Haiyan S. Li

5.6k total citations · 1 hit paper
43 papers, 2.8k citations indexed

About

Haiyan S. Li is a scholar working on Immunology, Oncology and Cancer Research. According to data from OpenAlex, Haiyan S. Li has authored 43 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Immunology, 20 papers in Oncology and 7 papers in Cancer Research. Recurrent topics in Haiyan S. Li's work include Immune Cell Function and Interaction (18 papers), T-cell and B-cell Immunology (15 papers) and Immunotherapy and Immune Responses (14 papers). Haiyan S. Li is often cited by papers focused on Immune Cell Function and Interaction (18 papers), T-cell and B-cell Immunology (15 papers) and Immunotherapy and Immune Responses (14 papers). Haiyan S. Li collaborates with scholars based in United States, Canada and China. Haiyan S. Li's co-authors include Stephanie S. Watowich, Huiyuan Zhang, George Carayanniotis, Hoainam Nguyen-Jackson, Athanasia D. Panopoulos, Panayotis Verginis, Shao‐Cong Sun, Cliff Y. Yang, Ananda W. Goldrath and Hongbo Hu and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Haiyan S. Li

42 papers receiving 2.7k citations

Hit Papers

STAT3 signaling in immunity 2016 2026 2019 2022 2016 100 200 300 400 500

Peers

Haiyan S. Li
Cong‐Qiu Chu United States
Gerald M. Feldman United States
Ming-Hong Xie United States
Hu Zeng United States
Alena Donda Switzerland
Shuhua Han United States
Cong‐Qiu Chu United States
Haiyan S. Li
Citations per year, relative to Haiyan S. Li Haiyan S. Li (= 1×) peers Cong‐Qiu Chu

Countries citing papers authored by Haiyan S. Li

Since Specialization
Citations

This map shows the geographic impact of Haiyan S. Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Haiyan S. Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Haiyan S. Li more than expected).

Fields of papers citing papers by Haiyan S. Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Haiyan S. Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Haiyan S. Li. The network helps show where Haiyan S. Li may publish in the future.

Co-authorship network of co-authors of Haiyan S. Li

This figure shows the co-authorship network connecting the top 25 collaborators of Haiyan S. Li. A scholar is included among the top collaborators of Haiyan S. Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Haiyan S. Li. Haiyan S. Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Marié, Isabelle, Doua F. Azzouz, Ze Chen, et al.. (2021). Tonic interferon restricts pathogenic IL-17-driven inflammatory disease via balancing the microbiome. eLife. 10. 22 indexed citations
2.
Wang, Junmei, Haiyan S. Li, Jungsun Park, et al.. (2020). Histone Deacetylase Inhibitors and IL21 Cooperate to Reprogram Human Effector CD8+ T Cells to Memory T Cells. Cancer Immunology Research. 8(6). 794–805. 17 indexed citations
3.
Zhou, Yifan, Haiyan S. Li, Bhakti Patel, et al.. (2020). STAT3 Inhibits CD103+ cDC1 Vaccine Efficacy in Murine Breast Cancer. Cancers. 12(1). 128–128. 16 indexed citations
4.
Zhou, Yifan, Oleksandr Kyrysyuk, Yusra B. Medik, et al.. (2020). Vaccine efficacy against primary and metastatic cancer with in vitro-generated CD103+ conventional dendritic cells. Journal for ImmunoTherapy of Cancer. 8(1). e000474–e000474. 84 indexed citations
5.
Zhang, Huiyuan, Haiyan S. Li, Yang Zhao, et al.. (2018). Genetic rescue of lineage-balanced blood cell production reveals a crucial role for STAT3 antiinflammatory activity in hematopoiesis. Proceedings of the National Academy of Sciences. 115(10). E2311–E2319. 13 indexed citations
6.
Zhou, Yifan, et al.. (2018). Molecular regulation of dendritic cell development and function in homeostasis, inflammation, and cancer. Molecular Immunology. 110. 24–39. 46 indexed citations
7.
Xiao, Yichuan, Qiang Zou, Xiaoping Xie, et al.. (2017). The kinase TBK1 functions in dendritic cells to regulate T cell homeostasis, autoimmunity, and antitumor immunity. The Journal of Experimental Medicine. 214(5). 1493–1507. 65 indexed citations
8.
Landers, Cameron, et al.. (2017). MicroRNA-22 controls interferon alpha production and erythroid maturation in response to infectious stress in mice. Experimental Hematology. 56. 7–15. 18 indexed citations
9.
Zhang, Huiyuan, et al.. (2016). STAT3 signaling in immunity. Cytokine & Growth Factor Reviews. 31. 1–15. 539 indexed citations breakdown →
10.
Zou, Qiang, Jin Jin, Hongbo Hu, et al.. (2014). USP15 stabilizes MDM2 to mediate cancer-cell survival and inhibit antitumor T cell responses. Nature Immunology. 15(6). 562–570. 198 indexed citations
11.
Jin, Jin, Hongbo Hu, Haiyan S. Li, et al.. (2014). Noncanonical NF-κB Pathway Controls the Production of Type I Interferons in Antiviral Innate Immunity. Immunity. 40(3). 342–354. 113 indexed citations
12.
Zhang, Huiyuan, Hongbo Hu, Nathaniel Greeley, et al.. (2014). STAT3 restrains RANK- and TLR4-mediated signalling by suppressing expression of the E2 ubiquitin-conjugating enzyme Ubc13. Nature Communications. 5(1). 5798–5798. 57 indexed citations
13.
Nurieva, Roza, Andrew Podd, Yuhong Chen, et al.. (2012). STAT5 Protein Negatively Regulates T Follicular Helper (Tfh) Cell Generation and Function. Journal of Biological Chemistry. 287(14). 11234–11239. 187 indexed citations
14.
Yang, Cliff Y., J. Adam Best, Jamie Knell, et al.. (2011). The transcriptional regulators Id2 and Id3 control the formation of distinct memory CD8+ T cell subsets. Nature Immunology. 12(12). 1221–1229. 303 indexed citations
15.
Li, Haiyan S., Davinna L. Ligons, Noel R. Rose, & Mehmet Güler. (2008). Genetic Differences in Bone Marrow-Derived Lymphoid Lineages Control Susceptibility to Experimental Autoimmune Myocarditis. The Journal of Immunology. 180(11). 7480–7484. 7 indexed citations
16.
Ligons, Davinna L., Mehmet Güler, Haiyan S. Li, & Noel R. Rose. (2008). A locus on chromosome 1 promotes susceptibility of experimental autoimmune myocarditis and lymphocyte cell death. Clinical Immunology. 130(1). 74–82. 9 indexed citations
17.
Jiang, Hong, et al.. (2007). Variable influences of iodine on the T‐cell recognition of a single thyroglobulin epitope. Immunology. 121(3). 370–376. 10 indexed citations
18.
Li, Haiyan S., Hong Jiang, & George Carayanniotis. (2007). Modifying effects of iodine on the immunogenicity of thyroglobulin peptides. Journal of Autoimmunity. 28(4). 171–176. 27 indexed citations
19.
Li, Haiyan S. & George Carayanniotis. (2006). Iodination of Tyrosyls in Thyroglobulin Generates Neoantigenic Determinants That Cause Thyroiditis. The Journal of Immunology. 176(7). 4479–4483. 22 indexed citations
20.
Li, Haiyan S. & George Carayanniotis. (2005). Detection of thyroglobulin mRNA as truncated isoform(s) in mouse thymus. Immunology. 115(1). 85–89. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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