L. A. Sandkuyl

1.4k citations
29 papers · 1.1k indexed · h-index 18
Topics
Immunodeficiency and Autoimmune Disorders (8 papers)Chronic Lymphocytic Leukemia Research (4 papers)Blood disorders and treatments (2 papers)

In The Last Decade

L. A. Sandkuyl

29 papers receiving 1.0k citations

Peers

L. A. Sandkuyl
Comparison fields: 5 of 89
  • Molecular Biology 550
  • Genetics 297
  • Immunology 204
  • Genetics 182
  • Surgery 111
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F. Jouen France
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Teresa Mattina Italy
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Gökçe Törüner United States
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Citations per field
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Citations per year

Countries citing papers authored by L. A. Sandkuyl

Since Specialization
Citations

This map shows the geographic impact of L. A. Sandkuyl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L. A. Sandkuyl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L. A. Sandkuyl more than expected).

Fields of papers citing papers by L. A. Sandkuyl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by L. A. Sandkuyl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L. A. Sandkuyl. The network helps show where L. A. Sandkuyl may publish in the future.

Co-authorship network of co-authors of L. A. Sandkuyl

This figure shows the co-authorship network connecting the top 25 collaborators of L. A. Sandkuyl. A scholar is included among the top collaborators of L. A. Sandkuyl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with L. A. Sandkuyl. L. A. Sandkuyl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 23
2
Progress in the search for genetic linkage with Tourette syndrome: An exclusion map covering more than 50% of the autosomal genome
21
3 98
4 16
5 22
6
Diagnosis of Wiskott-Aldrich syndrome by analysis of the X chromosome inactivation patterns in maternal leucocyte populations using the hypervariable DXS255 locus.
21
7 28
8 42
9 50
10
Genetic mapping of X-linked albinism-deafness syndrome (ADFN) to Xq26.3-q27.I.
26
11 8
12 1
13 3
14 98
15 8
16 45
17 3
18 44
19 15
20 284

About L. A. Sandkuyl

L. A. Sandkuyl is a scholar working on Immunology, Developmental Biology and Genetics, having authored 29 papers that have together received 1.1k indexed citations. Recurring topics across this work include Immunodeficiency and Autoimmune Disorders (8 papers), Chronic Lymphocytic Leukemia Research (4 papers) and Blood disorders and treatments (2 papers). The work is most often cited by research in Genetics (182 citations), Genetics (297 citations) and Immunology (204 citations). L. A. Sandkuyl has collaborated with scholars based in Netherlands, United States and United Kingdom. Frequent co-authors include M. G. J. Jahoda, E. S. Sachs, Ruud K. B. Schuurman, H.F. Willard, P. Pearson, A. Thompson, Klaus Wrogemann, Egbert Bakker, E. J. B. M. Mensink and A.J. van Essen. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Lancet and The EMBO Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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