Agatha Schlüter

11.9k citations
42 papers · 1.8k indexed · h-index 23
Topics
Peroxisome Proliferator-Activated Receptors (19 papers)Adipose Tissue and Metabolism (10 papers)Mitochondrial Function and Pathology (6 papers)
Partner nations
SpainUnited StatesFrance

In The Last Decade

Agatha Schlüter

41 papers receiving 1.8k citations

Peers

Agatha Schlüter
Comparison fields: 5 of 97
  • Molecular Biology 1.2k
  • Physiology 701
  • Epidemiology 237
  • Biochemistry 195
  • Neurology 165
Replace Ágnes Zvara with:
Ágnes Zvara Hungary
Tatiana R. Rosenstock Brazil
Marioly Müller Chile
Christopher J. Lelliott United Kingdom
David Sebastián Spain
Jessica M. Silvaggi United States
Fuminori Yamaguchi Japan
Torgeir Holen Norway
Naïg Guéguen France
Monika Pawlik United States
Agatha Schlüter relative to Ágnes Zvara Hungary Ágnes Zvara's profile →
Citations per field
00.5×1.5×2.1×
Ágnes Zvara · 1×
Citations per year

Countries citing papers authored by Agatha Schlüter

Since Specialization
Citations

This map shows the geographic impact of Agatha Schlüter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Agatha Schlüter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Agatha Schlüter more than expected).

Fields of papers citing papers by Agatha Schlüter

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Agatha Schlüter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Agatha Schlüter. The network helps show where Agatha Schlüter may publish in the future.

Co-authorship network of co-authors of Agatha Schlüter

This figure shows the co-authorship network connecting the top 25 collaborators of Agatha Schlüter. A scholar is included among the top collaborators of Agatha Schlüter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Agatha Schlüter. Agatha Schlüter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 0
2 7
3 9
4 18
5 19
6 33
7 31
8 34
9 105
10 120
11 7
12 38
13 72
14 60
15 109
16 6
17 86
18 132
19 36
20 41

About Agatha Schlüter

Agatha Schlüter is a scholar working on Biochemistry, Physiology and Molecular Biology, having authored 42 papers that have together received 1.8k indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (19 papers), Adipose Tissue and Metabolism (10 papers) and Mitochondrial Function and Pathology (6 papers). The work is most often cited by research in Physiology (701 citations), Biochemistry (195 citations) and Clinical Biochemistry (159 citations). Agatha Schlüter has collaborated with scholars based in Spain, United States and France. Frequent co-authors include Aurora Pujol, Francesc Villarroya, Stéphane Fourcade, Marta Giralt, Roser Iglesias, Maria José Barberà, Isidró Ferrer, Neus Pedraza, Montserrat Ruíz and Manuel Portero-Otı́n. Their work appears in journals such as Nucleic Acids Research, Journal of Biological Chemistry and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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