Kristian P. Doyle
- Neurology top 0.5%
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 5%
- Immunology top 10%
- Epidemiology top 10%
- Co-authors
- Mary P. Stenzel‐PooreRoger P. SimonMarion S. BuckwalterEgle CekanaviciuteThuy-Vi V. NguyenLauren MamerNikola LessovNancy Fathali
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (26 papers)Neurological Disease Mechanisms and Treatments (11 papers)Neurogenesis and neuroplasticity mechanisms (7 papers)
- Partner nations
- United StatesGermanySpain
In The Last Decade
Kristian P. Doyle
43 papers receiving 2.6k citations
Hit Papers
Peers
Comparison fields: 5 of 119
- Neurology 1.2k
- Molecular Biology 878
- Cellular and Molecular Neuroscience 447
- Immunology 390
- Epidemiology 351
Countries citing papers authored by Kristian P. Doyle
This map shows the geographic impact of Kristian P. Doyle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kristian P. Doyle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kristian P. Doyle more than expected).
Fields of papers citing papers by Kristian P. Doyle
This network shows the impact of papers produced by Kristian P. Doyle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kristian P. Doyle. The network helps show where Kristian P. Doyle may publish in the future.
Co-authorship network of co-authors of Kristian P. Doyle
This figure shows the co-authorship network connecting the top 25 collaborators of Kristian P. Doyle. A scholar is included among the top collaborators of Kristian P. Doyle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kristian P. Doyle. Kristian P. Doyle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 1 | |
| 4 | 1 | |
| 5 | 1 | |
| 6 | 6 | |
| 7 | 8 | |
| 8 | 1 | |
| 9 | 9 | |
| 10 | 19 | |
| 11 | 15 | |
| 12 | 52 | |
| 13 | 94 | |
| 14 | 32 | |
| 15 | 30 | |
| 16 | 263 | |
| 17 | 10 | |
| 18 | 50 | |
| 19 | 40 | |
| 20 | 194 |
About Kristian P. Doyle
Kristian P. Doyle is a scholar working on Neurology, Developmental Neuroscience and Biological Psychiatry, having authored 45 papers that have together received 2.6k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (26 papers), Neurological Disease Mechanisms and Treatments (11 papers) and Neurogenesis and neuroplasticity mechanisms (7 papers). The work is most often cited by research in Neurology (1.2k citations), Developmental Neuroscience (225 citations) and Biological Psychiatry (94 citations). Kristian P. Doyle has collaborated with scholars based in United States, Germany and Spain. Frequent co-authors include Mary P. Stenzel‐Poore, Roger P. Simon, Marion S. Buckwalter, Egle Cekanaviciute, Thuy-Vi V. Nguyen, Lauren Mamer, Nikola Lessov, Nancy Fathali, Frank M. Longo and Susan L. Stevens. Their work appears in journals such as Journal of Neuroscience, The Journal of Immunology and Brain.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.