Thomas Rival

1.3k total citations
18 papers, 1.0k citations indexed

About

Thomas Rival is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Pharmacology. According to data from OpenAlex, Thomas Rival has authored 18 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 10 papers in Cellular and Molecular Neuroscience and 4 papers in Pharmacology. Recurrent topics in Thomas Rival's work include Mitochondrial Function and Pathology (8 papers), Neurobiology and Insect Physiology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Thomas Rival is often cited by papers focused on Mitochondrial Function and Pathology (8 papers), Neurobiology and Insect Physiology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Thomas Rival collaborates with scholars based in France, United States and United Kingdom. Thomas Rival's co-authors include Jean-Charles Liévens, Laurent Soustelle, Bilal Khalil, Serge Birman, Serge Birman, Julien Royet, Karine Narbonne-Reveau, Bernard Charroux, Hervé Chneiweiss and Edward J. Ryder and has published in prestigious journals such as Nature Communications, Current Biology and Journal of Cell Science.

In The Last Decade

Thomas Rival

17 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Rival France	15	507	394	210	164	117	18	1.0k
Julide Bilen United States	8	744 1.5×	545 1.4×	133 0.6×	97 0.6×	125 1.1×	10	1.1k
Xianrong Mao United States	20	765 1.5×	331 0.8×	210 1.0×	156 1.0×	152 1.3×	25	1.7k
Namita Agrawal India	20	1.0k 2.0×	689 1.7×	119 0.6×	92 0.6×	159 1.4×	63	1.6k
Rhoda Stefanatos United Kingdom	17	881 1.7×	366 0.9×	230 1.1×	170 1.0×	62 0.5×	21	1.6k
Hideki Yoshida Japan	21	1.0k 2.0×	238 0.6×	78 0.4×	79 0.5×	141 1.2×	105	1.4k
S. Tariq Ahmad United States	12	389 0.8×	191 0.5×	227 1.1×	326 2.0×	216 1.8×	26	903
Ilaria Drago Italy	12	1.3k 2.6×	371 0.9×	246 1.2×	114 0.7×	54 0.5×	14	1.7k
Michael J. Palladino United States	24	1.3k 2.5×	283 0.7×	256 1.2×	67 0.4×	48 0.4×	47	1.8k
Stephen M. Altmann United States	6	526 1.0×	306 0.8×	270 1.3×	283 1.7×	253 2.2×	7	1.4k
Raphaël Courjaret Qatar	19	527 1.0×	281 0.7×	132 0.6×	61 0.4×	55 0.5×	38	1.0k

Countries citing papers authored by Thomas Rival

Since Specialization

Citations

This map shows the geographic impact of Thomas Rival's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Rival with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Rival more than expected).

Fields of papers citing papers by Thomas Rival

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Rival. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Rival. The network helps show where Thomas Rival may publish in the future.

Co-authorship network of co-authors of Thomas Rival

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Rival. A scholar is included among the top collaborators of Thomas Rival based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Rival. Thomas Rival is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Rival, Thomas, Aı̈cha Aouane, Nuno Miguel Luis, et al.. (2023). M1BP is an essential transcriptional activator of oxidative metabolism during Drosophila development. Nature Communications. 14(1). 3187–3187. 5 indexed citations

Daian, Fabrice, et al.. (2021). Myofibril and mitochondria morphogenesis are coordinated by a mechanical feedback mechanism in muscle. Nature Communications. 12(1). 2091–2091. 49 indexed citations

Rival, Thomas, Alice Carrier, Olga Corti, et al.. (2021). TP53INP1 exerts neuroprotection under ageing and Parkinson’s disease-related stress condition. Cell Death and Disease. 12(5). 460–460. 14 indexed citations

Crowther, Damian C., et al.. (2020). Using a Drosophila model of Alzheimer's disease. PubMed. 60. 57–77.

Rojo, Manuel, et al.. (2018). Mitofusin gain and loss of function drive pathogenesis in Drosophila models of CMT 2A neuropathy. EMBO Reports. 19(8). 62 indexed citations

Khalil, Bilal, et al.. (2017). Glial lipid droplets and neurodegeneration in a Drosophila model of complex I deficiency. Glia. 66(4). 874–888. 34 indexed citations

Khalil, Bilal, et al.. (2015). PINK1-induced mitophagy promotes neuroprotection in Huntington’s disease. Cell Death and Disease. 6(1). e1617–e1617. 186 indexed citations

Cassar, Marlène, Abdul-Raouf Issa, Thomas Riemensperger, et al.. (2014). A dopamine receptor contributes to paraquat-induced neurotoxicity in Drosophila. Human Molecular Genetics. 24(1). 197–212. 63 indexed citations

Tufi, Roberta, et al.. (2012). The Drosophila inner-membrane protein PMI controls cristae biogenesis and mitochondrial diameter. Journal of Cell Science. 126(Pt 3). 814–24. 18 indexed citations

10.

Rival, Thomas, Laetitia Pelloquin, Mickaël Poidevin, et al.. (2011). Inner‐membrane proteins PMI/TMEM11 regulate mitochondrial morphogenesis independently of the DRP1/MFN fission/fusion pathways. EMBO Reports. 12(3). 223–230. 35 indexed citations

11.

Conil, Jean‐Marie, Bernard Georges, Stéphanie Ruiz, et al.. (2010). Tobramycin disposition in ICU patients receiving a once daily regimen: population approach and dosage simulations. British Journal of Clinical Pharmacology. 71(1). 61–71. 33 indexed citations

12.

Rival, Thomas, Richard Page, Edward J. Ryder, et al.. (2009). Fenton chemistry and oxidative stress mediate the toxicity of the β‐amyloid peptide in a Drosophila model of Alzheimer’s disease. European Journal of Neuroscience. 29(7). 1335–1347. 144 indexed citations

13.

Charroux, Bernard, Thomas Rival, Karine Narbonne-Reveau, & Julien Royet. (2009). Bacterial detection by Drosophila peptidoglycan recognition proteins. Microbes and Infection. 11(6-7). 631–636. 67 indexed citations

14.

Rival, Thomas, et al.. (2006). Physiological requirement for the glutamate transporter dEAAT1 at the adult Drosophila neuromuscular junction. HAL (Le Centre pour la Communication Scientifique Directe). 1 indexed citations

15.

Rival, Thomas, et al.. (2006). Physiological requirement for the glutamate transporter dEAAT1 at the adult Drosophila neuromuscular junction. Journal of Neurobiology. 66(10). 1061–1074. 50 indexed citations

16.

Liévens, Jean-Charles, et al.. (2005). Expanded polyglutamine peptides disrupt EGF receptor signaling and glutamate transporter expression in Drosophila. Human Molecular Genetics. 14(5). 713–724. 74 indexed citations

17.

Rival, Thomas, et al.. (2004). Decreasing Glutamate Buffering Capacity Triggers Oxidative Stress and Neuropil Degeneration in the Drosophila Brain. Current Biology. 14(7). 599–605. 142 indexed citations

18.

Soustelle, Laurent, et al.. (2002). Terminal Glial Differentiation Involves Regulated Expression of the Excitatory Amino Acid Transporters in the Drosophila Embryonic CNS. Developmental Biology. 248(2). 294–306. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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