Laetitia Pelloquin

6.1k total citations · 1 hit paper
26 papers, 2.9k citations indexed

About

Laetitia Pelloquin is a scholar working on Molecular Biology, Clinical Biochemistry and Cell Biology. According to data from OpenAlex, Laetitia Pelloquin has authored 26 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 5 papers in Clinical Biochemistry and 5 papers in Cell Biology. Recurrent topics in Laetitia Pelloquin's work include Mitochondrial Function and Pathology (20 papers), ATP Synthase and ATPases Research (14 papers) and Metabolism and Genetic Disorders (5 papers). Laetitia Pelloquin is often cited by papers focused on Mitochondrial Function and Pathology (20 papers), ATP Synthase and ATPases Research (14 papers) and Metabolism and Genetic Disorders (5 papers). Laetitia Pelloquin collaborates with scholars based in France, Chile and United States. Laetitia Pelloquin's co-authors include Pascale Belenguer, Guy Lenaers, Cécile Delettre, Bernard Ducommun, Christian Hamel, Laurent J. Emorine, Claude Turc‐Carel, Josiane Grosgeorge, Nadine Gigarel and Catherine Astarie‐Dequeker and has published in prestigious journals such as Nature Genetics, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Laetitia Pelloquin

26 papers receiving 2.9k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy

2000 1.1k citations Cécile Delettre, Guy Lenaers et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Laetitia Pelloquin France	16	2.6k	769	311	311	309	26	2.9k
Christiane Alexander Germany	11	2.5k 0.9×	750 1.0×	297 1.0×	302 1.0×	252 0.8×	14	2.7k
Cécile Delettre France	27	3.4k 1.3×	974 1.3×	278 0.9×	339 1.1×	384 1.2×	41	3.8k
Arnaud Chevrollier France	31	2.0k 0.8×	526 0.7×	195 0.6×	285 0.9×	506 1.6×	73	2.6k
Veronica Costa Italy	10	2.2k 0.8×	504 0.7×	321 1.0×	433 1.4×	343 1.1×	11	2.5k
Lodovica Vergani Italy	26	1.9k 0.7×	620 0.8×	144 0.5×	276 0.9×	477 1.5×	50	2.4k
Kie Itoh United States	18	1.2k 0.5×	283 0.4×	377 1.2×	271 0.9×	189 0.6×	31	1.6k
Vincent Soubannier Canada	17	2.7k 1.0×	561 0.7×	831 2.7×	390 1.3×	219 0.7×	26	3.3k
Florence Burté United Kingdom	15	838 0.3×	183 0.2×	134 0.4×	215 0.7×	152 0.5×	23	1.2k
Heleen te Brinke Netherlands	19	1.1k 0.4×	328 0.4×	158 0.5×	326 1.0×	89 0.3×	25	1.6k
Stefano DiDonato Italy	35	3.4k 1.3×	2.1k 2.8×	122 0.4×	294 0.9×	738 2.4×	63	4.0k

Countries citing papers authored by Laetitia Pelloquin

Since Specialization

Citations

This map shows the geographic impact of Laetitia Pelloquin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laetitia Pelloquin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laetitia Pelloquin more than expected).

Fields of papers citing papers by Laetitia Pelloquin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Laetitia Pelloquin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laetitia Pelloquin. The network helps show where Laetitia Pelloquin may publish in the future.

Co-authorship network of co-authors of Laetitia Pelloquin

This figure shows the co-authorship network connecting the top 25 collaborators of Laetitia Pelloquin. A scholar is included among the top collaborators of Laetitia Pelloquin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Laetitia Pelloquin. Laetitia Pelloquin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Moulédous, Lionel, Cédrick Florian, Macarena S. Arrázola, et al.. (2023). Mitochondrial OPA1 Deficiency Is Associated to Reversible Defects in Spatial Memory Related to Adult Neurogenesis in Mice. eNeuro. 10(11). ENEURO.0073–23.2023. 4 indexed citations

Barca, Juan Manuel Chao de la, Macarena S. Arrázola, Cinzia Bocca, et al.. (2019). The Metabolomic Signature of Opa1 Deficiency in Rat Primary Cortical Neurons Shows Aspartate/Glutamate Depletion and Phospholipids Remodeling. Scientific Reports. 9(1). 6107–6107. 10 indexed citations

Tribouillard‐Tanvier, Déborah, Marlène Daloyau, Laurent J. Emorine, et al.. (2019). A yeast-based screening assay identifies repurposed drugs that suppress mitochondrial fusion and mtDNA maintenance defects. Disease Models & Mechanisms. 12(2). 15 indexed citations

Arrázola, Macarena S., Marion Szelechowski, Lionel Moulédous, et al.. (2018). Mitochondria in Developmental and Adult Neurogenesis. Neurotoxicity Research. 36(2). 257–267. 37 indexed citations

Moulis, Manon, Aurélie Millet, Marlène Daloyau, et al.. (2016). OPA1 haploinsufficiency induces a BNIP3‐dependent decrease in mitophagy in neurons: relevance to Dominant Optic Atrophy. Journal of Neurochemistry. 140(3). 485–494. 25 indexed citations

Bertholet, Ambre M., Aurélie Millet, Manon Moulis, et al.. (2015). Mitochondrial fusion/fission dynamics in neurodegeneration and neuronal plasticity. Neurobiology of Disease. 90. 3–19. 287 indexed citations

Gonzalvez, François, Marilena D’Aurelio, Marie Boutant, et al.. (2013). Barth syndrome: Cellular compensation of mitochondrial dysfunction and apoptosis inhibition due to changes in cardiolipin remodeling linked to tafazzin (TAZ) gene mutation. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(8). 1194–1206. 135 indexed citations

Rival, Thomas, Laetitia Pelloquin, Mickaël Poidevin, et al.. (2011). Inner‐membrane proteins PMI/TMEM11 regulate mitochondrial morphogenesis independently of the DRP1/MFN fission/fusion pathways. EMBO Reports. 12(3). 223–230. 35 indexed citations

Sauvanet, Cécile, Laetitia Pelloquin, Claudine David, Pascale Belenguer, & Manuel Rojo. (2010). Dynamique et morphologie mitochondriales. médecine/sciences. 26(10). 823–829. 9 indexed citations

10.

Diot, Alan, Marlène Daloyau, Laetitia Pelloquin, et al.. (2010). Processing of the dynamin Msp1p in S. pombe reveals an evolutionary switch between its orthologs Mgm1p in S. cerevisiae and OPA1 in mammals. FEBS Letters. 584(14). 3153–3157. 12 indexed citations

11.

Olichon, Aurélien, Emmanuelle Guillou, Cécile Delettre, et al.. (2006). Mitochondrial dynamics and disease, OPA1. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1763(5-6). 500–509. 176 indexed citations

12.

Olichon, Aurélien, Thomas Landes, Laetitia Pelloquin, et al.. (2006). Effects of OPA1 mutations on mitochondrial morphology and apoptosis: Relevance to ADOA pathogenesis. Journal of Cellular Physiology. 211(2). 423–430. 123 indexed citations

13.

Lenaers, Guy, Laetitia Pelloquin, Aurélien Olichon, et al.. (2002). What similarity between human and fission yeast proteins is required for orthology?. Yeast. 19(13). 1125–1126. 9 indexed citations

14.

Olichon, Aurélien, Laurent J. Emorine, Laetitia Pelloquin, et al.. (2002). The human dynamin‐related protein OPA1 is anchored to the mitochondrial inner membrane facing the inter‐membrane space. FEBS Letters. 523(1-3). 171–176. 344 indexed citations

15.

Delettre, Cécile, Guy Lenaers, Laetitia Pelloquin, Pascale Belenguer, & Christian P. Hamel. (2002). OPA1 (Kjer Type) Dominant Optic Atrophy: A Novel Mitochondrial Disease. Molecular Genetics and Metabolism. 75(2). 97–107. 144 indexed citations

16.

Delettre, Cécile, Guy Lenaers, Jean‐Michel Griffoin, et al.. (2000). Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy. Nature Genetics. 26(2). 207–210. 1131 indexed citations breakdown →

17.

Pelloquin, Laetitia, Pascale Belenguer, Yoann Menon, Nicole Gas, & Bernard Ducommun. (1999). Fission yeast msp1 is a mitochondrial dynamin-related protein. Journal of Cell Science. 112(22). 4151–4161. 57 indexed citations

18.

Pelloquin, Laetitia, Pascale Belenguer, Yoann Menon, & Bernard Ducommun. (1998). Identification of a Fission Yeast Dynamin-Related Protein Involved in Mitochondrial DNA Maintenance. Biochemical and Biophysical Research Communications. 251(3). 720–726. 68 indexed citations

19.

Belenguer, Pascale, et al.. (1997). Role of the Fission Yeast nim1 Protein Kinase in the Cell Cycle Response to Nutritional Signals. Biochemical and Biophysical Research Communications. 232(1). 204–208. 12 indexed citations

20.

Belenguer, Pascale, et al.. (1995). The fission yeast Nim1/Cdr1 kinase: A link between nutritional state and cell cycle control. PubMed. 1. 207–214. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact