Kennard L. Thomas

1.6k total citations
17 papers, 1.3k citations indexed

About

Kennard L. Thomas is a scholar working on Molecular Biology, Immunology and Allergy and Immunology. According to data from OpenAlex, Kennard L. Thomas has authored 17 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Immunology and Allergy and 6 papers in Immunology. Recurrent topics in Kennard L. Thomas's work include Cell Adhesion Molecules Research (6 papers), Retinal Diseases and Treatments (4 papers) and Microbial metabolism and enzyme function (3 papers). Kennard L. Thomas is often cited by papers focused on Cell Adhesion Molecules Research (6 papers), Retinal Diseases and Treatments (4 papers) and Microbial metabolism and enzyme function (3 papers). Kennard L. Thomas collaborates with scholars based in United States, Italy and Germany. Kennard L. Thomas's co-authors include Ali Hafezi‐Moghadam, Denisa D. Wagner, Klaus Ley, Alyson Prorock, Yuqing Huo, Jean-Christophe Plumier, Brent A. French, Chung-Ming Hsieh, Florian P. Limbourg and Michael A. Moskowitz and has published in prestigious journals such as Nature Medicine, The Journal of Experimental Medicine and The Journal of Immunology.

In The Last Decade

Kennard L. Thomas

16 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kennard L. Thomas United States	14	466	239	232	190	177	17	1.3k
Sigrid Hoffmann Germany	24	995 2.1×	142 0.6×	125 0.5×	327 1.7×	203 1.1×	63	2.0k
Yagna Jarajapu United States	22	658 1.4×	155 0.6×	317 1.4×	155 0.8×	147 0.8×	64	1.4k
Hosung Bae South Korea	17	670 1.4×	138 0.6×	215 0.9×	94 0.5×	75 0.4×	24	1.5k
A Leövey Hungary	19	179 0.4×	279 1.2×	99 0.4×	49 0.3×	268 1.5×	75	1.0k
Stefano Giannini Italy	25	682 1.5×	439 1.8×	261 1.1×	41 0.2×	595 3.4×	50	2.2k
Patrick G. Quinn United States	24	831 1.8×	114 0.5×	152 0.7×	454 2.4×	233 1.3×	38	2.0k
Tatsuo Kohriyama Japan	27	592 1.3×	204 0.9×	179 0.8×	19 0.1×	52 0.3×	104	2.2k
Mi Tian China	30	742 1.6×	426 1.8×	170 0.7×	409 2.2×	123 0.7×	103	2.5k
Paul L. Penar United States	26	519 1.1×	39 0.2×	208 0.9×	61 0.3×	37 0.2×	60	1.7k
Jean‐Sébastien Joyal Canada	24	807 1.7×	278 1.2×	134 0.6×	626 3.3×	30 0.2×	59	1.9k

Countries citing papers authored by Kennard L. Thomas

Since Specialization

Citations

This map shows the geographic impact of Kennard L. Thomas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kennard L. Thomas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kennard L. Thomas more than expected).

Fields of papers citing papers by Kennard L. Thomas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kennard L. Thomas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kennard L. Thomas. The network helps show where Kennard L. Thomas may publish in the future.

Co-authorship network of co-authors of Kennard L. Thomas

This figure shows the co-authorship network connecting the top 25 collaborators of Kennard L. Thomas. A scholar is included among the top collaborators of Kennard L. Thomas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kennard L. Thomas. Kennard L. Thomas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Almulki, L., Kousuke Noda, Shintaro Nakao, et al.. (2009). Localization of vascular adhesion protein-1 (VAP-1) in the human eye. Experimental Eye Research. 90(1). 26–32. 26 indexed citations

Noda, Kousuke, Haicheng She, Toru Nakazawa, et al.. (2008). Vascular adhesion protein‐1 blockade suppresses choroidal neovascularization. The FASEB Journal. 22(8). 2928–2935. 52 indexed citations

Skondra, Dimitra, Kousuke Noda, L. Almulki, et al.. (2008). Characterization of Azurocidin as a Permeability Factor in the Retina: Involvement in VEGF-Induced and Early Diabetic Blood-Retinal Barrier Breakdown. Investigative Ophthalmology & Visual Science. 49(2). 726–726. 28 indexed citations

Miyahara, Shinsuke, L. Almulki, Kousuke Noda, et al.. (2008). In vivo imaging of endothelial injury in choriocapillaris during endotoxin‐induced uveitis. The FASEB Journal. 22(6). 1973–1980. 13 indexed citations

Almulki, L., Kousuke Noda, Reza Amini, et al.. (2008). Surprising up‐regulation of P‐selectin glycoprotein ligand‐1 (PSGL‐1) in endotoxin‐induced uveitis. The FASEB Journal. 23(3). 929–939. 20 indexed citations

Noda, Kousuke, Shinsuke Miyahara, Toru Nakazawa, et al.. (2007). Inhibition of vascular adhesion protein‐1 suppresses endotoxin‐induced uveitis. The FASEB Journal. 22(4). 1094–1103. 50 indexed citations

Nakazawa, Toru, Akihisa Matsubara, Kousuke Noda, et al.. (2006). Characterization of cytokine responses to retinal detachment in rats.. PubMed. 12. 867–78. 141 indexed citations

Hafezi‐Moghadam, Ali, Kennard L. Thomas, & Denisa D. Wagner. (2006). ApoE deficiency leads to a progressive age-dependent blood-brain barrier leakage. American Journal of Physiology-Cell Physiology. 292(4). C1256–C1262. 131 indexed citations

Hafezi‐Moghadam, Ali, et al.. (2004). A novel mouse-driven ex vivo flow chamber for the study of leukocyte and platelet function. American Journal of Physiology-Cell Physiology. 286(4). C876–C892. 26 indexed citations

10.

Kondo, Tatsuya, Ali Hafezi‐Moghadam, Kennard L. Thomas, Denisa D. Wagner, & C.Ronald Kahn. (2004). Mice lacking insulin or insulin-like growth factor 1 receptors in vascular endothelial cells maintain normal blood–brain barrier. Biochemical and Biophysical Research Communications. 317(2). 315–320. 33 indexed citations

11.

Forlow, S. Bradley, et al.. (2003). T Cell Requirement for Development of Chronic Ulcerative Dermatitis in E- and P-Selectin-Deficient Mice. The Journal of Immunology. 170(1). 643–643. 1 indexed citations

12.

Forlow, S. Bradley, et al.. (2002). T Cell Requirement for Development of Chronic Ulcerative Dermatitis in E- and P-Selectin-Deficient Mice. The Journal of Immunology. 169(9). 4797–4804. 15 indexed citations

13.

Hafezi‐Moghadam, Ali, Tommaso Simoncini, Zequan Yang, et al.. (2002). Acute cardiovascular protective effects of corticosteroids are mediated by non-transcriptional activation of endothelial nitric oxide synthase. Nature Medicine. 8(5). 473–479. 454 indexed citations

14.

Hafezi‐Moghadam, Ali, Kennard L. Thomas, Alyson Prorock, Yuqing Huo, & Klaus Ley. (2001). L-Selectin Shedding Regulates Leukocyte Recruitment. The Journal of Experimental Medicine. 193(7). 863–872. 179 indexed citations

15.

Methia, Nassia, et al.. (2001). ApoE Deficiency Compromises the Blood Brain Barrier Especially After Injury. Molecular Medicine. 7(12). 810–815. 130 indexed citations

16.

Snowden, Lonnie R., Anne M. Libby, & Kennard L. Thomas. (1998). Health-care-related attitudes and utilization among African American women.. PubMed. 3(3-4). 301–14. 15 indexed citations

17.

Thomas, Kennard L., et al.. (1968). Zur Silicose-Entstehung. Die Naturwissenschaften. 55(1). 22–25. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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