Ko Sahashi

1.5k total citations
42 papers, 857 citations indexed

About

Ko Sahashi is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Ko Sahashi has authored 42 papers receiving a total of 857 indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 14 papers in Neurology and 11 papers in Cellular and Molecular Neuroscience. Recurrent topics in Ko Sahashi's work include Mitochondrial Function and Pathology (14 papers), Metabolism and Genetic Disorders (9 papers) and Genetic Neurodegenerative Diseases (9 papers). Ko Sahashi is often cited by papers focused on Mitochondrial Function and Pathology (14 papers), Metabolism and Genetic Disorders (9 papers) and Genetic Neurodegenerative Diseases (9 papers). Ko Sahashi collaborates with scholars based in Japan, United States and Taiwan. Ko Sahashi's co-authors include Tohru Ibi, Kinji Ohno, Naoki Nakao, Mikako Ito, Akira Takahashi, Andrew G. Engel, Hiroshi Nishida, Bisei Ohkawara, Akio Masuda and Yu Kawakami and has published in prestigious journals such as Brain, Neurology and Annals of Neurology.

In The Last Decade

Ko Sahashi

41 papers receiving 840 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ko Sahashi Japan 16 535 289 247 119 95 42 857
I. Butler United States 17 390 0.7× 208 0.7× 221 0.9× 148 1.2× 106 1.1× 34 855
Sumimasa Yamashita Japan 17 568 1.1× 103 0.4× 231 0.9× 99 0.8× 143 1.5× 53 994
Teeratorn Pulkes Thailand 15 586 1.1× 184 0.6× 153 0.6× 305 2.6× 98 1.0× 37 856
E. Pennisi Italy 16 308 0.6× 156 0.5× 153 0.6× 83 0.7× 174 1.8× 41 667
Nobuyoshi Fukuhara Japan 17 693 1.3× 139 0.5× 205 0.8× 261 2.2× 120 1.3× 35 1.1k
Linton C. Hopkins United States 14 729 1.4× 221 0.8× 168 0.7× 337 2.8× 150 1.6× 15 1.1k
Hideki Houzen Japan 14 234 0.4× 253 0.9× 101 0.4× 40 0.3× 105 1.1× 41 788
J. Colomer Spain 17 479 0.9× 213 0.7× 214 0.9× 50 0.4× 57 0.6× 42 934
M G Sweeney United Kingdom 18 1.0k 1.9× 128 0.4× 314 1.3× 577 4.8× 50 0.5× 21 1.2k
Kenji Mokuno Japan 17 416 0.8× 252 0.9× 237 1.0× 14 0.1× 108 1.1× 43 839

Countries citing papers authored by Ko Sahashi

Since Specialization
Citations

This map shows the geographic impact of Ko Sahashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ko Sahashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ko Sahashi more than expected).

Fields of papers citing papers by Ko Sahashi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ko Sahashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ko Sahashi. The network helps show where Ko Sahashi may publish in the future.

Co-authorship network of co-authors of Ko Sahashi

This figure shows the co-authorship network connecting the top 25 collaborators of Ko Sahashi. A scholar is included among the top collaborators of Ko Sahashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ko Sahashi. Ko Sahashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ito, Mikako, Bisei Ohkawara, Akio Masuda, et al.. (2015). Collagen Q and anti-MuSK autoantibody competitively suppress agrin/LRP4/MuSK signaling. Scientific Reports. 5(1). 13928–13928. 54 indexed citations
2.
Yamashita, Yoshihiro, Tohru Matsuura, Tatsuaki Kurosaki, et al.. (2014). LDB3 splicing abnormalities are specific to skeletal muscles of patients with myotonic dystrophy type 1 and alter its PKC binding affinity. Neurobiology of Disease. 69. 200–205. 26 indexed citations
3.
Kokunai, Yosuke, Keigo Goto, Tomoya Kubota, et al.. (2012). A sodium channel myotonia due to a novel SCN4A mutation accompanied by acquired autoimmune myasthenia gravis. Neuroscience Letters. 519(1). 67–72. 9 indexed citations
4.
5.
Kawakami, Yu, Mikako Ito, Masaaki Hirayama, et al.. (2011). Anti-MuSK autoantibodies block binding of collagen Q to MuSK. Neurology. 77(20). 1819–1826. 105 indexed citations
6.
Nishigaki, Yutaka, Hitomi Ueno, Jorida Çoku, et al.. (2010). Extensive screening system using suspension array technology to detect mitochondrial DNA point mutations. Mitochondrion. 10(3). 300–308. 24 indexed citations
7.
Hoshino, Akira, Yasushi Iwasaki, Shinya Kimura, et al.. (2008). MM1‐type sporadic Creutzfeldt‐Jakob disease with unusually prolonged disease duration presenting with panencephalopathic‐type pathology. Neuropathology. 28(3). 326–332. 5 indexed citations
8.
Nozaki, Hiroaki, Takeshi Ikeuchi, Akio Kawakami, et al.. (2007). Clinical and genetic characterizations of 16q‐linked autosomal dominant spinocerebellar ataxia (AD‐SCA) and frequency analysis of AD‐SCA in the Japanese population. Movement Disorders. 22(6). 857–862. 26 indexed citations
9.
Iwasaki, Yasushi, et al.. (2006). MM2-thalamic-type sporadic Creutzfeldt-Jakob disease with widespread neocortical pathology. Acta Neuropathologica. 112(4). 503–511. 23 indexed citations
10.
Naoi, Makoto, Wakako Maruyama, Naoki Nakao, et al.. (1998). (R) salsolinol N‐methyltransferase activity increases in parkinsonian lymphocytes. Annals of Neurology. 43(2). 212–216. 49 indexed citations
11.
Ikeuchi, Takeshi, Hiroki Takano, Reiji Koide, et al.. (1997). Spinocerebellar ataxia type 6: CAG repeat expansion in α1a voltage‐dependent calcium channel gene and clinical variations in japanese population. Annals of Neurology. 42(6). 879–884. 111 indexed citations
12.
Sahashi, Ko, et al.. (1997). [Clinico-pathological significance of the immunostaining of myosin heavy chain isoforms in pathological human skeletal muscle].. PubMed. 37(5). 366–73. 1 indexed citations
13.
Sahashi, Ko, et al.. (1997). [Immunohistochemical localization of chymase; a mast cell marker and clinical significance in diseased human skeletal muscle].. PubMed. 37(7). 615–20. 5 indexed citations
14.
Nakao, Naoki, Ko Sahashi, Masahiko Takahashi, Tohru Ibi, & Yoshio Hashizume. (1995). Clinical and neuropathological features of a neurodegenerative disorder in the central nervous system with progressive head drooping (Kubisagari). Acta Neuropathologica. 90(2). 208–212. 2 indexed citations
15.
Sahashi, Ko, et al.. (1995). Extensive Tissue Oxygenation Associated with Mitochondrial DNA Mutations. Biochemical and Biophysical Research Communications. 213(2). 432–438. 23 indexed citations
16.
Sahashi, Ko, et al.. (1992). Increased Mitochondrial DNA Deletions in the Skeletal Muscle of Myotonic Dystrophy. Gerontology. 38(1-2). 18–29. 20 indexed citations
17.
Ohno, Kinji, Masashi Tanaka, Ko Sahashi, et al.. (1991). Mitochondrial DNA deletions in inherited recurrent myoglobinuria. Annals of Neurology. 29(4). 364–369. 64 indexed citations
18.
Ohno, Kinji, Masashi Tanaka, Hidekazu Ino, et al.. (1991). Direct DNA sequencing from colony: analysis of multiple deletions of mitochondrial genome. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1090(1). 9–16. 6 indexed citations
19.
Sahashi, Ko, Kinji Ohno, Masashi Tanaka, et al.. (1990). Cytoplasmic body and mitochondrial DNA deletion. Journal of the Neurological Sciences. 99(2-3). 291–300. 4 indexed citations
20.
Mitsuma, Terunori, T Nogimori, Ko Sahashi, et al.. (1986). Thyrotropin Releasing Hormone Levels in Human Cerebrospinal Fluid in Various Neurologic Diseases. The American Journal of the Medical Sciences. 291(3). 164–167. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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