Tekli Semere

893 total citations
8 papers, 734 citations indexed

About

Tekli Semere is a scholar working on Immunology, Rheumatology and Immunology and Allergy. According to data from OpenAlex, Tekli Semere has authored 8 papers receiving a total of 734 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 4 papers in Rheumatology and 3 papers in Immunology and Allergy. Recurrent topics in Tekli Semere's work include Mast cells and histamine (8 papers), Cell Adhesion Molecules Research (3 papers) and Eosinophilic Disorders and Syndromes (2 papers). Tekli Semere is often cited by papers focused on Mast cells and histamine (8 papers), Cell Adhesion Molecules Research (3 papers) and Eosinophilic Disorders and Syndromes (2 papers). Tekli Semere collaborates with scholars based in United States, Japan and Israel. Tekli Semere's co-authors include Dean D. Metcalfe, Arnold S. Kirshenbaum, Linda M. Scott, Alexandra S. Worobec, Julie P. Goff, Barbara A. Foster, Hiroshi Nagata, Cem Akin, A S Worobec and D D Metcalfe and has published in prestigious journals such as Blood, Cancer and Biochemical Journal.

In The Last Decade

Tekli Semere

8 papers receiving 723 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tekli Semere United States	8	653	242	183	182	152	8	734
Anne Marie Irani United States	5	632 1.0×	144 0.6×	299 1.6×	248 1.4×	164 1.1×	9	723
H. Semper Austria	12	487 0.7×	229 0.9×	493 2.7×	326 1.8×	77 0.5×	17	917
K. HAMANN Germany	10	323 0.5×	95 0.4×	176 1.0×	130 0.7×	82 0.5×	12	480
Oliver Kilgus Austria	8	601 0.9×	94 0.4×	343 1.9×	263 1.4×	83 0.5×	12	872
Noriyasu Seki Japan	13	407 0.6×	113 0.5×	44 0.2×	81 0.4×	206 1.4×	26	693
Jonathan Keller United States	6	328 0.5×	28 0.1×	67 0.4×	89 0.5×	239 1.6×	8	608
F S Rosen United States	11	214 0.3×	46 0.2×	101 0.6×	27 0.1×	211 1.4×	16	559
Sayo Kataoka Japan	12	380 0.6×	148 0.6×	45 0.2×	56 0.3×	143 0.9×	21	577
Paola Pittoni Italy	12	571 0.9×	58 0.2×	33 0.2×	47 0.3×	125 0.8×	13	689

Countries citing papers authored by Tekli Semere

Since Specialization

Citations

This map shows the geographic impact of Tekli Semere's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tekli Semere with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tekli Semere more than expected).

Fields of papers citing papers by Tekli Semere

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tekli Semere. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tekli Semere. The network helps show where Tekli Semere may publish in the future.

Co-authorship network of co-authors of Tekli Semere

This figure shows the co-authorship network connecting the top 25 collaborators of Tekli Semere. A scholar is included among the top collaborators of Tekli Semere based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tekli Semere. Tekli Semere is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Akin, Cem, Arnold S. Kirshenbaum, Tekli Semere, et al.. (2000). Analysis of the surface expression of c-kit and occurrence of the c-kit Asp816Val activating mutation in T cells, B cells, and myelomonocytic cells in patients with mastocytosis. Experimental Hematology. 28(2). 140–147. 139 indexed citations

2.

Kirshenbaum, Arnold S., Julie P. Goff, Tekli Semere, et al.. (1999). Demonstration That Human Mast Cells Arise From a Progenitor Cell Population That Is CD34+, c-kit+, and Expresses Aminopeptidase N (CD13). Blood. 94(7). 2333–2342. 329 indexed citations

3.

Nagata, Hiroshi, A S Worobec, Tekli Semere, & D D Metcalfe. (1998). Elevated expression of the proto-oncogene c-kit in patients with mastocytosis. Leukemia. 12(2). 175–181. 29 indexed citations

4.

Worobec, Alexandra S., Tekli Semere, Hiroshi Nagata, & Dean D. Metcalfe. (1998). Clinical correlates of the presence of the asp816Val c-kit mutation in the peripheral blood mononuclear cells of patients with mastocytosis. Cancer. 83(10). 2120–2129. 113 indexed citations

5.

Kirshenbaum, Arnold S., A S Worobec, Todd A. Davis, et al.. (1998). Inhibition of human mast cell growth and differentiation by interferon gamma-1b.. PubMed. 26(3). 245–51. 37 indexed citations

6.

Nagata, Hiroshi, Tadashi Okada, Alexandra S. Worobec, Tekli Semere, & Dean D. Metcalfe. (1997). <i>c-ki</i><i>t</i> Mutation in a Population of Patients with Mastocytosis. International Archives of Allergy and Immunology. 113(1-3). 184–186. 61 indexed citations

7.

Mekori, Yoseph A., Chang Kyu Oh, Jarosław Dastych, et al.. (1997). Characterization of a mast cell line that lacks the extracellular domain of membrane c‐kit. Immunology. 90(4). 518–525. 16 indexed citations

8.

Oh, Chad K., et al.. (1997). Two different negative regulatory elements control the transcription of T-cell activation gene 3 in activated mast cells. Biochemical Journal. 323(2). 511–519. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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