Kui Lin

2.4k total citations · 1 hit paper
15 papers, 1.2k citations indexed

About

Kui Lin is a scholar working on Molecular Biology, Oncology and Organic Chemistry. According to data from OpenAlex, Kui Lin has authored 15 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 4 papers in Oncology and 2 papers in Organic Chemistry. Recurrent topics in Kui Lin's work include PI3K/AKT/mTOR signaling in cancer (6 papers), Click Chemistry and Applications (2 papers) and Ubiquitin and proteasome pathways (2 papers). Kui Lin is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (6 papers), Click Chemistry and Applications (2 papers) and Ubiquitin and proteasome pathways (2 papers). Kui Lin collaborates with scholars based in United States, France and Spain. Kui Lin's co-authors include Cynthia Kenyon, Honor Hsin, Jie Lin, Geng Wu, Wen‐I Wu, Lori S. Friedman, Guy Vigers, Tony Morales, Brian B. Lee and Joshua A. Ballard and has published in prestigious journals such as Nature Communications, Nature Genetics and The Journal of Experimental Medicine.

In The Last Decade

Kui Lin

15 papers receiving 1.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Regulation of the Caenorhabditis elegans longevity protein DAF-16 by insulin/IGF-1 and germline signaling

2001 829 citations Kui Lin, Honor Hsin et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kui Lin United States	8	701	677	310	218	89	15	1.2k
Haralambos Hadjivassiliou United States	8	451 0.6×	434 0.6×	172 0.6×	201 0.9×	53 0.6×	10	814
Christian G. Riedel Sweden	16	424 0.6×	1.0k 1.5×	103 0.3×	133 0.6×	49 0.6×	33	1.4k
Florence Solari France	19	415 0.6×	645 1.0×	137 0.4×	238 1.1×	70 0.8×	25	1.0k
Ekaterina Plyusnina Russia	12	277 0.4×	402 0.6×	42 0.1×	163 0.7×	34 0.4×	17	782
Lear E. Brace United States	10	140 0.2×	712 1.1×	79 0.3×	312 1.4×	115 1.3×	15	1.3k
Juan Carlos Fierro-González Sweden	12	216 0.3×	521 0.8×	60 0.2×	66 0.3×	73 0.8×	14	869
Shaosong Yang United States	12	120 0.2×	745 1.1×	43 0.1×	105 0.5×	98 1.1×	13	1.1k
V. Pragathi Masamsetti Australia	9	190 0.3×	632 0.9×	24 0.1×	136 0.6×	110 1.2×	15	936
Jarrett R. Remsberg United States	15	50 0.1×	482 0.7×	203 0.7×	191 0.9×	75 0.8×	17	830
Nargis Nasrin United States	15	151 0.2×	996 1.5×	29 0.1×	310 1.4×	119 1.3×	25	1.5k

Countries citing papers authored by Kui Lin

Since Specialization

Citations

This map shows the geographic impact of Kui Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kui Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kui Lin more than expected).

Fields of papers citing papers by Kui Lin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kui Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kui Lin. The network helps show where Kui Lin may publish in the future.

Co-authorship network of co-authors of Kui Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Kui Lin. A scholar is included among the top collaborators of Kui Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kui Lin. Kui Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

15 of 15 papers shown

Lin, Kui, et al.. (2025). Profiling and Optimizing Targeted Covalent Inhibitors through EGFR-Guided Studies. Journal of Medicinal Chemistry. 68(16). 17917–17932. 1 indexed citations

Schmid, Peter, Delphine Loirat, Peter Savas, et al.. (2021). Abstract PD14-03: Molecular mechanism of ipatasertib (IPAT) and its combination with atezolizumab (atezo) in patients (pts) with locally advanced/metastatic triple-negative breast cancer (aTNBC). Cancer Research. 81(4_Supplement). PD14–3. 1 indexed citations

Yamazoe, Sayumi, Jeffrey Tom, Yue Fu, et al.. (2019). Heterobifunctional Molecules Induce Dephosphorylation of Kinases–A Proof of Concept Study. Journal of Medicinal Chemistry. 63(6). 2807–2813. 100 indexed citations

Lin, Kui & Geng Wu. (2018). Isothermal Titration Calorimetry Assays to Measure Binding Affinities In Vitro. Methods in molecular biology. 1893. 257–272. 27 indexed citations

Lin, Kui, Maxine V. Holder, Zhongchao Gai, et al.. (2017). Stable MOB1 interaction with Hippo/MST is not essential for development and tissue growth control. Nature Communications. 8(1). 695–695. 33 indexed citations

Lin, Kui. (2013). The Akt DUBbed InAktive. Science Signaling. 6(257). pe1–pe1. 6 indexed citations

Nannini, Michelle, Rebecca Hong, Brian B. Lee, et al.. (2012). Abstract 873: Combined targeting of Akt and MEK with the Akt inhibitor GDC-0068 and MEK inhibitor GDC-0973 demonstrates synergistic anti-tumor effects. Cancer Research. 72(8_Supplement). 873–873. 2 indexed citations

Savage, Heidi, et al.. (2012). Abstract 966: Predictive biomarkers of the AKT inhibitor, GDC-0068, in single agent and combination studies. Cancer Research. 72(8_Supplement). 966–966. 2 indexed citations

Lin, Kui, Jie Lin, Wen‐I Wu, et al.. (2012). An ATP-Site On-Off Switch That Restricts Phosphatase Accessibility of Akt. Science Signaling. 5(223). ra37–ra37. 132 indexed citations

10.

Lin, Kui, James R. Taylor, Thomas D. Wu, et al.. (2011). TMEFF2 Is a PDGF-AA Binding Protein with Methylation-Associated Gene Silencing in Multiple Cancer Types Including Glioma. PLoS ONE. 6(4). e18608–e18608. 38 indexed citations

11.

Lin, Kui. (2011). Abstract DDT02-01: GDC-0068: A novel, selective, ATP-competitive inhibitor of Akt. Cancer Research. 71(8_Supplement). DDT02–1. 2 indexed citations

12.

Yan, Yibing, Marie-Claire Wagle, Elizabeth A. Punnoose, et al.. (2011). Abstract B154: A first-in-human trial of GDC-0068: A novel, oral, ATP-competitive Akt inhibitor, demonstrates robust suppression of the Akt pathway in surrogate and tumor tissues.. Molecular Cancer Therapeutics. 10(11_Supplement). B154–B154. 5 indexed citations

13.

Lin, Kui, et al.. (2001). Regulation of the Caenorhabditis elegans longevity protein DAF-16 by insulin/IGF-1 and germline signaling. Nature Genetics. 28(2). 139–145. 829 indexed citations breakdown →

14.

Lin, Kui, Nancy S. Longo, Xin Wang, J. Hewitt, & Kristin M. Abraham. (2000). Lck Domains Differentially Contribute to Pre–T Cell Receptor (Tcr)–And TCR-α/β–Regulated Developmental Transitions. The Journal of Experimental Medicine. 191(4). 703–716. 15 indexed citations

15.

Lin, Kui. (1997). Targets of p56(lck) activity in immature thymoblasts: stimulation of the Ras/Raf/MAPK pathway. International Immunology. 9(2). 291–306. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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