Keiichi Ito

903 total citations · 1 hit paper
29 papers, 579 citations indexed

About

Keiichi Ito is a scholar working on Molecular Biology, Surgery and Hepatology. According to data from OpenAlex, Keiichi Ito has authored 29 papers receiving a total of 579 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Surgery and 9 papers in Hepatology. Recurrent topics in Keiichi Ito's work include Liver physiology and pathology (9 papers), Pancreatic function and diabetes (7 papers) and Pluripotent Stem Cells Research (4 papers). Keiichi Ito is often cited by papers focused on Liver physiology and pathology (9 papers), Pancreatic function and diabetes (7 papers) and Pluripotent Stem Cells Research (4 papers). Keiichi Ito collaborates with scholars based in Japan, United States and India. Keiichi Ito's co-authors include Robert G. Roeder, Hiromitsu Nakauchi, Ayaka Yanagida, Akihide Kamiya, Heankel Lyons, Benjamin R. Sabari, Hiromi Chikada, Alan Gerber, Ayumi Umino and Ken Okada and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Keiichi Ito

28 papers receiving 576 citations

Hit Papers

Functional partitioning of transcriptional regulators by ... 2023 2026 2024 2025 2023 50 100 150

Peers

Keiichi Ito
Ho‐June Lee South Korea
Shi-Rong Cai United States
Nicole Prior Germany
Raymond J. Peroutka United States
Ho‐June Lee South Korea
Keiichi Ito
Citations per year, relative to Keiichi Ito Keiichi Ito (= 1×) peers Ho‐June Lee

Countries citing papers authored by Keiichi Ito

Since Specialization
Citations

This map shows the geographic impact of Keiichi Ito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keiichi Ito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keiichi Ito more than expected).

Fields of papers citing papers by Keiichi Ito

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keiichi Ito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keiichi Ito. The network helps show where Keiichi Ito may publish in the future.

Co-authorship network of co-authors of Keiichi Ito

This figure shows the co-authorship network connecting the top 25 collaborators of Keiichi Ito. A scholar is included among the top collaborators of Keiichi Ito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keiichi Ito. Keiichi Ito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tajima, Yoko, Keiichi Ito, Wei Wang, et al.. (2025). A humanized NOVA1 splicing factor alters mouse vocal communications. Nature Communications. 16(1). 1542–1542. 3 indexed citations
2.
Nakadai, Tomoyoshi, Miho Shimada, Keiichi Ito, et al.. (2023). Two target gene activation pathways for orphan ERR nuclear receptors. Cell Research. 33(2). 165–183. 14 indexed citations
3.
Lyons, Heankel, et al.. (2023). Functional partitioning of transcriptional regulators by patterned charge blocks. Cell. 186(2). 327–345.e28. 160 indexed citations breakdown →
4.
Tajima, Yoko, Keiichi Ito, Yuan Yuan, et al.. (2023). NOVA1 acts on Impact to regulate hypothalamic function and translation in inhibitory neurons. Cell Reports. 42(2). 112050–112050. 8 indexed citations
5.
Lee, Yu-Ling, Keiichi Ito, Wen‐Chieh Pi, et al.. (2021). Mediator subunit MED1 is required for E2A-PBX1–mediated oncogenic transcription and leukemic cell growth. Proceedings of the National Academy of Sciences. 118(6). 20 indexed citations
6.
Ito, Keiichi, Marc Schneeberger, Alan Gerber, et al.. (2021). Critical roles of transcriptional coactivator MED1 in the formation and function of mouse adipose tissues. Genes & Development. 35(9-10). 729–748. 9 indexed citations
7.
Jishage, Miki, Keiichi Ito, Chi-Shuen Chu, et al.. (2020). Transcriptional down-regulation of metabolic genes by Gdown1 ablation induces quiescent cell re-entry into the cell cycle. Genes & Development. 34(11-12). 767–784. 4 indexed citations
8.
Gerber, Alan, et al.. (2020). Gene-Specific Control of tRNA Expression by RNA Polymerase II. Molecular Cell. 78(4). 765–778.e7. 45 indexed citations
9.
Obata, Yuuki, Yasushi Hara, Isamu Shiina, et al.. (2019). N822K- or V560G-mutated KIT activation preferentially occurs in lipid rafts of the Golgi apparatus in leukemia cells. Cell Communication and Signaling. 17(1). 114–114. 7 indexed citations
10.
Tajima, Yoko, Keiichi Ito, Ayumi Umino, et al.. (2017). Continuous cell supply from Krt7-expressing hematopoietic stem cells during native hematopoiesis revealed by targeted in vivo gene transfer method. Scientific Reports. 7(1). 40684–40684. 12 indexed citations
11.
Masaki, Hideki, Megumi Kato‐Itoh, Yusuke Takahashi, et al.. (2016). Inhibition of Apoptosis Overcomes Stage-Related Compatibility Barriers to Chimera Formation in Mouse Embryos. Cell stem cell. 19(5). 587–592. 77 indexed citations
12.
Kamiya, Akihide, Keiichi Ito, Ayaka Yanagida, et al.. (2015). MEK-ERK Activity Regulates the Proliferative Activity of Fetal Hepatoblasts Through Accumulation of p16/19 cdkn2a. Stem Cells and Development. 24(21). 2525–2535. 7 indexed citations
13.
Yanagida, Ayaka, Hiromi Chikada, Keiichi Ito, et al.. (2015). Liver maturation deficiency in p57−/− mice occurs in a hepatocytic p57Kip2 expression-independent manner. Developmental Biology. 407(2). 331–343. 1 indexed citations
14.
Chikada, Hiromi, Keiichi Ito, Ayaka Yanagida, Hiromitsu Nakauchi, & Akihide Kamiya. (2015). The basic helix-loop-helix transcription factor, Mist1, induces maturation of mouse fetal hepatoblasts. Scientific Reports. 5(1). 14989–14989. 13 indexed citations
15.
Ito, Keiichi, Jun Aida, Noriko Cable, et al.. (2015). International Comparative Research of Oral Health Inequality between Japan and England.. International Journal of Epidemiology. 44(suppl_1). i200–i201. 1 indexed citations
16.
Ito, Keiichi, Satoshi Yamazaki, Ryō Yamamoto, et al.. (2014). Gene Targeting Study Reveals Unexpected Expression of Brain-expressed X-linked 2 in Endocrine and Tissue Stem/Progenitor Cells in Mice. Journal of Biological Chemistry. 289(43). 29892–29911. 19 indexed citations
17.
Yanagida, Ayaka, Keiichi Ito, Hiromi Chikada, Hiromitsu Nakauchi, & Akihide Kamiya. (2013). An In Vitro Expansion System for Generation of Human iPS Cell-Derived Hepatic Progenitor-Like Cells Exhibiting a Bipotent Differentiation Potential. PLoS ONE. 8(7). e67541–e67541. 59 indexed citations
18.
Okada, Ken, Akihide Kamiya, Keiichi Ito, et al.. (2011). Prospective Isolation and Characterization of Bipotent Progenitor Cells in Early Mouse Liver Development. Stem Cells and Development. 21(7). 1124–1133. 27 indexed citations
19.
Ito, Keiichi, Katsuya Shiraki, Hiroshi Okano, et al.. (2004). Terminal ileal lipoma. Gastrointestinal Endoscopy. 60(2). 260–261.
20.
Ito, Keiichi, et al.. (1995). Analysis of agriculture-forestry production and settlement promotion policies in rural areas (I) On the possibility of transfer into rural areas.. Journal of the Japanese Forest Society. 77(5). 421–428. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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