Linda E. Nee

9.7k total citations
57 papers, 2.7k citations indexed

About

Linda E. Nee is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Linda E. Nee has authored 57 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Physiology, 24 papers in Molecular Biology and 13 papers in Cellular and Molecular Neuroscience. Recurrent topics in Linda E. Nee's work include Alzheimer's disease research and treatments (28 papers), Prion Diseases and Protein Misfolding (8 papers) and Genetic Neurodegenerative Diseases (7 papers). Linda E. Nee is often cited by papers focused on Alzheimer's disease research and treatments (28 papers), Prion Diseases and Protein Misfolding (8 papers) and Genetic Neurodegenerative Diseases (7 papers). Linda E. Nee collaborates with scholars based in United States, Canada and Japan. Linda E. Nee's co-authors include Ronald J. Polinsky, Michael H. Ebert, Peter St George‐Hyslop, Joseph Higgins, Carol F. Lippa, Thomas D. Bird, Eric D. Caine, Rudolph E. Tanzi, John Q. Trojanowski and Virginia M.‐Y. Lee and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Journal of Cell Biology.

In The Last Decade

Linda E. Nee

57 papers receiving 2.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Linda E. Nee United States 26 1.3k 920 836 611 387 57 2.7k
M. Axel Wollmer Germany 25 1.3k 1.0× 954 1.0× 408 0.5× 474 0.8× 351 0.9× 56 2.9k
Lucio Tremolizzo Italy 26 434 0.3× 967 1.1× 523 0.6× 551 0.9× 431 1.1× 134 2.6k
María Martínez France 36 1.2k 0.9× 1.6k 1.8× 710 0.8× 865 1.4× 826 2.1× 132 4.8k
Silvia Bagnoli Italy 29 1.0k 0.8× 814 0.9× 229 0.3× 363 0.6× 431 1.1× 135 2.4k
Jennifer S. Yokoyama United States 30 790 0.6× 741 0.8× 589 0.7× 339 0.6× 508 1.3× 91 2.6k
Martine Vercelletto France 16 942 0.7× 635 0.7× 388 0.5× 250 0.4× 409 1.1× 46 1.9k
Eitan Friedman United States 29 475 0.4× 1.3k 1.4× 186 0.2× 1.1k 1.8× 222 0.6× 60 3.2k
Marwa Elamin Ireland 31 797 0.6× 571 0.6× 2.9k 3.5× 391 0.6× 266 0.7× 51 3.5k
Gary W. Beecham United States 24 714 0.5× 994 1.1× 581 0.7× 418 0.7× 181 0.5× 89 2.5k
Marta Barrachina Spain 30 706 0.5× 1.3k 1.4× 720 0.9× 651 1.1× 134 0.3× 45 2.8k

Countries citing papers authored by Linda E. Nee

Since Specialization
Citations

This map shows the geographic impact of Linda E. Nee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Linda E. Nee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Linda E. Nee more than expected).

Fields of papers citing papers by Linda E. Nee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Linda E. Nee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Linda E. Nee. The network helps show where Linda E. Nee may publish in the future.

Co-authorship network of co-authors of Linda E. Nee

This figure shows the co-authorship network connecting the top 25 collaborators of Linda E. Nee. A scholar is included among the top collaborators of Linda E. Nee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Linda E. Nee. Linda E. Nee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ishii, Kazuhiro, Carol F. Lippa, Takami Tomiyama, et al.. (2001). Distinguishable effects of Presenilin-1 and APP717 mutations on amyloid plaque deposition. Neurobiology of Aging. 22(3). 367–376. 18 indexed citations
2.
Imafuku, Ichiro, Toshihiro Masaki, Masaaki Waragai, et al.. (1999). Presenilin 1 Suppresses the Function of C-Jun Homodimers via Interaction with Qm/Jif-1. The Journal of Cell Biology. 147(1). 121–134. 68 indexed citations
3.
Lippa, Carol F., Hideo Fujiwara, David M. A. Mann, et al.. (1998). Lewy Bodies Contain Altered α-Synuclein in Brains of Many Familial Alzheimer's Disease Patients with Mutations in Presenilin and Amyloid Precursor Protein Genes. American Journal Of Pathology. 153(5). 1365–1370. 429 indexed citations
4.
Higgins, Joseph, et al.. (1997). A gene (ETM) for essential tremor maps to chromosome 2p22‐p25. Movement Disorders. 12(6). 859–864. 150 indexed citations
5.
Okochi, Masayasu, Mihoko Usami, Naruhiko Sahara, et al.. (1997). Proteolytic processing of presenilin‐1 (PS‐1) is not associated with Alzheimer's disease with or without PS‐1 mutations. FEBS Letters. 418(1-2). 162–166. 31 indexed citations
6.
Higgins, Joe, et al.. (1997). Evidence for a new spinocerebellar ataxia locus. Movement Disorders. 12(3). 412–417. 8 indexed citations
7.
Higgins, Joe, Linda E. Nee, Olavo M. Vasconcelos, et al.. (1996). Mutations in American families with spinocerebellar ataxia (SCA) type 3. Neurology. 46(1). 208–213. 41 indexed citations
8.
Ikeda, Masaki, Vikram Sharma, S. M. Sumi, et al.. (1996). The Clinical phenotype of two missense mutations in the presenilin I gene in Japanese patients. Annals of Neurology. 40(6). 912–917. 49 indexed citations
9.
Lippa, Carol F., Thomas W. Smith, Linda E. Nee, et al.. (1995). Familial Alzheimer’s Disease and Cortical Lewy Bodies: Is There a Genetic Susceptibility Factor?. Dementia and Geriatric Cognitive Disorders. 6(4). 191–194. 12 indexed citations
10.
Nee, Linda E.. (1995). Effects of Psychosocial Interactions at a Cellular Level. Social Work. 40(2). 259–62. 7 indexed citations
11.
Dahlström, Annica, et al.. (1994). Alzheimer's disease cerebrospinal fluid antibodies display selectivity for microglia. Molecular Neurobiology. 9(1-3). 41–54. 15 indexed citations
12.
Nee, Linda E., James A. Scott, & Ronald J. Polinsky. (1993). Olfactory dysfunction in the Shy—Drager syndrome. Clinical Autonomic Research. 3(4). 281–282. 13 indexed citations
13.
Nechiporuk, Alex, Pamela R. Fain, Edward N. Kort, et al.. (1993). Linkage of familial alzheimer disease to chromosome 14 in two large early‐onset pedigrees: Effects of marker allele frequencies on lod scores. American Journal of Medical Genetics. 48(1). 63–66. 22 indexed citations
14.
Mullins, R. Dyche, Terry Newcomb, Gabriela Pavlínková, et al.. (1993). Serum- and bradykinin-induced calcium transients in familial Alzheimer's fibroblasts. Neurobiology of Aging. 14(5). 447–455. 32 indexed citations
15.
Vaula, Giovanna, Marzia Mortilla, Rossella Tupler, et al.. (1992). A novel but non-pathogenic mutation in exon 4 of the human amyloid precursor protein (APP) gene. Neuroscience Letters. 144(1-2). 46–48. 9 indexed citations
16.
Pulst, S. M., Pamela R. Fain, V H Cohn, et al.. (1991). Exclusion of linkage to the pericentromeric region of chromosome 21 in the Canadian pedigree with familial Alzheimer disease. Human Genetics. 87(2). 159–161. 2 indexed citations
17.
Struble, Robert G., Ronald J. Polinsky, John C. Hedreen, et al.. (1991). Hippocampal Lesions in Dominantly Inherited Alzheimer's Disease. Journal of Neuropathology & Experimental Neurology. 50(1). 82–94. 18 indexed citations
18.
Frommelt, Peter C., et al.. (1991). Familial Alzheimer Disease: A Large, Multigeneration German Kindred. Alzheimer Disease & Associated Disorders. 5(1). 36–43. 25 indexed citations
19.
Nee, Linda E., et al.. (1991). Environmental— occupational risk factors and familial associations in multiple system atrophy: A preliminary investigation. Clinical Autonomic Research. 1(1). 9–13. 78 indexed citations
20.
Jones, Susan, Linda E. Nee, Ronald J. Polinsky, et al.. (1989). Decreased DNA repair in familial Alzheimer's disease. Mutation Research/DNAging. 219(4). 247–255. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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