M. Grazia Cotticelli
- Genetics top 2%
- Genetics and Neurodevelopmental Disorders 5
- Genomic variations and chromosomal abnormalities 2
- Molecular Biology top 5%
- Mitochondrial Function and Pathology 10
- Chromatin Remodeling and Cancer 4
- DNA Repair Mechanisms 3
- Epigenetics and DNA Methylation 2
- Cancer Research top 10%
- Oncology top 10%
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- Genetic Neurodegenerative Diseases 10
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- Endoplasmic Reticulum Stress and Disease 2
- Co-authors
- Masataka OhtaRaffaele BaffaHiroshi InoueCarlo M. CroceTeresa DruckKumar KasturyZurab SiprashviliJuan Palazzo
- Partner nations
- United StatesItalyJapan
In The Last Decade
M. Grazia Cotticelli
19 papers receiving 2.1k citations
Hit Papers
Peers
Comparison fields: 5 of 85
- Genetics 1.0k
- Molecular Biology 1.6k
- Cancer Research 252
- Oncology 410
- Pathology and Forensic Medicine 227
Countries citing papers authored by M. Grazia Cotticelli
This map shows the geographic impact of M. Grazia Cotticelli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Grazia Cotticelli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Grazia Cotticelli more than expected).
Fields of papers citing papers by M. Grazia Cotticelli
This network shows the impact of papers produced by M. Grazia Cotticelli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Grazia Cotticelli. The network helps show where M. Grazia Cotticelli may publish in the future.
Co-authorship network
The 25 scholars most cited alongside M. Grazia Cotticelli, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2022 | 16 | |
| 2 | 2022 | 11 | |
| 3 | 2020 | 8 | |
| 4 | 2019 | 105 | |
| 5 | 2018 | 9 | |
| 6 | 2015 | 2 | |
| 7 | 2014 | 41 | |
| 8 | 2013 | 67 | |
| 9 | 2012 | 66 | |
| 10 | 2011 | 20 | |
| 11 | 2008 | 11 | |
| 12 | 2002 | 83 | |
| 13 | The murine Fhit locus: isolation, characterization, and expression in normal and tumor cells. | 1998 | 28 |
| 14 | Structure and expression of the human FHIT gene in normal and tumor cells. | 1997 | 173 |
| 15 | 1997 | 10 | |
| 16 | 1996 | 461 | |
| 17 | The FHIT Gene, Spanning the Chromosome 3p14.2 Fragile Site and Renal Carcinoma–Associated t(3;8) Breakpoint, Is Abnormal in Digestive Tract Cancersbreakdown → | 1996 | 884 |
| 18 | Potential gastrointestinal tumor suppressor locus at the 3p14.2 FRA3B site identified by homozygous deletions in tumor cell lines. | 1996 | 97 |
| 19 | 1991 | 30 |
About M. Grazia Cotticelli
M. Grazia Cotticelli is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Genetics, having authored 19 papers that have together received 2.1k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (10 papers), Genetic Neurodegenerative Diseases (10 papers), Genetics and Neurodevelopmental Disorders (5 papers), Chromatin Remodeling and Cancer (4 papers), DNA Repair Mechanisms (3 papers), Epigenetics and DNA Methylation (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers) and Genomic variations and chromosomal abnormalities (2 papers). The work is most often cited by research in Genetics (1.0k citations), Molecular Biology (1.6k citations) and Cancer Research (252 citations). M. Grazia Cotticelli has collaborated with scholars based in United States, Italy and Japan. Frequent co-authors include Masataka Ohta, Raffaele Baffa, Hiroshi Inoue, Carlo M. Croce, Teresa Druck, Kumar Kastury, Zurab Siprashvili, Juan Palazzo, Peter A. McCue and Kay Huebner. Their work appears in journals such as Cell, The Journal of Experimental Medicine and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.