Kazutoshi Nakano
- Molecular Biology
- Neurology top 5%
- Cancer Research
- Cellular and Molecular Neuroscience top 10%
- Clinical Biochemistry top 5%
- Co-authors
- Robert HillRichard HaasClifford W. ShultsMary PayDavid C. WardGiovanni ManfrediJennifer Q. KwongShigeo Ohta
- Topics
- Metabolism and Genetic Disorders (8 papers)Mitochondrial Function and Pathology (7 papers)ATP Synthase and ATPases Research (3 papers)
- Partner nations
- JapanUnited StatesFrance
In The Last Decade
Kazutoshi Nakano
20 papers receiving 746 citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 495
- Neurology 216
- Cancer Research 132
- Cellular and Molecular Neuroscience 125
- Clinical Biochemistry 104
Countries citing papers authored by Kazutoshi Nakano
This map shows the geographic impact of Kazutoshi Nakano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazutoshi Nakano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazutoshi Nakano more than expected).
Fields of papers citing papers by Kazutoshi Nakano
This network shows the impact of papers produced by Kazutoshi Nakano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazutoshi Nakano. The network helps show where Kazutoshi Nakano may publish in the future.
Co-authorship network of co-authors of Kazutoshi Nakano
This figure shows the co-authorship network connecting the top 25 collaborators of Kazutoshi Nakano. A scholar is included among the top collaborators of Kazutoshi Nakano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kazutoshi Nakano. Kazutoshi Nakano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 26 | |
| 3 | 1 | |
| 4 | 24 | |
| 5 | 3 | |
| 6 | 8 | |
| 7 | 8 | |
| 8 | 234 | |
| 9 | 35 | |
| 10 | 17 | |
| 11 | A Preliminary Study of Methylcobalamin Therapy in Autism | 7 |
| 12 | 6 | |
| 13 | [A two-year-old infant with a myopathic form of very-long-chain Acyl-CoA dehydrogenase deficiency]. | 1 |
| 14 | 23 | |
| 15 | 5 | |
| 16 | 340 | |
| 17 | 15 | |
| 18 | 3 | |
| 19 | 1 | |
| 20 | 1 |
About Kazutoshi Nakano
Kazutoshi Nakano is a scholar working on Clinical Biochemistry, Obstetrics and Gynecology and Periodontics, having authored 21 papers that have together received 766 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (8 papers), Mitochondrial Function and Pathology (7 papers) and ATP Synthase and ATPases Research (3 papers). The work is most often cited by research in Clinical Biochemistry (104 citations), Neurology (216 citations) and Cancer Research (132 citations). Kazutoshi Nakano has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Robert Hill, Richard Haas, Clifford W. Shults, Mary Pay, David C. Ward, Giovanni Manfredi, Jennifer Q. Kwong, Shigeo Ohta, Takashi Kanamori and Hideaki Oda. Their work appears in journals such as Cancer Research, Annals of Neurology and The Journal of Pediatrics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.