Kay Klausing

713 citations
7 papers · 460 indexed · h-index 7
Topics
Peroxisome Proliferator-Activated Receptors (3 papers)Adipokines, Inflammation, and Metabolic Diseases (2 papers)Retinoids in leukemia and cellular processes (2 papers)
Partner nations
United StatesGermany

In The Last Decade

Kay Klausing

7 papers receiving 443 citations

Peers

Kay Klausing
Comparison fields: 5 of 60
  • Molecular Biology 360
  • Endocrinology, Diabetes and Metabolism 98
  • Physiology 98
  • Genetics 95
  • Surgery 61
Replace Anne C. Loweth with:
Anne C. Loweth United Kingdom
Sharon F. Clark Australia
András Divald United States
Marina Casimir Switzerland
Ranjan Mukherjee United States
Jackie Bodnar United States
Carina Hallberg Sweden
Françoise Chevy France
M.M. McGrane United States
Rogayah Carroll Canada
Kay Klausing relative to Anne C. Loweth United Kingdom Anne C. Loweth's profile →
Citations per field
00.5×
Anne C. Loweth · 1×
Citations per year

Countries citing papers authored by Kay Klausing

Since Specialization
Citations

This map shows the geographic impact of Kay Klausing's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kay Klausing with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kay Klausing more than expected).

Fields of papers citing papers by Kay Klausing

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kay Klausing. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kay Klausing. The network helps show where Kay Klausing may publish in the future.

Co-authorship network of co-authors of Kay Klausing

This figure shows the co-authorship network connecting the top 25 collaborators of Kay Klausing. A scholar is included among the top collaborators of Kay Klausing based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kay Klausing. Kay Klausing is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
#WorkIndexed citations
1 93
2 7
3 58
4 67
5 116
6 108
7 11

About Kay Klausing

Kay Klausing is a scholar working on Endocrinology, Diabetes and Metabolism, Dermatology and Genetics, having authored 7 papers that have together received 460 indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (3 papers), Adipokines, Inflammation, and Metabolic Diseases (2 papers) and Retinoids in leukemia and cellular processes (2 papers). The work is most often cited by research in Biochemistry (37 citations), Biochemistry (43 citations) and Endocrinology, Diabetes and Metabolism (98 citations). Kay Klausing has collaborated with scholars based in United States and Germany. Frequent co-authors include Rosemary M. Cesario, James Bilakovics, Mark D. Leibowitz, Claudia Stauber, Katsuyuki Yaginuma, Lars E. Theill, Pamela L. Mellon, Michael Karin, Dale E. Mais and James R. Paterniti. Their work appears in journals such as Journal of Biological Chemistry, Genes & Development and Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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