K. HAMANN

632 total citations
12 papers, 480 citations indexed

About

K. HAMANN is a scholar working on Immunology and Allergy, Immunology and Rheumatology. According to data from OpenAlex, K. HAMANN has authored 12 papers receiving a total of 480 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Immunology and Allergy, 9 papers in Immunology and 4 papers in Rheumatology. Recurrent topics in K. HAMANN's work include Mast cells and histamine (9 papers), Food Allergy and Anaphylaxis Research (5 papers) and Cell Adhesion Molecules Research (4 papers). K. HAMANN is often cited by papers focused on Mast cells and histamine (9 papers), Food Allergy and Anaphylaxis Research (5 papers) and Cell Adhesion Molecules Research (4 papers). K. HAMANN collaborates with scholars based in Germany. K. HAMANN's co-authors include Beate M. Czarnetzki, N. Haas, P. Welker, G. Kolde, Thomas A. Luger, Dirk Schadendorf, Undine Lippert, A. Möller, T. Rosenbach and Jürgen Grabbe and has published in prestigious journals such as The Journal of Immunology, The Journal of Pathology and British Journal of Dermatology.

In The Last Decade

K. HAMANN

12 papers receiving 463 citations

Peers

K. HAMANN
Anne Marie Irani United States
Tekli Semere United States
Jennifer N. Lilla United States
Ashley M. Mahler United States
Hyun Je Kim South Korea
Naoya Kameyama United States
Anne Marie Irani United States
K. HAMANN
Citations per year, relative to K. HAMANN K. HAMANN (= 1×) peers Anne Marie Irani

Countries citing papers authored by K. HAMANN

Since Specialization
Citations

This map shows the geographic impact of K. HAMANN's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. HAMANN with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. HAMANN more than expected).

Fields of papers citing papers by K. HAMANN

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K. HAMANN. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. HAMANN. The network helps show where K. HAMANN may publish in the future.

Co-authorship network of co-authors of K. HAMANN

This figure shows the co-authorship network connecting the top 25 collaborators of K. HAMANN. A scholar is included among the top collaborators of K. HAMANN based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K. HAMANN. K. HAMANN is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Dippel, Edgar, N. Haas, Jürgen Grabbe, et al.. (2006). Expression of the c-kit receptor in hypomelanosis: a comparative study between piebaldism, naevus depigmentosus and vitiligo. British Journal of Dermatology. 132(2). 182–189. 15 indexed citations
2.
Haas, N., et al.. (2006). β2-integrins in different forms of urticaria. British Journal of Dermatology. 133(1). 48–53. 4 indexed citations
3.
Haas, N., K. HAMANN, Jürgen Grabbe, et al.. (1997). Demonstration of the high‐affinity IgE receptor (FcɛRI) on Langerhans' cells of diseased nasal mucosa. Allergy. 52(4). 436–439. 10 indexed citations
4.
HAMANN, K., N. Haas, Jürgen Grabbe, P. Welker, & Beate M. Czarnetzki. (1995). Two novel mast cell phenotypic markers, monoclonal antibodies Ki-MC1 and Ki-M1P, identify distinct mast cell subtypes. British Journal of Dermatology. 133(4). 547–552. 14 indexed citations
5.
HAMANN, K., et al.. (1995). Expression of stem cell factor in cutaneous mastocytosis. British Journal of Dermatology. 133(2). 203–208. 47 indexed citations
6.
Haas, Norbert, et al.. (1995). Phenotypic characterization of skin lesions in urticaria pigmentosa and mastocytomas. Archives of Dermatological Research. 287(3-4). 242–248. 12 indexed citations
7.
HAMANN, K., Jürgen Grabbe, P. Welker, et al.. (1994). Phenotypic evaluation of cultured human mast and basophilic cells and of normal human skin mast cells. Archives of Dermatological Research. 286(7). 380–385. 45 indexed citations
8.
Möller, A., Undine Lippert, G. Kolde, et al.. (1993). Human mast cells produce IL-8.. The Journal of Immunology. 151(6). 3261–3266. 242 indexed citations
9.
Grabbe, Jürgen, N. Haas, K. HAMANN, et al.. (1993). Demonstration of the high-affinity IgE receptor on human Langerhans cells in normal and diseased skin. British Journal of Dermatology. 129(2). 120–123. 35 indexed citations
10.
Goerdt, Sergij, Gerhard Kolde, Gisela Bonsmann, et al.. (1993). Immunohistochemical comparison of cutaneous histiocytoses and related skin disorders: Diagnostic and histogenetic relevance of MS‐1 high molecular weight protein expression. The Journal of Pathology. 170(4). 421–427. 31 indexed citations
11.
Haas, N., K. HAMANN, Jürgen Grabbe, & Beate M. Czarnetzki. (1993). Demonstration of the high‐affinity IgE receptor (FcɛRI) on Langerhans cells of oral mucosa. Experimental Dermatology. 2(4). 157–160. 3 indexed citations
12.
Haas, N., et al.. (1992). Expression of the high affinity IgE-receptor on human Langerhans' cells. Elucidating the role of epidermal IgE in atopic eczema.. Acta Dermato Venereologica. 72(4). 271–272. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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