M. Staufenbiel
- Biological Psychiatry top 5%
- Tryptophan and brain disorders 1
- Neurology top 5%
- Neurological Disease Mechanisms and Treatments 1
- Traumatic Brain Injury and Neurovascular Disturbances 1
- Physiology top 5%
- Alzheimer's disease research and treatments 6
- Physiology top 10%
- Alzheimer's disease research and treatments 6
- Pharmacology top 10%
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- Prion Diseases and Protein Misfolding 2
- Mitochondrial Function and Pathology 1
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- Autophagy in Disease and Therapy 1
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- Neuroscience and Neuropharmacology Research 1
- Co-authors
- Martin J. BergJames PeddyChunfeng HuoChris N. GoulbourneCédric Bouchet‐MarquisEfrat LevyMonika PawlikRalph A. Nixon
- Partner nations
- GermanySwitzerlandUnited States
In The Last Decade
M. Staufenbiel
8 papers receiving 789 citations
Hit Papers
Peers
Comparison fields: 5 of 84
- Biological Psychiatry 77
- Neurology 224
- Physiology 518
- Physiology 39
- Pharmacology 127
Countries citing papers authored by M. Staufenbiel
This map shows the geographic impact of M. Staufenbiel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Staufenbiel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Staufenbiel more than expected).
Fields of papers citing papers by M. Staufenbiel
This network shows the impact of papers produced by M. Staufenbiel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Staufenbiel. The network helps show where M. Staufenbiel may publish in the future.
Co-authorship network
The 25 scholars most cited alongside M. Staufenbiel, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Faulty autolysosome acidification in Alzheimer’s disease mouse models induces autophagic build-up of Aβ in neurons, yielding senile plaquesbreakdown → | 2022 | 397 |
| 2 | 2016 | 6 | |
| 3 | 2014 | 101 | |
| 4 | 2011 | 149 | |
| 5 | 2010 | 68 | |
| 6 | 2007 | 31 | |
| 7 | Amyloid beta-protein and apolipoprotein E co-localise within perivascular drainage channels in wild-type and APP-transgenic mice | 2006 | 3 |
| 8 | 2006 | 40 |
About M. Staufenbiel
M. Staufenbiel is a scholar working on Biological Psychiatry, Developmental Neuroscience and Physiology, having authored 8 papers that have together received 795 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (6 papers), Prion Diseases and Protein Misfolding (2 papers), Autophagy in Disease and Therapy (1 paper), Neurological Disease Mechanisms and Treatments (1 paper), Mitochondrial Function and Pathology (1 paper), Tryptophan and brain disorders (1 paper), Neuroscience and Neuropharmacology Research (1 paper) and Traumatic Brain Injury and Neurovascular Disturbances (1 paper). The work is most often cited by research in Biological Psychiatry (77 citations), Neurology (224 citations) and Physiology (518 citations). M. Staufenbiel has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Martin J. Berg, James Peddy, Chunfeng Huo, Chris N. Goulbourne, Cédric Bouchet‐Marquis, Efrat Levy, Monika Pawlik, Ralph A. Nixon, Ju‐Hyun Lee and Cynthia Bleiwas. Their work appears in journals such as Nature Medicine, Journal of Neuroscience and Nature Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.