Katja Nieweg

651 total citations
13 papers, 506 citations indexed

About

Katja Nieweg is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Katja Nieweg has authored 13 papers receiving a total of 506 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Cellular and Molecular Neuroscience and 4 papers in Physiology. Recurrent topics in Katja Nieweg's work include Alzheimer's disease research and treatments (4 papers), Neuroscience and Neural Engineering (4 papers) and Pluripotent Stem Cells Research (3 papers). Katja Nieweg is often cited by papers focused on Alzheimer's disease research and treatments (4 papers), Neuroscience and Neural Engineering (4 papers) and Pluripotent Stem Cells Research (3 papers). Katja Nieweg collaborates with scholars based in Germany, France and Netherlands. Katja Nieweg's co-authors include Frank W. Pfrieger, Hubert Schaller, Kurt Gottmann, Andreas Müller‐Schiffmann, Carsten Korth, Kerstin Lenk, Christoph Redies, Kay Jüngling, Isabelle Buard and Robert Eckenstaler and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Brain.

In The Last Decade

Katja Nieweg

13 papers receiving 501 citations

Peers

Countries citing papers authored by Katja Nieweg

Since Specialization

Citations

This map shows the geographic impact of Katja Nieweg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katja Nieweg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katja Nieweg more than expected).

Fields of papers citing papers by Katja Nieweg

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katja Nieweg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katja Nieweg. The network helps show where Katja Nieweg may publish in the future.

Co-authorship network of co-authors of Katja Nieweg

This figure shows the co-authorship network connecting the top 25 collaborators of Katja Nieweg. A scholar is included among the top collaborators of Katja Nieweg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katja Nieweg. Katja Nieweg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

#	Work	Indexed citations
1	Pathogenic Aβ production by heterozygous PSEN1 mutations is intrinsic to the mutant protein and not mediated by conformational hindrance of wild-type PSEN1 2023 ·Journal of Biological Chemistry ·(unknown), Isabella Ogorek, (unknown), Javier López-Rı́os, (unknown), (unknown), Katja Nieweg, Anton Roebroek, Claus U. Pietrzik, Dirk Beher, Sascha Weggen	2
2	Contribution of astrocytes to metabolic dysfunction in the Alzheimer’s disease brain 2019 ·Biological Chemistry ·(unknown), (unknown), Katja Nieweg	36
3	Astrocyte lineage cells are essential for functional neuronal differentiation and synapse maturation in human iPSC‐derived neural networks 2019 ·Glia ·(unknown), (unknown), (unknown), Kerstin Lenk, Kurt Gottmann, Katja Nieweg	31
4	Episomal plasmid-based generation of an iPSC line from a 79-year-old individual carrying the APOE4/4 genotype: i11001 2016 ·Stem Cell Research ·(unknown), Bárbara Fritz, Katja Nieweg	3
5	Episomal plasmid-based generation of an iPSC line from an 83-year-old individual carrying the APOE4/4 genotype: i10984 2016 ·Stem Cell Research ·(unknown), Bárbara Fritz, Katja Nieweg	3
6	Alzheimer's disease-related amyloid-β induces synaptotoxicity in human iPS cell-derived neurons 2015 ·Cell Death and Disease ·Katja Nieweg, (unknown), (unknown), (unknown), Kurt Gottmann	79
7	mESC-Based in vitro Differentiation Models to Study Vascular Response and Functionality Following Genotoxic Insults 2014 ·Toxicological Sciences ·(unknown), Katja Nieweg, (unknown), Matthias U. Kassack, Kurt Gottmann, Gerhard Fritz	9
8	Stably BDNF-GFP expressing embryonic stem cells exhibit a BDNF release-dependent enhancement of neuronal differentiation 2013 ·Journal of Cell Science ·Julia Leschik, Robert Eckenstaler, Katja Nieweg, (unknown), Tanja Brigadski, Kurt Gottmann, Volkmar Leßmann, Beat Lutz	18
9	Asymmetric N-Cadherin Expression Results in Synapse Dysfunction, Synapse Elimination, and Axon Retraction in Cultured Mouse Neurons 2013 ·PLoS ONE ·(unknown), (unknown), (unknown), Katja Nieweg, Kay Jüngling, Christoph Redies, Kurt Gottmann	27
10	Embryonic stem cells stably expressing BDNF–GFP exhibit a BDNF-release-dependent enhancement of neuronal differentiation 2013 ·Development ·Julia Leschik, Robert Eckenstaler, Katja Nieweg, (unknown), Tanja Brigadski, Kurt Gottmann, Volkmar Leßmann, Beat Lutz	3
11	C-terminal fragment of N-cadherin accelerates synapse destabilization by amyloid-β 2012 ·Brain ·(unknown), Katja Nieweg, (unknown), (unknown), Andreas Müller‐Schiffmann, Carsten Korth, Kurt Gottmann	40
12	Marked differences in cholesterol synthesis between neurons and glial cells from postnatal rats 2009 ·Journal of Neurochemistry ·Katja Nieweg, Hubert Schaller, Frank W. Pfrieger	223
13	Glia‐induced neuronal differentiation by transcriptional regulation 2007 ·Glia ·Christian Göritz, (unknown), Luc‐Henri Tessier, Katja Nieweg, Christoph Moehle, Isabelle Buard, Jean‐Luc Dupont, Leon J. Schurgers, Gerd Schmitz, Frank W. Pfrieger	32

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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