Lucija Tudor
- Biological Psychiatry top 2%
- Molecular Biology
- Physiology
- Behavioral Neuroscience top 5%
- Cellular and Molecular Neuroscience
- Co-authors
- Nela PivacGordana Nedić ErjavecDubravka Švob ŠtracMatea Nikolac PerkovićMarcela KonjevodMarina ŠagudSuzana UzunOliver Kozumplik
- Topics
- Tryptophan and brain disorders (15 papers)Nerve injury and regeneration (7 papers)Schizophrenia research and treatment (6 papers)
- Journals
- Free Radical Biology and MedicineInternational Journal of Molecular SciencesPsychiatry Research
- Partner nations
- CroatiaSpainUnited States
In The Last Decade
Lucija Tudor
43 papers receiving 661 citations
Peers
Comparison fields: 5 of 102
- Biological Psychiatry 193
- Molecular Biology 186
- Physiology 136
- Behavioral Neuroscience 110
- Cellular and Molecular Neuroscience 101
Countries citing papers authored by Lucija Tudor
This map shows the geographic impact of Lucija Tudor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lucija Tudor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lucija Tudor more than expected).
Fields of papers citing papers by Lucija Tudor
This network shows the impact of papers produced by Lucija Tudor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lucija Tudor. The network helps show where Lucija Tudor may publish in the future.
Co-authorship network of co-authors of Lucija Tudor
This figure shows the co-authorship network connecting the top 25 collaborators of Lucija Tudor. A scholar is included among the top collaborators of Lucija Tudor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lucija Tudor. Lucija Tudor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 6 | |
| 3 | 8 | |
| 4 | 5 | |
| 5 | 2 | |
| 6 | 2 | |
| 7 | 20 | |
| 8 | 15 | |
| 9 | 12 | |
| 10 | 8 | |
| 11 | 19 | |
| 12 | 6 | |
| 13 | 2 | |
| 14 | 12 | |
| 15 | 11 | |
| 16 | 31 | |
| 17 | 31 | |
| 18 | 2 | |
| 19 | 18 | |
| 20 | 34 |
About Lucija Tudor
Lucija Tudor is a scholar working on Biological Psychiatry, Behavioral Neuroscience and Developmental Neuroscience, having authored 43 papers that have together received 669 indexed citations. Recurring topics across this work include Tryptophan and brain disorders (15 papers), Nerve injury and regeneration (7 papers) and Schizophrenia research and treatment (6 papers). The work is most often cited by research in Biological Psychiatry (193 citations), Behavioral Neuroscience (110 citations) and Developmental Neuroscience (40 citations). Lucija Tudor has collaborated with scholars based in Croatia, Spain and United States. Frequent co-authors include Nela Pivac, Gordana Nedić Erjavec, Dubravka Švob Štrac, Matea Nikolac Perković, Marcela Konjevod, Marina Šagud, Suzana Uzun, Oliver Kozumplik, Neven Žarković and Ninoslav Mimica. Their work appears in journals such as Free Radical Biology and Medicine, International Journal of Molecular Sciences and Psychiatry Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.