William A. Eimer

4.1k total citations · 2 hit papers
19 papers, 2.9k citations indexed

About

William A. Eimer is a scholar working on Physiology, Molecular Biology and Pharmacology. According to data from OpenAlex, William A. Eimer has authored 19 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Physiology, 6 papers in Molecular Biology and 6 papers in Pharmacology. Recurrent topics in William A. Eimer's work include Alzheimer's disease research and treatments (14 papers), Cholinesterase and Neurodegenerative Diseases (6 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). William A. Eimer is often cited by papers focused on Alzheimer's disease research and treatments (14 papers), Cholinesterase and Neurodegenerative Diseases (6 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). William A. Eimer collaborates with scholars based in United States, United Kingdom and Switzerland. William A. Eimer's co-authors include Robert Vassar, Rudolph E. Tanzi, Deepak Kumar Vijaya Kumar, Robert D. Moir, Kevin J. Washicosky, Katherine R. Sadleir, Se Hoon Choi, Gawain McColl, Lee E. Goldstein and Stephanie C. Tucker and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Nature Neuroscience.

In The Last Decade

William A. Eimer

18 papers receiving 2.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease

2016 749 citations Deepak Kumar Vijaya Kumar, Se Hoon Choi et al. Science Translational Medicine profile →
Alzheimer’s Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection

2018 484 citations William A. Eimer, Deepak Kumar Vijaya Kumar et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
William A. Eimer United States	13	1.9k	1.1k	707	472	429	19	2.9k
Kristina Endres Germany	32	1.7k 0.9×	1.9k 1.7×	413 0.6×	527 1.1×	413 1.0×	104	3.6k
Demian Obregon United States	27	1.3k 0.7×	761 0.7×	736 1.0×	444 0.9×	305 0.7×	47	2.7k
Elizabeth A. Eckman United States	24	2.6k 1.4×	1.6k 1.5×	611 0.9×	577 1.2×	150 0.3×	40	3.9k
Vitaly Vasilevko United States	28	1.5k 0.8×	847 0.8×	881 1.2×	414 0.9×	314 0.7×	49	2.8k
Timothy Y. Huang United States	26	1.2k 0.6×	1.4k 1.3×	883 1.2×	498 1.1×	231 0.5×	43	3.2k
Kevin J. Washicosky United States	9	1.3k 0.7×	1.0k 0.9×	642 0.9×	225 0.5×	502 1.2×	11	2.5k
Balmiki Ray United States	28	1.0k 0.5×	1.0k 0.9×	361 0.5×	531 1.1×	208 0.5×	51	2.8k
M. Azhar Chishti United States	19	2.0k 1.0×	1.0k 1.0×	721 1.0×	457 1.0×	134 0.3×	29	2.7k
Nan Sun China	24	784 0.4×	1.2k 1.1×	559 0.8×	506 1.1×	320 0.7×	80	2.9k
Domenica Donatella Li Puma Italy	23	895 0.5×	666 0.6×	468 0.7×	461 1.0×	259 0.6×	36	1.9k

Countries citing papers authored by William A. Eimer

Since Specialization

Citations

This map shows the geographic impact of William A. Eimer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William A. Eimer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William A. Eimer more than expected).

Fields of papers citing papers by William A. Eimer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William A. Eimer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William A. Eimer. The network helps show where William A. Eimer may publish in the future.

Co-authorship network of co-authors of William A. Eimer

This figure shows the co-authorship network connecting the top 25 collaborators of William A. Eimer. A scholar is included among the top collaborators of William A. Eimer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William A. Eimer. William A. Eimer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Eimer, William A., Alex S. Rodriguez, Joseph Park, et al.. (2025). Phosphorylated tau exhibits antimicrobial activity capable of neutralizing herpes simplex virus 1 infectivity in human neurons. Nature Neuroscience. 29(3). 604–616. 1 indexed citations

Piłat, Dominika, Dmitry Prokopenko, Chih‐Chung Lin, et al.. (2025). The gain-of-function TREM2-T96K mutation increases risk for Alzheimer’s disease by impairing microglial function. Neuron. 114(1). 46–66.e13.

Shanmugam, Nanda Kumar N., Frank Zamudio, Deepak Kumar Vijaya Kumar, et al.. (2025). Acute experimental colitis in 5xFAD Alzheimer's disease mice leads to enhanced monocyte infiltration into the brain accompanied by reduced β‐amyloid deposition. Alzheimer s & Dementia. 21(6). e70292–e70292. 2 indexed citations

Kumar, Deepak Kumar Vijaya, Teryn Mitchell, Nanda Kumar N. Shanmugam, et al.. (2025). Human amylin is a potent antimicrobial peptide that exhibits antimicrobial synergism with the amyloid beta protein. Alzheimer s & Dementia. 21(8). e70490–e70490. 1 indexed citations

Shanmugam, Nanda Kumar N., William A. Eimer, Deepak Kumar Vijaya Kumar, & Rudolph E. Tanzi. (2024). The brain pathobiome in Alzheimer's disease. Neurotherapeutics. 21(6). e00475–e00475. 3 indexed citations

Lathe, Richard, Brian J. Balin, Garth D. Ehrlich, et al.. (2023). Establishment of a consensus protocol to explore the brain pathobiome in patients with mild cognitive impairment and Alzheimer's disease. Alzheimer s & Dementia. 19(11). 5209–5231. 18 indexed citations

Cruz, Lilian, Bence György, Pike-See Cheah, et al.. (2020). Mutant Allele-Specific CRISPR Disruption in DYT1 Dystonia Fibroblasts Restores Cell Function. Molecular Therapy — Nucleic Acids. 21. 1–12. 10 indexed citations

Eimer, William A., Deepak Kumar Vijaya Kumar, Nanda Kumar N. Shanmugam, et al.. (2018). Alzheimer’s Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection. Neuron. 99(1). 56–63.e3. 484 indexed citations breakdown →

Kumar, Deepak Kumar Vijaya, Se Hoon Choi, Kevin J. Washicosky, et al.. (2016). Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease. Science Translational Medicine. 8(340). 340ra72–340ra72. 749 indexed citations breakdown →

10.

Kumar, Deepak Kumar Vijaya, William A. Eimer, Rudolph E. Tanzi, & Robert D. Moir. (2016). Alzheimer’s Disease: the Potential Therapeutic Role of the Natural Antibiotic amyloid-β Peptide. Neurodegenerative Disease Management. 6(5). 345–348. 34 indexed citations

11.

Sadleir, Katherine R., William A. Eimer, Sarah L. Cole, & Robert Vassar. (2015). Aβ reduction in BACE1 heterozygous null 5XFAD mice is associated with transgenic APP level. Molecular Neurodegeneration. 10(1). 1–1. 168 indexed citations

12.

Sadleir, Katherine R., William A. Eimer, Randal J. Kaufman, Pavel Osten, & Robert Vassar. (2014). Genetic Inhibition of Phosphorylation of the Translation Initiation Factor eIF2α Does Not Block Aβ-Dependent Elevation of BACE1 and APP Levels or Reduce Amyloid Pathology in a Mouse Model of Alzheimer’s Disease. PLoS ONE. 9(7). e101643–e101643. 32 indexed citations

13.

Eimer, William A. & Robert Vassar. (2013). Neuron loss in the 5XFAD mouse model of Alzheimer’s disease correlates with intraneuronal Aβ42 accumulation and Caspase-3 activation. Molecular Neurodegeneration. 8(1). 2–2. 318 indexed citations

14.

Kandalepas, Patty C., Katherine R. Sadleir, William A. Eimer, et al.. (2013). The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques. Acta Neuropathologica. 126(3). 329–352. 196 indexed citations

15.

Youmans, Katherine L., Leon M. Tai, Evelyn Nwabuisi‐Heath, et al.. (2012). APOE4-specific Changes in Aβ Accumulation in a New Transgenic Mouse Model of Alzheimer Disease. Journal of Biological Chemistry. 287(50). 41774–41786. 214 indexed citations

16.

Youmans, Katherine L., Leon M. Tai, Takahisa Kanekiyo, et al.. (2012). Intraneuronal Aβ detection in 5xFAD mice by a new Aβ-specific antibody. Molecular Neurodegeneration. 7(1). 8–8. 143 indexed citations

17.

Hitt, Brian D., Sean M. Riordan, Lokesh Kukreja, et al.. (2012). β-Site Amyloid Precursor Protein (APP)-cleaving Enzyme 1 (BACE1)-deficient Mice Exhibit a Close Homolog of L1 (CHL1) Loss-of-function Phenotype Involving Axon Guidance Defects. Journal of Biological Chemistry. 287(46). 38408–38425. 122 indexed citations

18.

Rajapaksha, Tharinda W., William A. Eimer, Thomas Bozza, & Robert Vassar. (2011). The Alzheimer's β-secretase enzyme BACE1 is required for accurate axon guidance of olfactory sensory neurons and normal glomerulus formation in the olfactory bulb. Molecular Neurodegeneration. 6(1). 88–88. 84 indexed citations

19.

O’Connor, Tracy, Katherine R. Sadleir, Erika Maus, et al.. (2008). Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis. Neuron. 60(6). 988–1009. 344 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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