Koichi Isogawa

1.5k total citations
40 papers, 938 citations indexed

About

Koichi Isogawa is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Koichi Isogawa has authored 40 papers receiving a total of 938 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Behavioral Neuroscience, 15 papers in Cellular and Molecular Neuroscience and 12 papers in Molecular Biology. Recurrent topics in Koichi Isogawa's work include Stress Responses and Cortisol (21 papers), Tryptophan and brain disorders (11 papers) and Receptor Mechanisms and Signaling (10 papers). Koichi Isogawa is often cited by papers focused on Stress Responses and Cortisol (21 papers), Tryptophan and brain disorders (11 papers) and Receptor Mechanisms and Signaling (10 papers). Koichi Isogawa collaborates with scholars based in Japan and United States. Koichi Isogawa's co-authors include Jotaro Akiyoshi, Takashi Tsutsumi, Haruo Nagayama, Hiroaki Hanada, Masayuki Kanehisa, Hiroshi Kunugi, Akira Sano, Kensuke Kodama, Yukiko Yamamoto and Shinichiro Nanko and has published in prestigious journals such as Biological Psychiatry, Journal of Neurochemistry and Psychiatry Research.

In The Last Decade

Koichi Isogawa

39 papers receiving 908 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Koichi Isogawa Japan 19 323 301 166 152 147 40 938
Judy McIntosh Canada 16 293 0.9× 389 1.3× 78 0.5× 113 0.7× 110 0.7× 22 971
Maya Lebow Israel 10 284 0.9× 362 1.2× 111 0.7× 64 0.4× 109 0.7× 12 889
Elin Åberg Sweden 14 239 0.7× 223 0.7× 149 0.9× 127 0.8× 48 0.3× 17 833
Lisa Y. Maeng United States 13 148 0.5× 387 1.3× 147 0.9× 98 0.6× 148 1.0× 18 894
Isabella J.E. Heuser United States 13 381 1.2× 635 2.1× 114 0.7× 132 0.9× 62 0.4× 19 1.6k
Héctor González‐Pardo Spain 22 414 1.3× 333 1.1× 65 0.4× 158 1.0× 71 0.5× 66 1.2k
Jessica A. Babb United States 17 291 0.9× 592 2.0× 100 0.6× 176 1.2× 116 0.8× 24 1.3k
Zhifeng Zhou China 17 392 1.2× 123 0.4× 131 0.8× 98 0.6× 52 0.4× 38 1.1k
Hiroshi Kawahara Japan 14 521 1.6× 162 0.5× 61 0.4× 106 0.7× 134 0.9× 40 942
Sandra Luz United States 13 132 0.4× 370 1.2× 66 0.4× 116 0.8× 218 1.5× 19 894

Countries citing papers authored by Koichi Isogawa

Since Specialization
Citations

This map shows the geographic impact of Koichi Isogawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Koichi Isogawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Koichi Isogawa more than expected).

Fields of papers citing papers by Koichi Isogawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Koichi Isogawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Koichi Isogawa. The network helps show where Koichi Isogawa may publish in the future.

Co-authorship network of co-authors of Koichi Isogawa

This figure shows the co-authorship network connecting the top 25 collaborators of Koichi Isogawa. A scholar is included among the top collaborators of Koichi Isogawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Koichi Isogawa. Koichi Isogawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tsuru, Jusen, Yoshinobu Ishitobi, Taiga Ninomiya, et al.. (2013). The thyrotropin-releasing hormone test may predict recurrence of clinical depression within ten years after discharge.. PubMed. 34(5). 409–17. 9 indexed citations
2.
Ishitobi, Yoshinobu, Kentaro Kohno, Masayuki Kanehisa, et al.. (2012). Serum Ghrelin Levels and the Effects of Antidepressants in Major Depressive Disorder and Panic Disorder. Neuropsychobiology. 66(3). 185–192. 41 indexed citations
3.
Ishitobi, Yoshinobu, Shinya Nakayama, Masayuki Kanehisa, et al.. (2012). Association of CRHR1 and CRHR2 with major depressive disorder and panic disorder in a Japanese population. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 159B(4). 429–436. 55 indexed citations
4.
Tanaka, Yoshihiro, Yoshinobu Ishitobi, Yoshihiro Maruyama, et al.. (2012). Salivary alpha-amylase and cortisol responsiveness following electrical stimulation stress in panic disorder patients. Neuroscience Research. 73(1). 80–84. 33 indexed citations
5.
Higuma, Haruka, Masayuki Kanehisa, Yoshihiro Maruyama, et al.. (2012). Aripiprazole augmentation in 13 patients with refractory obsessive-compulsive disorder: A case series. The World Journal of Biological Psychiatry. 13(1). 14–21. 9 indexed citations
6.
7.
Tanaka, Yoshihiro, Yoshinobu Ishitobi, Yoshihiro Maruyama, et al.. (2011). Salivary alpha-amylase and cortisol responsiveness following electrical stimulation stress in major depressive disorder patients. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 36(2). 220–224. 38 indexed citations
8.
Ishitobi, Yoshinobu, Jotaro Akiyoshi, Taiga Ninomiya, et al.. (2011). Administration of antisense DNA for GPR39-1b causes anxiolytic-like responses and appetite loss in rats. Neuroscience Research. 72(3). 257–262. 13 indexed citations
9.
Ishitobi, Yoshinobu, Jotaro Akiyoshi, Yoshihiro Tanaka, et al.. (2010). Elevated salivary α-amylase and cortisol levels in unremitted and remitted depressed patients. International Journal of Psychiatry in Clinical Practice. 14(4). 268–273. 31 indexed citations
10.
11.
Akiyoshi, Jotaro, et al.. (2007). Administration of antisense DNA for hepatocyte growth factor causes an depressive and anxiogenic response in rats. Neuropeptides. 41(6). 477–483. 7 indexed citations
12.
Tsuchiyama, Kounosuke, et al.. (2005). Predicting efficacy of electroconvulsive therapy in major depressive disorder. Psychiatry and Clinical Neurosciences. 59(5). 546–550. 13 indexed citations
13.
14.
Akiyoshi, Jotaro, Nobuyoshi Ishii, Kensuke Kodama, et al.. (2003). Spatial memory impairment in OLETF rats without cholecystokinin – a receptor. Neuropeptides. 37(5). 271–276. 22 indexed citations
15.
Akiyoshi, Jotaro, et al.. (2003). Association between the CCK‐A receptor gene and panic disorder. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 118B(1). 29–31. 23 indexed citations
16.
17.
Kunugi, Hiroshi, Akira Sano, Takashi Tsutsumi, et al.. (1999). Association between serotonin transporter gene polymorphism and anxiety-related traits. Biological Psychiatry. 45(3). 368–370. 162 indexed citations
18.
Akiyoshi, Jotaro, et al.. (1998). Relationship between SCL-90, Maudsley Personality Inventory and CCK4-induced intracellular calcium response in T cells. Psychiatry Research. 81(3). 381–386. 5 indexed citations
19.
Yamamoto, Yukiko, et al.. (1997). Effect of CRH receptor antagonist CP-154, 526 on conditioned fear stress. 17(6). 284. 1 indexed citations
20.
Akiyoshi, Jotaro, Koichi Isogawa, Kenji Yamada, Haruo Nagayama, & Isao Fujii. (1996). Effects of antidepressants on intracellular Ca2+ mobilization in CHO cells transfected with the human 5-HT2C receptors. Biological Psychiatry. 39(12). 1000–1008. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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