Leonie A. Cluse

4.0k total citations · 1 hit paper
21 papers, 2.4k citations indexed

About

Leonie A. Cluse is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Leonie A. Cluse has authored 21 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 8 papers in Oncology and 4 papers in Immunology. Recurrent topics in Leonie A. Cluse's work include Histone Deacetylase Inhibitors Research (11 papers), Protein Degradation and Inhibitors (10 papers) and Ubiquitin and proteasome pathways (4 papers). Leonie A. Cluse is often cited by papers focused on Histone Deacetylase Inhibitors Research (11 papers), Protein Degradation and Inhibitors (10 papers) and Ubiquitin and proteasome pathways (4 papers). Leonie A. Cluse collaborates with scholars based in Australia, United States and Austria. Leonie A. Cluse's co-authors include Ricky W. Johnstone, Andrew W. Roberts, Adrian P. Wiegmans, Warren S. Alexander, Donald Metcalf, Douglas J. Hilton, Lynne Hartley, Nicos A. Nicola, Andrea Newbold and Ben A. Croker and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Blood and Immunity.

In The Last Decade

Leonie A. Cluse

21 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Small molecule obatoclax (GX15-070) antagonizes MCL-1 and overcomes MCL-1-mediated resistance to apoptosis

2007 538 citations Mai Nguyen, Richard Marcellus et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Leonie A. Cluse Australia	18	1.7k	884	638	279	160	21	2.4k
Martin Hegen United States	26	900 0.5×	992 1.1×	761 1.2×	137 0.5×	231 1.4×	35	2.3k
Qing Rao China	24	1.0k 0.6×	717 0.8×	547 0.9×	425 1.5×	193 1.2×	145	1.9k
Muxiang Zhou United States	26	1.6k 1.0×	893 1.0×	355 0.6×	291 1.0×	525 3.3×	51	2.3k
Anna Scuto United States	19	1.4k 0.8×	667 0.8×	462 0.7×	301 1.1×	278 1.7×	30	2.2k
Koh Yamamoto Japan	14	629 0.4×	1.1k 1.2×	1.3k 2.0×	185 0.7×	175 1.1×	32	2.2k
Rémy Castellano France	24	968 0.6×	803 0.9×	1.0k 1.6×	297 1.1×	258 1.6×	49	2.3k
Erik A. Nelson United States	23	907 0.5×	887 1.0×	869 1.4×	530 1.9×	212 1.3×	38	2.3k
Stephanie Grabow Australia	19	1.6k 1.0×	590 0.7×	595 0.9×	196 0.7×	272 1.7×	33	2.2k
Sarah R. Walker United States	22	868 0.5×	812 0.9×	456 0.7×	219 0.8×	377 2.4×	40	2.2k
Wendy Schober United States	34	1.8k 1.1×	729 0.8×	663 1.0×	751 2.7×	355 2.2×	64	3.0k

Countries citing papers authored by Leonie A. Cluse

Since Specialization

Citations

This map shows the geographic impact of Leonie A. Cluse's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonie A. Cluse with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonie A. Cluse more than expected).

Fields of papers citing papers by Leonie A. Cluse

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leonie A. Cluse. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonie A. Cluse. The network helps show where Leonie A. Cluse may publish in the future.

Co-authorship network of co-authors of Leonie A. Cluse

This figure shows the co-authorship network connecting the top 25 collaborators of Leonie A. Cluse. A scholar is included among the top collaborators of Leonie A. Cluse based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leonie A. Cluse. Leonie A. Cluse is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Todorovski, Izabela, et al.. (2025). Lipid-metabolism-focused CRISPR screens identify enzymes of the mevalonate pathway as essential for prostate cancer growth. Cell Reports. 44(4). 115470–115470. 4 indexed citations

Cluse, Leonie A., Iva Nikolić, Deborah A. Knight, et al.. (2018). A Comprehensive Protocol Resource for Performing Pooled shRNA and CRISPR Screens. Methods in molecular biology. 1725. 201–227. 3 indexed citations

Hogg, Simon J., Stephin J. Vervoort, Sumit Deswal, et al.. (2017). BET-Bromodomain Inhibitors Engage the Host Immune System and Regulate Expression of the Immune Checkpoint Ligand PD-L1. Cell Reports. 18(9). 2162–2174. 238 indexed citations

Hogg, Simon J., Andrea Newbold, Stephin J. Vervoort, et al.. (2016). BET Inhibition Induces Apoptosis in Aggressive B-Cell Lymphoma via Epigenetic Regulation of BCL-2 Family Members. Molecular Cancer Therapeutics. 15(9). 2030–2041. 50 indexed citations

Matthews, Geoffrey M., Parinaz Mehdipour, Leonie A. Cluse, et al.. (2015). Functional-genetic dissection of HDAC dependencies in mouse lymphoid and myeloid malignancies. Blood. 126(21). 2392–2403. 46 indexed citations

Matthews, Geoffrey M., Leonie A. Cluse, Eric Wang, et al.. (2014). Abstract 5533: RNAi-mediated depletion of histone deacetylases highlights the potential for isoform-specific inhibitors in B-cell lymphoma and acute myeloid leukemia. Cancer Research. 74(19_Supplement). 5533–5533. 1 indexed citations

West, Alison C., Stephen R. Mattarollo, Jake Shortt, et al.. (2013). An Intact Immune System Is Required for the Anticancer Activities of Histone Deacetylase Inhibitors. Cancer Research. 73(24). 7265–7276. 117 indexed citations

Newbold, Andrea, Geoffrey M. Matthews, Michael Bots, et al.. (2013). Molecular and Biologic Analysis of Histone Deacetylase Inhibitors with Diverse Specificities. Molecular Cancer Therapeutics. 12(12). 2709–2721. 40 indexed citations

Shi, Wei, Kacper Jankowski, Leonie A. Cluse, et al.. (2013). HDAC inhibitors induce tumor-cell-selective pro-apoptotic transcriptional responses. Cell Death and Disease. 4(2). e519–e519. 150 indexed citations

10.

Santoro, Fabio, Oronza A. Botrugno, Roberto Dal Zuffo, et al.. (2013). A dual role for Hdac1: oncosuppressor in tumorigenesis, oncogene in tumor maintenance. Blood. 121(17). 3459–3468. 107 indexed citations

11.

Lindqvist, Lisa, Ingela B. Vikstrom, Jennifer M. Chambers, et al.. (2012). Translation inhibitors induce cell death by multiple mechanisms and Mcl-1 reduction is only a minor contributor. Cell Death and Disease. 3(10). e409–e409. 45 indexed citations

12.

Wiegmans, Adrian P., Amber E. Alsop, Michael Bots, et al.. (2011). Deciphering the Molecular Events Necessary for Synergistic Tumor Cell Apoptosis Mediated by the Histone Deacetylase Inhibitor Vorinostat and the BH3 Mimetic ABT-737. Cancer Research. 71(10). 3603–3615. 45 indexed citations

13.

Ellis, Leigh, Michael Bots, Ralph K. Lindemann, et al.. (2009). The histone deacetylase inhibitors LAQ824 and LBH589 do not require death receptor signaling or a functional apoptosome to mediate tumor cell death or therapeutic efficacy. Blood. 114(2). 380–393. 99 indexed citations

14.

Newbold, Andrea, Ralph K. Lindemann, Leonie A. Cluse, et al.. (2008). Characterisation of the novel apoptotic and therapeutic activities of the histone deacetylase inhibitor romidepsin. Molecular Cancer Therapeutics. 7(5). 1066–1079. 56 indexed citations

15.

Whitecross, Kate F., Amber E. Alsop, Leonie A. Cluse, et al.. (2008). Defining the target specificity of ABT-737 and synergistic antitumor activities in combination with histone deacetylase inhibitors. Blood. 113(9). 1982–1991. 71 indexed citations

16.

Lindemann, Ralph K., Andrea Newbold, Kate F. Whitecross, et al.. (2007). Analysis of the apoptotic and therapeutic activities of histone deacetylase inhibitors by using a mouse model of B cell lymphoma. Proceedings of the National Academy of Sciences. 104(19). 8071–8076. 166 indexed citations

17.

Nguyen, Mai, Richard Marcellus, Anne Roulston, et al.. (2007). Small molecule obatoclax (GX15-070) antagonizes MCL-1 and overcomes MCL-1-mediated resistance to apoptosis. Proceedings of the National Academy of Sciences. 104(49). 19512–19517. 538 indexed citations breakdown →

18.

Croker, Ben A., Donald Metcalf, Lorraine Robb, et al.. (2004). SOCS3 Is a Critical Physiological Negative Regulator of G-CSF Signaling and Emergency Granulopoiesis. Immunity. 20(2). 153–165. 234 indexed citations

19.

Croker, Ben A., David M. Tarlinton, Leonie A. Cluse, et al.. (2002). The Rac2 Guanosine Triphosphatase Regulates B Lymphocyte Antigen Receptor Responses and Chemotaxis and Is Required for Establishment of B-1a and Marginal Zone B Lymphocytes. The Journal of Immunology. 168(7). 3376–3386. 104 indexed citations

20.

Croker, Ben A., Emanuela Handman, John D. Hayball, et al.. (2002). Rac2‐deficient mice display perturbed T‐cell distribution and chemotaxis, but only minor abnormalities in T_H1 responses. Immunology and Cell Biology. 80(3). 231–240. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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