Junichi Hitomi

6.1k total citations · 3 hit papers
17 papers, 4.6k citations indexed

About

Junichi Hitomi is a scholar working on Molecular Biology, Cell Biology and Epidemiology. According to data from OpenAlex, Junichi Hitomi has authored 17 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Cell Biology and 6 papers in Epidemiology. Recurrent topics in Junichi Hitomi's work include Endoplasmic Reticulum Stress and Disease (7 papers), Autophagy in Disease and Therapy (6 papers) and Cell death mechanisms and regulation (5 papers). Junichi Hitomi is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (7 papers), Autophagy in Disease and Therapy (6 papers) and Cell death mechanisms and regulation (5 papers). Junichi Hitomi collaborates with scholars based in Japan, United States and Canada. Junichi Hitomi's co-authors include Alexei Degterev, Junying Yuan, Taiichi Katayama, Masaya Tohyama, Kazunori Imaizumi, Takashi Kudo, Scott A. Gerber, Dana E. Christofferson, Takayuki Manabe and Gregory D. Cuny and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Junichi Hitomi

17 papers receiving 4.5k citations

Hit Papers

Identification of RIP1 kinase as a specific cellular targ... 2004 2026 2011 2018 2008 2008 2004 500 1000 1.5k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Junichi Hitomi Japan 15 3.1k 1.4k 1.1k 1.1k 595 17 4.6k
Clark Distelhorst United States 43 3.8k 1.2× 1.1k 0.8× 835 0.8× 759 0.7× 294 0.5× 82 5.3k
Sanjeev Gupta Ireland 29 2.7k 0.9× 1.8k 1.3× 1.2k 1.1× 472 0.4× 428 0.7× 62 4.5k
Katiuscia Bianchi United Kingdom 17 2.7k 0.9× 871 0.6× 1.2k 1.0× 586 0.5× 564 0.9× 27 4.1k
Yoav D. Shaul Israel 23 3.2k 1.0× 925 0.7× 554 0.5× 642 0.6× 420 0.7× 40 4.6k
Angelika S. Rambold Germany 18 2.9k 0.9× 717 0.5× 1.5k 1.4× 933 0.9× 747 1.3× 26 4.8k
Kei Tobiume Japan 22 3.2k 1.0× 1.2k 0.9× 684 0.6× 689 0.6× 296 0.5× 45 4.6k
Thore Hettmann United States 14 2.0k 0.7× 1.7k 1.2× 724 0.7× 488 0.5× 287 0.5× 22 3.5k
Guadalupe Sabio Spain 31 2.5k 0.8× 556 0.4× 1.1k 1.0× 736 0.7× 927 1.6× 71 4.6k
Shani Bialik Israel 28 3.0k 1.0× 859 0.6× 2.1k 1.9× 443 0.4× 301 0.5× 46 4.7k
Michael S. Kilberg United States 31 2.9k 0.9× 2.0k 1.5× 778 0.7× 343 0.3× 695 1.2× 46 4.8k

Countries citing papers authored by Junichi Hitomi

Since Specialization
Citations

This map shows the geographic impact of Junichi Hitomi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junichi Hitomi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junichi Hitomi more than expected).

Fields of papers citing papers by Junichi Hitomi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Junichi Hitomi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junichi Hitomi. The network helps show where Junichi Hitomi may publish in the future.

Co-authorship network of co-authors of Junichi Hitomi

This figure shows the co-authorship network connecting the top 25 collaborators of Junichi Hitomi. A scholar is included among the top collaborators of Junichi Hitomi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Junichi Hitomi. Junichi Hitomi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Shinohara, Mitsuru, et al.. (2023). APOEgenotypes modify the obesity paradox in dementia. Journal of Neurology Neurosurgery & Psychiatry. 94(9). 670–680. 11 indexed citations
2.
Christofferson, Dana E., Ying Li, Junichi Hitomi, et al.. (2012). A novel role for RIP1 kinase in mediating TNFα production. Cell Death and Disease. 3(6). e320–e320. 168 indexed citations
3.
Hitomi, Junichi, Dana E. Christofferson, Aylwin Ng, et al.. (2008). Identification of a Molecular Signaling Network that Regulates a Cellular Necrotic Cell Death Pathway. Cell. 135(7). 1311–1323. 784 indexed citations breakdown →
4.
Degterev, Alexei, Junichi Hitomi, Irene L. Ch’en, et al.. (2008). Identification of RIP1 kinase as a specific cellular target of necrostatins. Nature Chemical Biology. 4(5). 313–321. 1603 indexed citations breakdown →
5.
Matsuzaki, Shinsuke, Keisuke Kawamoto, Yoshihisa Koyama, et al.. (2007). Presenilin-1 mutation activates the signaling pathway of caspase-4 in endoplasmic reticulum stress-induced apoptosis. Neurochemistry International. 52(4-5). 683–687. 44 indexed citations
6.
Yamagishi, Satoru, Yoshihisa Koyama, Taiichi Katayama, et al.. (2007). An In Vitro Model for Lewy Body-Like Hyaline Inclusion/Astrocytic Hyaline Inclusion: Induction by ER Stress with an ALS-Linked SOD1 Mutation. PLoS ONE. 2(10). e1030–e1030. 31 indexed citations
7.
Katayama, Taiichi, Kazunori Imaizumi, Takayuki Manabe, et al.. (2004). Induction of neuronal death by ER stress in Alzheimer’s disease. Journal of Chemical Neuroanatomy. 28(1-2). 67–78. 238 indexed citations
8.
Hitomi, Junichi, Taiichi Katayama, Manabu Taniguchi, et al.. (2004). Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12. Neuroscience Letters. 357(2). 127–130. 221 indexed citations
9.
Katayama, Taiichi, Kazunori Imaizumi, Takunari Yoneda, et al.. (2004). Role of ARF4L in Recycling Between Endosomes and the Plasma Membrane. Cellular and Molecular Neurobiology. 24(1). 137–147. 9 indexed citations
10.
Yoneda, Takunari, Takayuki Manabe, Satoru Yamagishi, et al.. (2004). JAB1 participates in unfolded protein responses by association and dissociation with IRE1. Neurochemistry International. 45(5). 765–772. 67 indexed citations
11.
Hitomi, Junichi, Taiichi Katayama, Yutaka Eguchi, et al.. (2004). Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Aβ-induced cell death. The Journal of Cell Biology. 165(3). 347–356. 710 indexed citations breakdown →
12.
Manabe, Takayuki, Taiichi Katayama, Naoyuki Sato, et al.. (2003). Induced HMGA1a expression causes aberrant splicing of Presenilin-2 pre-mRNA in sporadic Alzheimer's disease. Cell Death and Differentiation. 10(6). 698–708. 62 indexed citations
13.
Sato, Naoya, Kazunori Imaizumi, Takayuki Manabe, et al.. (2001). Increased Production of β-Amyloid and Vulnerability to Endoplasmic Reticulum Stress by an Aberrant Spliced Form of Presenilin 2. Journal of Biological Chemistry. 276(3). 2108–2114. 101 indexed citations
14.
Morihara, Takashi, Taiichi Katayama, Naoya Sato, et al.. (2000). Absence of endoproteolysis but no effects on amyloid β production by alternative splicing forms of presenilin-1, which lack exon 8 and replace D257A. Molecular Brain Research. 85(1-2). 85–90. 15 indexed citations
15.
Miyoshi, Ko, Taiichi Katayama, Kazunori Imaizumi, et al.. (2000). Characterization of mouse Ire1α: cloning, mRNA localization in the brain and functional analysis in a neural cell line. Molecular Brain Research. 85(1-2). 68–76. 23 indexed citations
16.
Gomi, Fumi, Kazunori Imaizumi, Takunari Yoneda, et al.. (2000). Molecular Cloning of a Novel Membrane Glycoprotein, Pal, Specifically Expressed in Photoreceptor Cells of the Retina and Containing Leucine-Rich Repeat. Journal of Neuroscience. 20(9). 3206–3213. 22 indexed citations
17.
Katayama, Taiichi, Kazunori Imaizumi, Naoya Sato, et al.. (1999). Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response. Nature Cell Biology. 1(8). 479–485. 449 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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