Luitpold E. Miller
- Cellular and Molecular Neuroscience top 10%
- Molecular Biology
- Physiology
- Neurology top 10%
- Rheumatology top 10%
- Co-authors
- Rainer H. StraubHans‐Peter JüstenJürgen SchölmerichWerner FalkMaurizio CutoloJoachim GrifkaPeter AngeleJens Schaumburger
- Topics
- Neuropeptides and Animal Physiology (4 papers)Peripheral Neuropathies and Disorders (4 papers)Pain Mechanisms and Treatments (3 papers)
In The Last Decade
Luitpold E. Miller
8 papers receiving 563 citations
Peers
Comparison fields: 5 of 79
- Cellular and Molecular Neuroscience 200
- Molecular Biology 141
- Physiology 116
- Neurology 94
- Rheumatology 93
Countries citing papers authored by Luitpold E. Miller
This map shows the geographic impact of Luitpold E. Miller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luitpold E. Miller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luitpold E. Miller more than expected).
Fields of papers citing papers by Luitpold E. Miller
This network shows the impact of papers produced by Luitpold E. Miller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luitpold E. Miller. The network helps show where Luitpold E. Miller may publish in the future.
Co-authorship network of co-authors of Luitpold E. Miller
This figure shows the co-authorship network connecting the top 25 collaborators of Luitpold E. Miller. A scholar is included among the top collaborators of Luitpold E. Miller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Luitpold E. Miller. Luitpold E. Miller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 82 | |
| 2 | 52 | |
| 3 | Norepinephrine from synovial tyrosine hydroxylase positive cells is a strong indicator of synovial inflammation in rheumatoid arthritis. | 50 |
| 4 | 60 | |
| 5 | 53 | |
| 6 | 227 | |
| 7 | Neurotransmitter modulation of interleukin 6 (IL-6) and IL-8 secretion of synovial fibroblasts in patients with rheumatoid arthritis compared to osteoarthritis. | 43 |
| 8 | 7 |
About Luitpold E. Miller
Luitpold E. Miller is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 8 papers that have together received 574 indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (4 papers), Peripheral Neuropathies and Disorders (4 papers) and Pain Mechanisms and Treatments (3 papers). The work is most often cited by research in Behavioral Neuroscience (80 citations), Biological Psychiatry (34 citations) and Neurology (94 citations). Luitpold E. Miller has collaborated with scholars based in Germany and Italy. Frequent co-authors include Rainer H. Straub, Hans‐Peter Jüsten, Jürgen Schölmerich, Werner Falk, Maurizio Cutolo, Joachim Grifka, Peter Angele, Jens Schaumburger, Claudia Weidler and David S. Jessop. Their work appears in journals such as The FASEB Journal, Annals of the New York Academy of Sciences and Journal of Neurochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.