews Endocrinology ·Décio L. Eizirik, Máikel L. Colli, Fernanda Ortis	799
20	MDA5 and PTPN2, two candidate genes for type 1 diabetes, modify pancreatic β-cell responses to the viral by-product double-stranded RNA 2009 ·Human Molecular Genetics ·Máikel L. Colli, Fabrice Moore, Esteban N. Gurzov, Fernanda Ortis, Décio L. Eizirik	72

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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