Katie A. Matatall
- Ophthalmology top 0.5%
- Ocular Oncology and Treatments 4
- Hematology top 2%
- Hematopoietic Stem Cell Transplantation 8
- Acute Myeloid Leukemia Research 7
- Immunology top 5%
- Immune cells in cancer 2
- Immune Cell Function and Interaction 2
- Oncology top 5%
- Cytokine Signaling Pathways and Interactions 2
- Genetics top 5%
- Myeloproliferative Neoplasms: Diagnosis and Treatment 2
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- Epigenetics and DNA Methylation 5
- Co-authors
- Michael D. OnkenJ. William HarbourA. BowcockLori A. WorleyElisha RobersonCynthia HelmsShenghui DuanLi Cao
- Cited by
- OphthalmologyHematologyImmunology
- Partner nations
- United StatesPoland
In The Last Decade
Katie A. Matatall
16 papers receiving 2.2k citations
Hit Papers
Peers
Comparison fields: 5 of 78
- Ophthalmology 847
- Hematology 468
- Immunology 693
- Oncology 672
- Genetics 189
Countries citing papers authored by Katie A. Matatall
This map shows the geographic impact of Katie A. Matatall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katie A. Matatall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katie A. Matatall more than expected).
Fields of papers citing papers by Katie A. Matatall
This network shows the impact of papers produced by Katie A. Matatall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katie A. Matatall. The network helps show where Katie A. Matatall may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Katie A. Matatall, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Chronic infection drives Dnmt3a-loss-of-function clonal hematopoiesis via IFNγ signalingbreakdown → | 2021 | 211 |
| 2 | 2020 | 6 | |
| 3 | 2020 | 36 | |
| 4 | 2019 | 2 | |
| 5 | 2017 | 21 | |
| 6 | 2016 | 194 | |
| 7 | 2016 | 47 | |
| 8 | 2015 | 26 | |
| 9 | 2015 | 7 | |
| 10 | 2014 | 39 | |
| 11 | 2014 | 96 | |
| 12 | 2013 | 112 | |
| 13 | 2011 | 212 | |
| 14 | 2010 | 37 | |
| 15 | Frequent Mutation of BAP1 in Metastasizing Uveal Melanomasbreakdown → | 2010 | 1024 |
| 16 | 2007 | 116 |
About Katie A. Matatall
Katie A. Matatall is a scholar working on Hematology, Ophthalmology and Immunology, having authored 16 papers that have together received 2.2k indexed citations. Recurring topics across this work include Hematopoietic Stem Cell Transplantation (8 papers), Acute Myeloid Leukemia Research (7 papers), Epigenetics and DNA Methylation (5 papers), Ocular Oncology and Treatments (4 papers), Immune cells in cancer (2 papers), Immune Cell Function and Interaction (2 papers), Cytokine Signaling Pathways and Interactions (2 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (2 papers). The work is most often cited by research in Ophthalmology (847 citations), Hematology (468 citations) and Immunology (693 citations). Katie A. Matatall has collaborated with scholars based in United States and Poland. Frequent co-authors include Michael D. Onken, J. William Harbour, A. Bowcock, Lori A. Worley, Elisha Roberson, Cynthia Helms, Shenghui Duan, Li Cao, Katherine Y. King and Marek Kimmel. Their work appears in journals such as Science, Blood and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.