Lidia de Bari
About
Lidia de Bari is a scholar working on Molecular Biology, Clinical Biochemistry and Physiology.
According to data from OpenAlex, Lidia de Bari has authored 39 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 11 papers in Clinical Biochemistry and 7 papers in Physiology. Recurrent topics in Lidia de Bari's work include Mitochondrial Function and Pathology (19 papers), Metabolism and Genetic Disorders (7 papers) and ATP Synthase and ATPases Research (6 papers). Lidia de Bari is often cited by papers focused on Mitochondrial Function and Pathology (19 papers), Metabolism and Genetic Disorders (7 papers) and ATP Synthase and ATPases Research (6 papers). Lidia de Bari collaborates with scholars based in Italy, France and Poland. Lidia de Bari's co-authors include Anna Atlante, Daniela Valenti, Salvatore Passarella, Rosa Anna Vacca, Antonella Bobba, Bianca De Filippis, Alexandra Henrion‐Caude, Domenico De Rasmo, Ersilia Marra and Roberto Pizzuto and has published in prestigious journals such as Analytical Biochemistry, Biochemical Journal and Biochemical and Biophysical Research Communications.
In The Last Decade
Lidia de Bari
39 papers receiving 1.6k citations
Peers
Lidia de Bari
Domenico De Rasmo Italy
Raffaella Nicolai Italy
Rachel Karry Israel
Wagner Seixas da‐Silva Brazil
Blanca Rubı́ Spain
Marco Spinazzi Italy
Junko Wakabayashi Japan
Ilona I. Concha Chile
François Casas France
Larissa Daniele Bobermin Brazil
Citations per year, relative to Lidia de Bari Lidia de Bari (= 1×)
peers
Domenico De Rasmo
Countries citing papers authored by Lidia de Bari
Since
Specialization
Citations
This map shows the geographic impact of Lidia de Bari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lidia de Bari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lidia de Bari more than expected).
Fields of papers citing papers by Lidia de Bari
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Lidia de Bari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lidia de Bari. The network helps show where Lidia de Bari may publish in the future.
Co-authorship network of co-authors of Lidia de Bari
This figure shows the co-authorship network connecting the top 25 collaborators of Lidia de Bari. A scholar is included among the top collaborators of Lidia de Bari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lidia de Bari. Lidia de Bari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Armeni, Tatiana, Lidia de Bari, Andrea Scirè, et al.. (2025). Acetylcholine Sustains LNCaP Prostate Cancer Cell Migration, Invasion and Proliferation Through Glyoxalase 1/MG-H1 Axis with the Involvement of Osteopontin. International Journal of Molecular Sciences. 26(9). 4107–4107.
2.
Kalapos, Miklós Péter & Lidia de Bari. (2024). The evolutionary arch of bioenergetics from prebiotic mechanisms to the emergence of a cellular respiratory chain. Biosystems. 244. 105288–105288.
3.
Scirè, Andrea, Cristina Minnelli, Andrea Frontini, et al.. (2024). Overexpression of Glyoxalase 2 in Human Breast Cancer Cells: Implications for Cell Proliferation and Doxorubicin Resistance. International Journal of Molecular Sciences. 25(20). 10888–10888.
4.
Scirè, Andrea, et al.. (2023). Glutathionyl Hemoglobin and Its Emerging Role as a Clinical Biomarker of Chronic Oxidative Stress. Antioxidants. 12(11). 1976–1976.
5.
Kalapos, Miklós Péter, Cinzia Antognelli, & Lidia de Bari. (2022). Metabolic Shades of S-D-Lactoylglutathione. Antioxidants. 11(5). 1005–1005.
6.
Favia, Maria, et al.. (2019). Modulation of glucose-related metabolic pathways controls glucose level in airway surface liquid and fight oxidative stress in cystic fibrosis cells. Journal of Bioenergetics and Biomembranes. 51(3). 203–218.
7.
Bari, Lidia de, Anna Atlante, Tatiana Armeni, & Miklós Péter Kalapos. (2019). Synthesis and metabolism of methylglyoxal, S-D-lactoylglutathione and D-lactate in cancer and Alzheimer’s disease. Exploring the crossroad of eternal youth and premature aging. Ageing Research Reviews. 53. 100915–100915.
8.
Bari, Lidia de, et al.. (2018). Aberrant GSH reductase and NOX activities concur with defective CFTR to pro-oxidative imbalance in cystic fibrosis airways. Journal of Bioenergetics and Biomembranes. 50(2). 117–129.
9.
Atlante, Anna, Lidia de Bari, Antonella Bobba, & Giuseppina Amadoro. (2017). A disease with a sweet tooth: exploring the Warburg effect in Alzheimer’s disease. Biogerontology. 18(3). 301–319.
10.
Valenti, Daniela, Lidia de Bari, Enza Lacivita, et al.. (2017). Stimulation of the brain serotonin receptor 7 rescues mitochondrial dysfunction in female mice from two models of Rett syndrome. Neuropharmacology. 121. 79–88.
11.
Filippis, Bianca De, Daniela Valenti, Valentina Chiodi, et al.. (2015). Modulation of Rho GTPases rescues brain mitochondrial dysfunction, cognitive deficits and aberrant synaptic plasticity in female mice modeling Rett syndrome. European Neuropsychopharmacology. 25(6). 889–901.
12.
Valenti, Daniela, Rosa Anna Vacca, & Lidia de Bari. (2015). 3-Bromopyruvate induces rapid human prostate cancer cell death by affecting cell energy metabolism, GSH pool and the glyoxalase system. Journal of Bioenergetics and Biomembranes. 47(6). 493–506.
13.
Valenti, Daniela, Lidia de Bari, Bianca De Filippis, Alexandra Henrion‐Caude, & Rosa Anna Vacca. (2014). Mitochondrial dysfunction as a central actor in intellectual disability-related diseases: An overview of Down syndrome, autism, Fragile X and Rett syndrome. Neuroscience & Biobehavioral Reviews. 46. 202–217.
14.
Valenti, Daniela, Lidia de Bari, Leonardo Rossi, et al.. (2013). Negative modulation of mitochondrial oxidative phosphorylation by epigallocatechin-3 gallate leads to growth arrest and apoptosis in human malignant pleural mesothelioma cells. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(12). 2085–2096.
15.
Valenti, Daniela, Domenico De Rasmo, Anna Signorile, et al.. (2013). Epigallocatechin-3-gallate prevents oxidative phosphorylation deficit and promotes mitochondrial biogenesis in human cells from subjects with Down's syndrome. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(4). 542–552.
16.
Bari, Lidia de, Loredana Moro, & Salvatore Passarella. (2013). Prostate cancer cells metabolize d ‐lactate inside mitochondria via a d ‐lactate dehydrogenase which is more active and highly expressed than in normal cells. FEBS Letters. 587(5). 467–473.
17.
Atlante, Anna, Giuseppina Amadoro, Antonella Bobba, et al.. (2008). A peptide containing residues 26–44 of tau protein impairs mitochondrial oxidative phosphorylation acting at the level of the adenine nucleotide translocator. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1777(10). 1289–1300.
18.
Bari, Lidia de, Daniela Valenti, Roberto Pizzuto, Anna Atlante, & Salvatore Passarella. (2007). Phosphoenolpyruvate metabolism in Jerusalem artichoke mitochondria. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1767(4). 281–294.
19.
Atlante, Anna, Lidia de Bari, Antonella Bobba, Ersilia Marra, & Salvatore Passarella. (2007). Transport and metabolism of l-lactate occur in mitochondria from cerebellar granule cells and are modified in cells undergoing low potassium dependent apoptosis. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1767(11). 1285–1299.
20.
Bari, Lidia de, Daniela Valenti, Roberto Pizzuto, et al.. (2005). Jerusalem artichoke mitochondria can export reducing equivalents in the form of malate as a result of d-lactate uptake and metabolism. Biochemical and Biophysical Research Communications. 335(4). 1224–1230.
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