Alessia Fabbri
- Endocrinology top 1%
- Escherichia coli research studies 9
- Immunology top 5%
- Toxin Mechanisms and Immunotoxins 11
- Genetics top 5%
- Bacterial Genetics and Biotechnology 17
- Genetics and Neurodevelopmental Disorders 7
- Infectious Diseases top 5%
- Clostridium difficile and Clostridium perfringens research 13
- Antimicrobial Resistance in Staphylococcus 6
- Molecular Biology top 5%
- Cell death mechanisms and regulation 6
- Gut microbiota and health 5
- Co-authors
- Carla FiorentiniSara TravaglioneLoredana FalzanoPaola MatarreseGianfranco DonelliPatrice BoquetWalter MalorniLaura Ricceri
- Cited by
- EndocrinologyImmunologyGenetics
In The Last Decade
Alessia Fabbri
75 papers receiving 2.5k citations
Peers
Comparison fields: 5 of 114
- Endocrinology 511
- Immunology 568
- Genetics 693
- Infectious Diseases 419
- Molecular Biology 1.3k
Countries citing papers authored by Alessia Fabbri
This map shows the geographic impact of Alessia Fabbri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alessia Fabbri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alessia Fabbri more than expected).
Fields of papers citing papers by Alessia Fabbri
This network shows the impact of papers produced by Alessia Fabbri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alessia Fabbri. The network helps show where Alessia Fabbri may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Alessia Fabbri, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 2 | |
| 2 | 2023 | 3 | |
| 3 | 2021 | 22 | |
| 4 | 2019 | 8 | |
| 5 | 2018 | 14 | |
| 6 | 2018 | 4 | |
| 7 | 2016 | 25 | |
| 8 | 2015 | 32 | |
| 9 | 2015 | 43 | |
| 10 | 2011 | 86 | |
| 11 | 2010 | 83 | |
| 12 | 2008 | 37 | |
| 13 | 2007 | 108 | |
| 14 | 2007 | 98 | |
| 15 | 2006 | 9 | |
| 16 | 2001 | 18 | |
| 17 | 2001 | 13 | |
| 18 | Toxin B Induces Apoptosis in Intestinal Cultured Cells. | 1999 | 1 |
| 19 | 1997 | 82 | |
| 20 | A toxic factor from pathogenic E. coli strains enhances actin assembly in epithelial cultured cells | 1995 | 1 |
About Alessia Fabbri
Alessia Fabbri is a scholar working on Endocrinology, Immunology and Genetics, having authored 76 papers that have together received 2.6k indexed citations. Recurring topics across this work include Bacterial Genetics and Biotechnology (17 papers), Clostridium difficile and Clostridium perfringens research (13 papers), Toxin Mechanisms and Immunotoxins (11 papers), Escherichia coli research studies (9 papers), Genetics and Neurodevelopmental Disorders (7 papers), Antimicrobial Resistance in Staphylococcus (6 papers), Cell death mechanisms and regulation (6 papers) and Gut microbiota and health (5 papers). The work is most often cited by research in Endocrinology (511 citations), Immunology (568 citations) and Genetics (693 citations). Alessia Fabbri has collaborated with scholars based in Italy, France and Denmark. Frequent co-authors include Carla Fiorentini, Sara Travaglione, Loredana Falzano, Paola Matarrese, Gianfranco Donelli, Patrice Boquet, Walter Malorni, Laura Ricceri, Marco Guidotti and Bianca De Filippis.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.