Natalie E. Scholpa

653 total citations
30 papers, 498 citations indexed

About

Natalie E. Scholpa is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Emergency Medicine. According to data from OpenAlex, Natalie E. Scholpa has authored 30 papers receiving a total of 498 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Pathology and Forensic Medicine and 5 papers in Emergency Medicine. Recurrent topics in Natalie E. Scholpa's work include Spinal Cord Injury Research (9 papers), Mitochondrial Function and Pathology (5 papers) and Cardiac Arrest and Resuscitation (4 papers). Natalie E. Scholpa is often cited by papers focused on Spinal Cord Injury Research (9 papers), Mitochondrial Function and Pathology (5 papers) and Cardiac Arrest and Resuscitation (4 papers). Natalie E. Scholpa collaborates with scholars based in United States, France and Türkiye. Natalie E. Scholpa's co-authors include Rick G. Schnellmann, Brian S. Cummings, Daniel Corum, Douglas G. Tilley, Patrick G. Sullivan, Heather A. Boger, Stephen Tomlinson, Alexander G. Rabchevsky, John J. Wagner and Wenxue Wang and has published in prestigious journals such as International Journal of Molecular Sciences, Kidney International and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Natalie E. Scholpa

27 papers receiving 493 citations

Peers

Natalie E. Scholpa
Xiaochen Yuan China
Weibin Lin China
Libing Ye China
Zhongkai Fan China
Qichao Wu China
Donghe Han China
Gang Lv China
An Xie China
Xiaochen Yuan China
Natalie E. Scholpa
Citations per year, relative to Natalie E. Scholpa Natalie E. Scholpa (= 1×) peers Xiaochen Yuan

Countries citing papers authored by Natalie E. Scholpa

Since Specialization
Citations

This map shows the geographic impact of Natalie E. Scholpa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalie E. Scholpa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalie E. Scholpa more than expected).

Fields of papers citing papers by Natalie E. Scholpa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Natalie E. Scholpa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalie E. Scholpa. The network helps show where Natalie E. Scholpa may publish in the future.

Co-authorship network of co-authors of Natalie E. Scholpa

This figure shows the co-authorship network connecting the top 25 collaborators of Natalie E. Scholpa. A scholar is included among the top collaborators of Natalie E. Scholpa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natalie E. Scholpa. Natalie E. Scholpa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Thompson, Austin, et al.. (2025). MC16 promotes mitochondrial biogenesis and ameliorates acute and diabetic nephropathy. British Journal of Pharmacology. 182(9). 1912–1929. 1 indexed citations
2.
Scholpa, Natalie E., et al.. (2025). MARY1 restores mitochondrial homeostasis and accelerates renal recovery following acute kidney injury. American Journal of Physiology-Renal Physiology. 329(4). F411–F421.
3.
Thompson, Austin, et al.. (2025). 5-Hydroxytryptamine 1F receptor loss reduces renal vasculature and prevents lasmiditan-induced recovery following moderate-severe acute kidney injury in mice. American Journal of Physiology-Renal Physiology. 329(6). F834–F852.
4.
Thompson, Austin, et al.. (2025). Lasmiditan induces mitochondrial biogenesis in primary mouse renal peritubular endothelial cells and augments wound healing and tubular network formation. American Journal of Physiology-Cell Physiology. 328(4). C1318–C1332. 1 indexed citations
5.
Thompson, Austin, et al.. (2025). Repurposing mitochondria-targeted therapeutics for kidney diseases. Kidney International. 107(4). 617–627. 2 indexed citations
6.
Liktor‐Busa, Erika, Kelly L. Karlage, Sheng‐Joue Young, et al.. (2024). Formoterol dynamically alters endocannabinoid tone in the periaqueductal gray inducing headache. The Journal of Headache and Pain. 25(1). 200–200.
7.
Scholpa, Natalie E., Jennifer B. Frye, Sanna Loppi, et al.. (2024). Formoterol alters chemokine expression and ameliorates pain behaviors after moderate spinal cord injury in female mice. Journal of Pharmacology and Experimental Therapeutics. 392(2). 100015–100015. 1 indexed citations
8.
Scholpa, Natalie E., et al.. (2024). Serotonin regulation of mitochondria in kidney diseases. Pharmacological Research. 203. 107154–107154. 8 indexed citations
9.
Scholpa, Natalie E., et al.. (2024). Evolution of Lipid Metabolism in the Injured Mouse Spinal Cord. Journal of Neurotrauma. 42(3-4). 182–196. 3 indexed citations
10.
Thompson, Austin, et al.. (2024). Isolation and monoculture of functional primary astrocytes from the adult mouse spinal cord. Frontiers in Neuroscience. 18. 1367473–1367473. 1 indexed citations
11.
Scholpa, Natalie E., et al.. (2024). 5-HT1F receptor agonism induces mitochondrial biogenesis and increases cellular function in brain microvascular endothelial cells. Frontiers in Cellular Neuroscience. 18. 1365158–1365158. 4 indexed citations
12.
Loppi, Sanna, et al.. (2023). Boosting Mitochondrial Biogenesis Diminishes Foam Cell Formation in the Post-Stroke Brain. International Journal of Molecular Sciences. 24(23). 16632–16632. 6 indexed citations
13.
Scholpa, Natalie E.. (2023). Role of DNA methylation during recovery from spinal cord injury with and without β2-adrenergic receptor agonism. Experimental Neurology. 368. 114494–114494. 5 indexed citations
14.
Scholpa, Natalie E., et al.. (2020). Time-to-treatment window and cross-sex potential of β2-adrenergic receptor-induced mitochondrial biogenesis-mediated recovery after spinal cord injury. Toxicology and Applied Pharmacology. 411. 115366–115366. 19 indexed citations
15.
Scholpa, Natalie E., et al.. (2020). Mitochondrial biogenesis as a therapeutic target for traumatic and neurodegenerative CNS diseases. Experimental Neurology. 329. 113309–113309. 84 indexed citations
16.
Scholpa, Natalie E., et al.. (2019). 5-hydroxytryptamine 1F Receptor Agonist Induces Mitochondrial Biogenesis and Promotes Recovery from Spinal Cord Injury. Journal of Pharmacology and Experimental Therapeutics. 372(2). 216–223. 25 indexed citations
17.
Scholpa, Natalie E., Wenxue Wang, Daniel Corum, et al.. (2018). Pharmacological Stimulation of Mitochondrial Biogenesis Using the Food and Drug Administration-Approved β 2 -Adrenoreceptor Agonist Formoterol for the Treatment of Spinal Cord Injury. Journal of Neurotrauma. 36(6). 962–972. 47 indexed citations
18.
Scholpa, Natalie E. & Rick G. Schnellmann. (2017). Mitochondrial-Based Therapeutics for the Treatment of Spinal Cord Injury: Mitochondrial Biogenesis as a Potential Pharmacological Target. Journal of Pharmacology and Experimental Therapeutics. 363(3). 303–313. 90 indexed citations
19.
Scholpa, Natalie E., et al.. (2016). Cyclin-Dependent Kinase Inhibitor 1a (p21) Modulates Response to Cocaine and Motivated Behaviors. Journal of Pharmacology and Experimental Therapeutics. 357(1). 56–65. 11 indexed citations
20.
Scholpa, Natalie E., et al.. (2014). Epigenetic Changes in p21 Expression in Renal Cells after Exposure to Bromate. Toxicological Sciences. 141(2). 432–440. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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