Katharine E. Black

1.8k total citations
22 papers, 744 citations indexed

About

Katharine E. Black is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Cell Biology. According to data from OpenAlex, Katharine E. Black has authored 22 papers receiving a total of 744 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Pulmonary and Respiratory Medicine, 8 papers in Molecular Biology and 6 papers in Cell Biology. Recurrent topics in Katharine E. Black's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (8 papers), Fibroblast Growth Factor Research (3 papers) and Proteoglycans and glycosaminoglycans research (3 papers). Katharine E. Black is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (8 papers), Fibroblast Growth Factor Research (3 papers) and Proteoglycans and glycosaminoglycans research (3 papers). Katharine E. Black collaborates with scholars based in United States, Germany and United Kingdom. Katharine E. Black's co-authors include Lindsey R. Baden, Samuel L. Collins, Yee Chan‐Li, Maureen R. Horton, Clemens K. Probst, Andrew M. Tager, Jonathan D. Powell, Lida P. Hariri, Barry S. Shea and Kara Scheibner and has published in prestigious journals such as New England Journal of Medicine, Journal of Clinical Investigation and The FASEB Journal.

In The Last Decade

Katharine E. Black

21 papers receiving 737 citations

Peers

Katharine E. Black
Comparison fields: 5 of 85
  • Molecular Biology 281
  • Pulmonary and Respiratory Medicine 252
  • Cell Biology 162
  • Epidemiology 105
  • Immunology 101
Patricia Brown‐Augsburger United States
Takahiro Iino Japan
Chung-Ping Liao United States
Miroslava Krzyžanková Czechia
Samuel L. Collins United States
Chirosree Bandyopadhyay United States
Debby Damm United States
Lu Cui China
Chuanyu Wei China
Mahalakshmi Ramadass United States
Patricia Brown‐Augsburger United States View profile →
Citations per field, relative to Katharine E. Black
Katharine E. Black · 1×
Citations per year, relative to Katharine E. Black
Katharine E. Black · 1×

Countries citing papers authored by Katharine E. Black

Since Specialization
Citations

This map shows the geographic impact of Katharine E. Black's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katharine E. Black with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katharine E. Black more than expected).

Fields of papers citing papers by Katharine E. Black

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katharine E. Black. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katharine E. Black. The network helps show where Katharine E. Black may publish in the future.

Co-authorship network of co-authors of Katharine E. Black

This figure shows the co-authorship network connecting the top 25 collaborators of Katharine E. Black. A scholar is included among the top collaborators of Katharine E. Black based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katharine E. Black. Katharine E. Black is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Title Journal Authors Indexed citations
1 Sanglifehrin A mitigates multiorgan fibrosis by targeting the collagen chaperone cyclophilin B JCI Insight Hope A. Flaxman, Hongwei Han et al. 4
2 Human Airway Basal Cells Undergo Reversible Squamous Differentiation and Reshape Innate Immunity American Journal of Respiratory Cell and Molecular Biology Yihan Zhang, Katharine E. Black et al. 10
3 Integrated Immunopeptidomic and Proteomic Analysis of COVID-19 lung biopsies Frontiers in Immunology Shanye Yin, Susan Klaeger et al. 4
4 Screening for Inhibitors of YAP Nuclear Localization Identifies Aurora Kinase A as a Modulator of Lung Fibrosis American Journal of Respiratory Cell and Molecular Biology Daniela M. Santos, Lorena Pantano et al. 9
5 Macrophage-derived netrin-1 drives adrenergic nerve–associated lung fibrosis Journal of Clinical Investigation Ruijuan Gao, Xueyan Peng et al. 43
6 Veno-Venous Extracorporeal Membrane Oxygenation for Myositis-Associated Rapidly Progressive Interstitial Lung Disease CHEST Journal Jonah Rubin, Katharine E. Black et al. 8
7 Pretreatment of aged mice with retinoic acid supports alveolar regeneration via upregulation of reciprocal PDGFA signalling Thorax Jason J. Gokey, John Snowball et al. 24
8 Screening for YAP Inhibitors Identifies Statins as Modulators of Fibrosis American Journal of Respiratory Cell and Molecular Biology Daniela M. Santos, Lorena Pantano et al. 47
9 Loss of Endothelial S1PR1 Exacerbates Bleomycin-Induced Pulmonary Fibrosis Through Intra-Alveolar Coagulation and Immune Cell Infiltration Rachel S. Knipe, E. Abe et al. 1
10 MEG3 is increased in idiopathic pulmonary fibrosis and regulates epithelial cell differentiation JCI Insight Jason J. Gokey, John Snowball et al. 50
11 The Rho Kinase Isoforms ROCK1 and ROCK2 Each Contribute to the Development of Experimental Pulmonary Fibrosis American Journal of Respiratory Cell and Molecular Biology Rachel S. Knipe, Clemens K. Probst et al. 88
12 Uncoupling of the profibrotic and hemostatic effects of thrombin in lung fibrosis JCI Insight Barry S. Shea, Clemens K. Probst et al. 66
13 Autotaxin activity increases locally following lung injury, but is not required for pulmonary lysophosphatidic acid production or fibrosis The FASEB Journal Katharine E. Black, Evgeny Berdyshev et al. 35
14 An Autotaxin/Lysophosphatidic Acid/Interleukin‐6 Amplification Loop Drives Scleroderma Fibrosis Arthritis & Rheumatology Flavia V. Castelino, Gretchen Bain et al. 78
15 Lovastatin Inhibits Low Molecular Weight Hyaluronan Induced Chemokine Expression via LFA-1 and Decreases Bleomycin-Induced Pulmonary Fibrosis International Journal of Biomedical Science Mark J. Hamblin, Michael Eberlein et al. 8
16 Hyaluronan fragments induce IFNβ via a novel TLR4-TRIF-TBK1-IRF3-dependent pathway Journal of Inflammation Katharine E. Black, Samuel L. Collins et al. 49
17 Hyaluronan Fragments Promote Inflammation by Down-Regulating the Anti-inflammatory A2a Receptor American Journal of Respiratory Cell and Molecular Biology Samuel L. Collins, Katharine E. Black et al. 49
18 Anti-oxidant inhibition of hyaluronan fragment-induced inflammatory gene expression Journal of Inflammation Michael Eberlein, Kara Scheibner et al. 44
19 The Adenosine A2a Receptor Inhibits Matrix-Induced Inflammation in a Novel Fashion American Journal of Respiratory Cell and Molecular Biology Kara Scheibner, Samuel L. Collins et al. 53
20 Fungal Infections of the CNS CNS Drugs Katharine E. Black, Lindsey R. Baden 66

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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