Kim Bruggink

422 total citations
8 papers, 340 citations indexed

About

Kim Bruggink is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Kim Bruggink has authored 8 papers receiving a total of 340 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 4 papers in Molecular Biology and 2 papers in Neurology. Recurrent topics in Kim Bruggink's work include Alzheimer's disease research and treatments (6 papers), Prion Diseases and Protein Misfolding (1 paper) and Face Recognition and Perception (1 paper). Kim Bruggink is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Prion Diseases and Protein Misfolding (1 paper) and Face Recognition and Perception (1 paper). Kim Bruggink collaborates with scholars based in Netherlands and United States. Kim Bruggink's co-authors include Marcel M. Verbeek, H. Bea Kuiperij, Wesley Jongbloed, Jurgen A.H.R. Claassen, Mareike Müller, Rob Veerhuis, Alexandra A. M. Versleijen, Robert M.W. de Waal, Ilona B. Bruinsma and Vinod Subramaniam and has published in prestigious journals such as Analytical Biochemistry, Journal of Neurochemistry and Proteins Structure Function and Bioinformatics.

In The Last Decade

Kim Bruggink

8 papers receiving 336 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kim Bruggink Netherlands	6	212	179	64	51	48	8	340
Sean R. Kundinger United States	6	284 1.3×	310 1.7×	40 0.6×	53 1.0×	28 0.6×	6	468
Tamta Arakhamia United States	2	222 1.0×	189 1.1×	28 0.4×	41 0.8×	17 0.4×	2	294
Kellen Voss United States	8	266 1.3×	186 1.0×	55 0.9×	36 0.7×	11 0.2×	9	386
Omar M. Kashmer United States	7	220 1.0×	197 1.1×	50 0.8×	25 0.5×	11 0.2×	9	321
Clément Despres France	8	236 1.1×	263 1.5×	50 0.8×	35 0.7×	10 0.2×	10	408
Melissa Huang United Kingdom	4	277 1.3×	231 1.3×	28 0.4×	37 0.7×	12 0.3×	5	394
Michala Kolarova Czechia	5	263 1.2×	172 1.0×	31 0.5×	26 0.5×	22 0.5×	8	402
Satoko Wada‐Kakuda Japan	7	232 1.1×	169 0.9×	41 0.6×	27 0.5×	18 0.4×	8	315
Dailu Chen United States	7	283 1.3×	248 1.4×	31 0.5×	40 0.8×	12 0.3×	10	389
Jason C. Sang United Kingdom	10	200 0.9×	196 1.1×	48 0.8×	85 1.7×	9 0.2×	13	384

Countries citing papers authored by Kim Bruggink

Since Specialization

Citations

This map shows the geographic impact of Kim Bruggink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kim Bruggink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kim Bruggink more than expected).

Fields of papers citing papers by Kim Bruggink

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kim Bruggink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kim Bruggink. The network helps show where Kim Bruggink may publish in the future.

Co-authorship network of co-authors of Kim Bruggink

This figure shows the co-authorship network connecting the top 25 collaborators of Kim Bruggink. A scholar is included among the top collaborators of Kim Bruggink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kim Bruggink. Kim Bruggink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Jongbloed, Wesley, Kim Bruggink, Maartje I. Kester, et al.. (2015). Amyloid-β Oligomers Relate to Cognitive Decline in Alzheimer's Disease. Journal of Alzheimer s Disease. 45(1). 35–43. 49 indexed citations

2.

Bruggink, Kim, H. Bea Kuiperij, Jolein Gloerich, et al.. (2015). Dickkopf‐related protein 3 is a potential Aβ‐associated protein in Alzheimer's Disease. Journal of Neurochemistry. 134(6). 1152–1162. 26 indexed citations

3.

Bruggink, Kim, H. Bea Kuiperij, Jurgen A.H.R. Claassen, & Marcel M. Verbeek. (2013). The Diagnostic Value of CSF Amyloid-β<sub>43</sub> in Differentiation of Dementia Syndromes. Current Alzheimer Research. 10(10). 1034–1040. 9 indexed citations

4.

Bruggink, Kim, et al.. (2013). What Are the Chances? fMRI Correlates of Observing High and Low-Probability Actions. Journal of Behavioral and Brain Science. 3(1). 49–56. 1 indexed citations

5.

Bruggink, Kim, Wesley Jongbloed, Rob Veerhuis, et al.. (2012). Amyloid-β oligomer detection by ELISA in cerebrospinal fluid and brain tissue. Analytical Biochemistry. 433(2). 112–120. 99 indexed citations

6.

Bruggink, Kim, H. Bea Kuiperij, Wilbert C. Boelens, & Marcel M. Verbeek. (2012). P1‐020: Preparation and detection of complexes of beta‐amyloid with beta‐amyloid–associated proteins. Alzheimer s & Dementia. 8(4S_Part_3). 1 indexed citations

7.

Bruggink, Kim, Mareike Müller, H. Bea Kuiperij, & Marcel M. Verbeek. (2012). Methods for Analysis of Amyloid-β Aggregates. Journal of Alzheimer s Disease. 28(4). 735–758. 59 indexed citations

8.

Bruinsma, Ilona B., Kim Bruggink, Alexandra A. M. Versleijen, et al.. (2011). Inhibition of α‐synuclein aggregation by small heat shock proteins. Proteins Structure Function and Bioinformatics. 79(10). 2956–2967. 96 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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