Junjun Ni

2.7k total citations · 1 hit paper
104 papers, 2.0k citations indexed

About

Junjun Ni is a scholar working on Molecular Biology, Neurology and Physiology. According to data from OpenAlex, Junjun Ni has authored 104 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Molecular Biology, 30 papers in Neurology and 29 papers in Physiology. Recurrent topics in Junjun Ni's work include Neuroinflammation and Neurodegeneration Mechanisms (28 papers), Alzheimer's disease research and treatments (20 papers) and S100 Proteins and Annexins (8 papers). Junjun Ni is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (28 papers), Alzheimer's disease research and treatments (20 papers) and S100 Proteins and Annexins (8 papers). Junjun Ni collaborates with scholars based in China, Japan and United Kingdom. Junjun Ni's co-authors include Zhou Wu, Hong Qing, Hiroshi Nakanishi, Yicong Liu, Jie Meng, Hiroshi Nakanishi, Yoshinori Hayashi, Fei Lan, Jessica L. Teeling and Fumiko Takayama and has published in prestigious journals such as Journal of Neuroscience, ACS Nano and PLoS ONE.

In The Last Decade

Junjun Ni

99 papers receiving 2.0k citations

Hit Papers

Cathepsin B in programmed cell death machinery: mechanism... 2023 2026 2024 2025 2023 25 50 75 100

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Junjun Ni China 26 732 654 421 390 175 104 2.0k
Zhou Wu Japan 39 1.2k 1.7× 1.1k 1.7× 1.2k 2.7× 480 1.2× 275 1.6× 108 3.5k
Isabel Carreras United States 24 808 1.1× 678 1.0× 303 0.7× 111 0.3× 111 0.6× 43 1.9k
Kevin J. Washicosky United States 9 1.0k 1.4× 1.3k 2.1× 642 1.5× 121 0.3× 502 2.9× 11 2.5k
Joseane Morari Brazil 27 641 0.9× 1.2k 1.8× 314 0.7× 31 0.1× 117 0.7× 75 3.1k
Erica Costantini Italy 22 432 0.6× 375 0.6× 246 0.6× 51 0.1× 152 0.9× 62 1.4k
Masayasu Minami Japan 18 507 0.7× 244 0.4× 605 1.4× 107 0.3× 71 0.4× 66 2.2k
William A. Eimer United States 13 1.1k 1.5× 1.9k 3.0× 707 1.7× 63 0.2× 429 2.5× 19 2.9k
Małgorzata Żendzian‐Piotrowska Poland 27 1.0k 1.4× 889 1.4× 81 0.2× 92 0.2× 37 0.2× 119 2.2k
Hae Jeong Park South Korea 23 697 1.0× 162 0.2× 76 0.2× 63 0.2× 114 0.7× 94 1.7k
Boryana Stamova United States 37 2.4k 3.3× 616 0.9× 1.3k 3.1× 61 0.2× 321 1.8× 82 4.8k

Countries citing papers authored by Junjun Ni

Since Specialization
Citations

This map shows the geographic impact of Junjun Ni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junjun Ni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junjun Ni more than expected).

Fields of papers citing papers by Junjun Ni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Junjun Ni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junjun Ni. The network helps show where Junjun Ni may publish in the future.

Co-authorship network of co-authors of Junjun Ni

This figure shows the co-authorship network connecting the top 25 collaborators of Junjun Ni. A scholar is included among the top collaborators of Junjun Ni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Junjun Ni. Junjun Ni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ni, Junjun, et al.. (2025). Celastrol targets CKB-mediated futile creatine cycle in human brown adipocytes thermogenesis. Metabolism Open. 26. 100359–100359. 1 indexed citations
2.
Xie, Zhen, Qinghu Yang, Fei Lan, et al.. (2025). Cathepsin B deficiency disrupts cortical development via PEG3, leading to depression-like behavior. Communications Biology. 8(1). 1097–1097. 1 indexed citations
3.
Song, Da, et al.. (2024). The regulatory effect of the anterior cingulate cortex on helping behavior in juvenile social isolation model mice. Physiology & Behavior. 287. 114698–114698. 1 indexed citations
5.
Zhao, Dan, Yue Zhou, Wei Kong, et al.. (2024). Cathepsin B modulates microglial migration and phagocytosis of amyloid β in Alzheimer’s disease through PI3K-Akt signaling. Neuropsychopharmacology. 50(4). 640–650. 4 indexed citations
6.
Liu, Yanqing, Hongyan Wang, Li Chen, et al.. (2024). Cathepsin B Modulates Alzheimer's Disease Pathology Through SAPK/JNK Signals Following Administration of Porphyromonas gingivalis‐Derived Outer Membrane Vesicles. Journal Of Clinical Periodontology. 52(3). 434–456. 7 indexed citations
7.
Yang, Qinghu, Zhen Xie, Liang Yang, et al.. (2024). Extracellular Matrix Remodeling Alleviates Memory Deficits in Alzheimer's Disease by Enhancing the Astrocytic Autophagy‐Lysosome Pathway. Advanced Science. 11(31). e2400480–e2400480. 9 indexed citations
8.
Quan, Zhenzhen, et al.. (2024). Extracellular vesicle-packed microRNAs profiling in Alzheimer’s disease: The molecular intermediary between pathology and diagnosis. Ageing Research Reviews. 104. 102614–102614. 2 indexed citations
9.
Song, Da, Chunjian Wang, Yan Yan, et al.. (2023). Mediodorsal thalamus-projecting anterior cingulate cortex neurons modulate helping behavior in mice. Current Biology. 33(20). 4330–4342.e5. 9 indexed citations
10.
Xie, Zhen, Mengyuan Zhao, Wei Kong, et al.. (2023). Cathepsin B in programmed cell death machinery: mechanisms of execution and regulatory pathways. Cell Death and Disease. 14(4). 255–255. 103 indexed citations breakdown →
11.
Yan, Yan, Da Song, Chunjian Wang, et al.. (2023). ACx-projecting cholinergic neurons in the NB influence the BLA ensembles to modulate the discrimination of auditory fear memory. Translational Psychiatry. 13(1). 79–79. 3 indexed citations
12.
Ni, Junjun, et al.. (2023). The Regulatory Mechanism of Rab21 in Human Diseases. Molecular Neurobiology. 60(10). 5944–5953. 2 indexed citations
13.
Li, Danyang, Zhenzhen Quan, Junjun Ni, Hui Li, & Hong Qing. (2023). The many faces of the zinc finger protein 335 in brain development and immune system. Biomedicine & Pharmacotherapy. 165. 115257–115257. 7 indexed citations
14.
Ni, Junjun, et al.. (2022). Role of Vitronectin and Its Receptors in Neuronal Function and Neurodegenerative Diseases. International Journal of Molecular Sciences. 23(20). 12387–12387. 27 indexed citations
15.
Yan, Yan, Chunjian Wang, Weiqi Jin, et al.. (2022). Neuronal Circuits Associated with Fear Memory: Potential Therapeutic Targets for Posttraumatic Stress Disorder. The Neuroscientist. 29(3). 332–351. 2 indexed citations
16.
Xie, Zhen, Jie Meng, Zhou Wu, et al.. (2022). The Dual Nature of Microglia in Alzheimer’s Disease: A Microglia-Neuron Crosstalk Perspective. The Neuroscientist. 29(5). 616–638. 15 indexed citations
17.
Lin, Xiang, Liang Yang, Si Wu, et al.. (2021). WS6 Induces Adult Hippocampal Neurogenesis in Correlation to its Antidepressant Effect on the Alleviation of Depressive-like Behaviors of Rats. Neuroscience. 473. 119–129. 6 indexed citations
20.
Ni, Junjun, et al.. (2006). Chemical Constituents of Active Extracts from the Seeds of Nigella glandulifera Freyn. 亚洲传统医药. 1(2). 69–72. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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