J. Amos

1.1k total citations
21 papers, 814 citations indexed

About

J. Amos is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Genetics. According to data from OpenAlex, J. Amos has authored 21 papers receiving a total of 814 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Pulmonary and Respiratory Medicine and 4 papers in Genetics. Recurrent topics in J. Amos's work include Cystic Fibrosis Research Advances (5 papers), Tracheal and airway disorders (4 papers) and Tuberous Sclerosis Complex Research (3 papers). J. Amos is often cited by papers focused on Cystic Fibrosis Research Advances (5 papers), Tracheal and airway disorders (4 papers) and Tuberous Sclerosis Complex Research (3 papers). J. Amos collaborates with scholars based in United States, United Kingdom and Canada. J. Amos's co-authors include John E. Mickle, Robert D. Oates, Aubrey Milunsky, W. Edward Highsmith, Claire Davis, Siobhan M. Cashman, Colin A. Graham, Milan Maçek, David R. Witt and S M Curristin and has published in prestigious journals such as Nature Genetics, Annals of the New York Academy of Sciences and The American Journal of Human Genetics.

In The Last Decade

J. Amos

21 papers receiving 777 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
J. Amos United States	12	363	273	189	105	93	21	814
John E. Mickle United States	11	841 2.3×	499 1.8×	161 0.9×	94 0.9×	149 1.6×	12	1.4k
Joy B. Redman United States	17	290 0.8×	301 1.1×	203 1.1×	71 0.7×	31 0.3×	27	781
Thomas A. Maher United States	13	137 0.4×	270 1.0×	147 0.8×	58 0.6×	112 1.2×	23	586
Valérie Drouin‐Garraud France	16	225 0.6×	492 1.8×	143 0.8×	24 0.2×	54 0.6×	32	875
Uta Grieshammer United States	12	172 0.5×	874 3.2×	232 1.2×	141 1.3×	92 1.0×	14	1.0k
Rosemarie Smith United States	11	105 0.3×	409 1.5×	336 1.8×	63 0.6×	151 1.6×	21	744
Christopher R. Futtner United States	12	195 0.5×	695 2.5×	232 1.2×	75 0.7×	80 0.9×	16	983
Valère Cacheux France	10	166 0.5×	415 1.5×	417 2.2×	70 0.7×	128 1.4×	12	847
Sumita Danda India	16	88 0.2×	489 1.8×	269 1.4×	38 0.4×	70 0.8×	122	1.0k
Sirpa Ala‐Mello Finland	17	88 0.2×	498 1.8×	468 2.5×	212 2.0×	187 2.0×	29	1.2k

Countries citing papers authored by J. Amos

Since Specialization

Citations

This map shows the geographic impact of J. Amos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Amos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Amos more than expected).

Fields of papers citing papers by J. Amos

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Amos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Amos. The network helps show where J. Amos may publish in the future.

Co-authorship network of co-authors of J. Amos

This figure shows the co-authorship network connecting the top 25 collaborators of J. Amos. A scholar is included among the top collaborators of J. Amos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Amos. J. Amos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Amos, J., Linda Huang, Julien Thévenon, et al.. (2016). Autosomal recessive mutations in THOC6 cause intellectual disability: syndrome delineation requiring forward and reverse phenotyping. Clinical Genetics. 91(1). 92–99. 16 indexed citations

Monaghan, Kristin G., W. Edward Highsmith, J. Amos, et al.. (2004). Genotype-phenotype correlation and frequency of the 3199del6 cystic fibrosis mutation among I148T carriers: Results from a collaborative study. Genetics in Medicine. 6(5). 421–425. 24 indexed citations

Mickle, John E., Aubrey Milunsky, J. Amos, & Robert D. Oates. (1995). Immunology: Congenital unilateral absence of the vas deferens: a heterogeneous disorder with two distinct subpopulations based upon aetiology and mutational status of the cystic fibrosis gene. Human Reproduction. 10(7). 1728–1735. 90 indexed citations

Hamosh, Ada, Garry R. Cutting, Robert D. Oates, & J. Amos. (1994). Correlation of the level of full-length CFTR transcript with pulmonary phenotype in patients carrying R117H and 1342-1,-2delAG mutations. The American Journal of Human Genetics. 55. 2 indexed citations

Torfs, Claudine P., et al.. (1994). Uniparental disomy (UPD) for fra(X) in a 47,XXY male. The American Journal of Human Genetics. 55. 1 indexed citations

Oates, Robert D. & J. Amos. (1993). Congenital bilateral absence of the vas deferens and cystic fibrosis. World Journal of Urology. 11(2). 1328–8. 26 indexed citations

Maçek, Milan, Claire Davis, S M Curristin, et al.. (1993). A mutation in CFTR produces different phenotypes depending on chromosomal background. Nature Genetics. 5(3). 274–278. 335 indexed citations

Farrer, Lindsay A., et al.. (1992). Waardenburg syndrome (WS) type I is caused by defects at multiple loci, one of which is near ALPP on chromosome 2: first report of the WS consortium.. PubMed. 50(5). 902–13. 143 indexed citations

Povey, Sue, Mari‐Wyn Burley, M. Smith, et al.. (1991). An Attempt to Map Two Genes for Tuberous Sclerosis Using Novel Two‐Point Methods^a. Annals of the New York Academy of Sciences. 615(1). 298–305. 16 indexed citations

10.

Povey, Sue, L. A. Sandkuyl, Dick Lindhout, et al.. (1991). A Comparative Study on Genetic Heterogeneity in Tuberous Sclerosis: Evidence for One Gene on 9q34 and a Second Gene on 11q22–23^a. Annals of the New York Academy of Sciences. 615(1). 306–315. 22 indexed citations

11.

Short, M. Priscilla, Jess Haines, Ann Jewell, et al.. (1991). Clinical Findings and Linkage Studies in Familial Tuberous Sclerosis. Annals of the New York Academy of Sciences. 615(1). 380–381. 2 indexed citations

12.

Haines, Jonathan L., M. Priscilla Short, D J Kwiatkowski, et al.. (1991). Localization of one gene for tuberous sclerosis within 9q32-9q34, and further evidence for heterogeneity.. PubMed. 49(4). 764–72. 53 indexed citations

13.

Amos, J., et al.. (1990). DNA analysis of cystic fibrosis genotypes in relatives with equivocal sweat test results.. PubMed. 13(1). 1–5. 2 indexed citations

14.

Kitsiou, Sofìa, et al.. (1988). [Polymorphism of restriction fragment length in the detection of the precise status of monosomy 21 in a deformed retarded girl].. PubMed. 36(1-2). 99–102. 1 indexed citations

15.

Phelan, Mary C., Cynthia C. Morton, Roger E. Stevenson, et al.. (1988). Molecular and cytogenetic characterization of a de novo t(5p;21q) in a patient previously diagnosed as monosomy 21.. PubMed. 43(4). 511–9. 25 indexed citations

16.

Amos, J., et al.. (1980). Isolation of Doubled-Haploid Plants Through Anther Culture in Arabidopsis thaliana. Zeitschrift für Pflanzenphysiologie. 96(5). 407–414. 8 indexed citations

17.

Amos, J., et al.. (1978). Induction of Haploid Callus from Anthers of Four Species of Arabidopsis. Zeitschrift für Pflanzenphysiologie. 90(1). 33–43. 14 indexed citations

18.

Amos, J.. (1960). POLYCYTHAEMIA VERA AND CHRISTMAS DISEASE WITH SLIGHTLY PROLONGED PROTHROMBIN TIME. Journal of Clinical Pathology. 13(4). 299–303. 1 indexed citations

19.

Amos, J. & R. A. Goodbody. (1959). Lymph Node and Liver Biopsy in the Myeloproliferative Disorders. British Journal of Cancer. 13(2). 173–180. 1 indexed citations

20.

Amos, J.. (1958). Thrombosis of the Major Pulmonary Arteries. BMJ. 2(5097). 659–662. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact